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Monash University2009 Alcohol and Other Drugs

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Title: Monash University2009 Alcohol and Other Drugs


1
Monash University-2009Alcohol and Other Drugs
  • Presenter Effie Moraitis
  • Senior Clinician

2
Topics covered today
  • What is a drug? some definitions
  • Theoretical models of drug use
  • Harm minimisation
  • Dependence Syndrome
  • Classification of drugs and their effects
  • AOD assessment some important points
  • Stages of Change model
  • Effects of Alcohol and Marijuana use. Especially
    Neurological impact.
  • Withdrawal Symptoms
  • Treatment Options

3
Making sense of AOD issues
  • What is a drug? Who uses drugs?
  • Theoretical models of drug use
  • Harm minimisation
  • Dependency, tolerance and withdrawal
  • Patterns of drug use

4
What is a drug? Definitions
  • World Health Organisation
  • Any substance which when taken into the body,
    alters its function physically and/or
    psychologically, excluding food, water and
    oxygen (cited in McCallum 1994 p 90 WHO 1994)
  • Any substance the people consider to be a drug,
    with the understanding that this will change from
    culture to culture and from time to time
    (Krivanek 1995 p 2)

5
Who Uses Drugs?
  • Drug use occurs across cultures, suburbs, genders
    and class systems.
  • Common thought is that people in lower
    socioeconomic communities use more drugs than
    those from affluent communities.
  • True or False?

6
Theoretical Models of drug use
  • Moral Model
  • Key assumption
  • using drugs is morally wrong and anti-social
  • Intervention
  • spiritual direction, gaol, providing an
    environment that promotes pro-social values

7
Theoretical Models of drug use
  • Disease Model (Medical Model)
  • Key assumptions
  • Some people have a natural predisposition to
    drug use
  • Dependency is controlled by physiological /
    genetic factors
  • Dependency will inevitably result in a loss of
    control and progression of their condition

8
Theoretical Models of drug use
  • Disease Model cont
  • Interventions
  • - Total abstinence
  • - Self-help (eg 12 step programs)
  • - Supporters of this model suggest that a person
    addicted to a drug / s will be unable to control
    their substance use.

9
Theoretical Models of drug use
  • Social learning model
  • Key assumptions
  • Focuses on the interaction between the
    environment, the individual and the drug. Drug
    use is learnt
  • Intervention
  • Learning new coping strategies
  • May use cognitive restructuring techniques

10
Theoretical Models of drug use
  • Public Health Model
  • Key assumptions
  • Looks at the availability of the drug, cost, the
    properties of the substance, individual factors
    and socio-political factors (advertising,
    economic gains from drug use, peer pressure)
  • Interventions
  • Education, political actions, legislation

11
Harm minimisation approach
  • Has underpinned Australias drug strategy since
    1985
  • Harm minimisation accepts that the use of drugs
    is a part of life and that on many occasions,
    drug use is non-problematic. It also recognises
    that drug use can cause harm amount the people
    that use and the wider community. Harm
    minimisation seeks to reduce drug related harm

12
National Drug Strategic Framework
  • The National Drug Strategy focuses on three core
    elements of harm reduction
  • - demand reduction (prevention)
  • - supply reduction (law enforcement)
  • - harm reduction (education, information

13
Dependence, Tolerance and Withdrawal
  • Dependence
  • Maladaptive pattern of substance use, leading to
    clinically significant impairment or distress.
  • The substance is often taken in larger amounts or
    over a longer period than intended
  • There is a persistent desire or unsuccessful
    efforts to cut down or control substance use.

14
Dependence, Tolerance and Withdrawal
  • Tolerance
  • The need for increased amounts of the substance
    to achieve intoxication or the desired effect.
  • This may vary
  • - across individuals
  • - across substances
  • - across physiological systems

15
Dependence, Tolerance and Withdrawal
  • Withdrawal
  • Maladaptive behavioural change (which may be the
    opposite to the acute effect of the substance
  • Withdrawal syndromes may change according to the
    substance
  • The same or closely related substances may be
    taken to relieve or avoid withdrawal symptoms

16
Patterns of consumption and types of drug use
  • Controlled use
  • Experimental use
  • Social / recreational use
  • Circumstantial use / situational
  • Intensive use
  • Dependency

17
Drug Classifications
  • Drugs are often classified in two ways
  • Legal status
  • Central nervous system effects
  • Classifications
  • Depressants
  • Stimulants
  • Hallucinogens

18
An AOD assessment
  • What are some important points to cover in an AOD
    assessment?

19
Assessment some points
  • Psychosocial history
  • Substance use history type of substances used,
    frequency, quantity, when and how they use,
    circumstances
  • Medical history and current medications
  • Psychiatric history
  • Four Ls Liver, Lover, Livelihood, Legal

20
Stages of Change Model
  • DiClementi Prochaska (1986)
  • Illustrates that change is a process, and change
    is a process that can take time
  • Is a useful tool in identifying where people are
    at in their change process
  • People can go forwards or backwards in this model.

21
Six stage model
  • Pre-contemplative stage
  • Person does not see they have a problem
  • Contemplative stage
  • Person is weighing up cost / benefits to change
  • Preparation stage
  • Person is preparing to change
  • Action stage
  • Person is actively pursuing change
  • Maintenance stage
  • Person is maintaining the change
  • Relapse stage
  • Person returns to previous levels of drug use

22
Alcohol What is it?
  • Alcohol is ethyl alcohol or ethanol. It is a
    natural product of fermenting sugars. Its
    usually made from grains such as hops, barley,
    rice and/or fruits. It can also be made from
    other plants.
  • The concentration of alcohol varies widely
    according to the type of alcoholic drink. Hence
    different standard drink variations.
  • ROA Oral/Swallowed

23
Side Effects
  • Feelings of relaxation, lowered inhibition,
    increased sociability.
  • In higher doses alcohol can cause dizziness,
    nausea, slurred speech, slower reflexes,
    sleepiness, dehydration and bad judgement.
  • In even higher doses it can cause blackouts,
    organ failure, liver damage, coma and in extreme
    cases death.

24
Neurological Side Effects
  • When Alcohol hits your lips your whole body is
    affected.
  • Within the lining of your mouth a small
    percentage of alcohol is absorbed. It irritates
    the mouth lining as well as the oesophagus,
    acting like an anaesthetic.
  • From there

25
Neurological side effects cont.
  • Alcohol travels to your stomach
  • This is where its absorbed into the bloodstream.
  • Then it continues to the small intestine and from
    here it it is completely absorbed into the
    bloodstream. Alcohol can reach the small
    intestine within 5 minutes.
  • At this point the alcohol can reach every cell in
    the body

26
Effects of alcohol on the body
  • Alcohol shares many properties with water. It is
    highly soluble in water and travels through the
    body as water does.
  • In its circulation through the body, the alcohol
    reaches the brain.
  • The feelings of intoxication now begin. They are
    dependant on concentration of alcohol in the body
    and how fast it reaches the small intestine, the
    strength, whether there has been food
    consumption, age gender, body size..

27
Effects cont
  • The liver metabolises 90 of the alcohol in your
    body. The rest is eliminated by perspiration, or
    via kidneys and lungs.
  • The remaining alcohol continues its circulation
    throughout the body.
  • Where to from here?

28
Effects cont
  • Alcohols effect on the brain is abnormal, as the
    brain is usually protected from chemicals and
    drugs by the blood/brain barrier which acts as
    a filter.
  • The simple molecular structure of alcohol allows
    the penetration into the brain. This occurs in
    the frontal lobe.

29
Effects cont
  • At this point there is a loss of reason, caution
    and inhibitions.
  • At the Parietal Lobe there is a loss of fine
    motor skill, slower reaction, reflex time and
    shaking.
  • In the Temporal lobe occurs the slurred speech as
    well as impaired hearing.
  • At the Occipital Lobe blurred vision and
    judgement, and loss of vital functions

30
Chronic Alcohol Consumption
  • When people consume large quantities of alcohol,
    they develop a Thiamine Deficiency(Vitamin B1).
  • This causes the neurological disorder called
    Wernicke-Korsakoff Syndrome. Wernickes
    encephalopathy and Korsakoffs psychosis are the
    acute and chronic phases of this condition
    especially affecting short term memory.
  • An absence of Thiamine results in an inadequate
    supply of energy to the brain.

31
Chronic Consumption cont
  • In chronic heavy alcohol consumers, the
    frontal lobes of the brain shrink. This is
    probably partly due to loss of water and partly
    due to cell death.
  • The lobes may expand again if the person stops
    drinking, but evidence of cell death remains in
    impaired function.

32
Did You Know?
  • When people become intoxicated it is common to
    feel warmth, however this is misleading. Alcohol
    acts as a vasodilator, dilating surface blood
    cells. This actually expands blood vessels
    causing people to lose body heat.

33
Withdrawal Symptoms of Alcohol
  • Sweating, facial flushing
  • Tremors
  • Agitation
  • Palpitations, hypertension
  • Poor appetite, nausea, vomiting, diarrhoea
  • Poor sleep, anxiety
  • Cravings, strong desire to drink
  • Poor concentration
  • Headaches

34
More Serious Symptoms
  • Severe hypertension
  • Seizures
  • Hallucinations, delirium
  • Arrhythmias
  • Precipitation/ exacerbation of underlying medical
    or psychiatric disorders
  • Mood swings
  •  

35
Illnesses caused by alcohol
  • Sleep and sexual disorders
  • Psoriasis of the liver
  • Psychotic and mood disorders
  • Foetal Alcohol Syndrome
  • Depression
  • Heart failure
  • Wernicke-Korsakoff Syndrome

36
Marijuana What Is It?
  • Marijuana also known as Cannabis, is a green,
    brown, or grey mixture of dried, shredded leaves,
    stems, seeds, and flowers of the hemp plant. You
    may hear marijuana called by street names such as
    pot, herb, weed, grass, boom, Mary Jane,
    gangster, or chronic. There are more than 200
    slang terms for marijuana.Sinsemilla
    (sin-seh-me-yah a Spanish word), hashish ("hash"
    for short), and hash oil are stronger forms of
    marijuana.

37
The Classification
  • All forms of marijuana are mind-altering. In
    other words, they change how the brain works.
    They all contain THC (delta-9-tetrahydrocannabinol
    ), the main active chemical in marijuana. They
    also contain more than 400 other chemicals.
    Marijuana's effects on the user depend on the
    strength or potency of the THC it contains (5).
    THC potency of marijuana has increased since the
    1970s but has been about the same since the
    mid-1980s.

38
Effects of Marijuana
  • Impaired perception
  • Diminished short-term memory
  • Loss of concentration and coordination
  • Impaired judgement
  • Increased risk of accidents
  • Loss of motivation
  • Diminished inhibitions/Increased heart rate
  •  

39
Effects of Marijuana cont
  • Anxiety, panic attacks, and paranoia
  • Hallucinations/Delusions
  • Damage to the respiratory, reproductive, and
    immune systems
  • Increased risk of cancer
  • Psychological dependency

40
Neurological Effects of Marijuana
  • When someone smokes Marijuana, THC rapidly passes
    through the bloodstream.
  • This carries the chemical to organs throughout
    the body, including the brain

41
Neurological effects cont..
  • Cannabinoids is an active ingredient of
    Marijuana. The most psychoactive cannabinoids
    chemical in Marijuana that has the biggest impact
    on the brain is tetrahydrocannibol, or THC. THC
    is the main active ingredient in marijuana
    because it affects the brain by binding to and
    activating specific receptors, known as
    cannabinoid receptors. "These receptors control
    memory, thought, concentration, time and depth,
    and coordinated movement. THC also affects the
    production, release or re-uptake (a regulating
    mechanism) of various neurotransmitters

42
Neurological Effects of Marijuana cont..
  • Neurotransmitters are chemical messenger
    molecules that carry signals between neurons.
    Some of these affects are personality
    disturbances, depression and chronic anxiety.
    Psychiatrists who treat schizophrenic patients
    advise them to not use this drug because
    marijuana can trigger severe mental disturbances
    and cause a relapse.

43
Memory Loss
  • When one's memory is affected by high doses of
    marijuana, short-term memory is the first to be
    triggered. Marijuana's damage to short-term
    memory occurs because THC alters the way in which
    information is processed by the hippocampus, a
    brain area responsible for memory formation. One
    region of the brain that contains a lot of THC
    receptors is the hippocampus, which processes
    memory.

44
Emotional Impairment
  • Marijuana also impairs emotions. When smoking
    marijuana, the user may have uncontrollable
    laughter one minute and paranoia the next. This
    instant change in emotions has to do with the way
    that THC affects the brain's limbic system. The
    limbic system is another region of the brain that
    governs one's behaviour and emotions.

45
Cognitive Impairment
  • The chemicals in Marijuana bring cognitive
    impairment and troubles with learning for the
    user. Smoking marijuana causes some changes in
    the brain that are like those caused but cocaine,
    heroin, and alcohol.

46
Withdrawal Symptoms of Marijuana
  • Anxiety, agitation, restlessness, irritability
  • Nausea
  • Dysphoria, lethargy
  • Cravings, strong desire to use
  • Sleep disturbances (including vivid dreams,
    nightmares, insomnia)
  • Sweating
  • Headaches
  • Mood disturbances
  •  

47
Illnesses caused by marijuana
  • Cannabis is linked with Mental Health
    Disorders.
  • If there is a predisposition in a persons
    family history of a mental health disorder,
    Marijuana can trigger it to occur.
  • Short Term Memory loss
  • Research has begun on potential Learning
    difficulties experienced by children whose
    mothers used Marijuana during pregnancy and
    breastfeeding.

48
Treatment Options
  • Treatment for any drug is more effective when
    tailored to the specific individual requirements.
  • It can involve a combination of methods
    including
  • Medication and GP/Psychiatric Involvement
  • Individual Counselling
  • Group Therapy
  • Home Based Withdrawal
  • Residential Withdrawal
  • Long Term Rehabilitation
  • And more

49
Treatment Options cont
  • In Victoria there are over 1,000 Alcohol and
    Other Drug Treatment Services.
  • Inclusive in these are 24/7, free and
    immediate Counselling, Information and referral
    services specifically for anyone who has any
    Alcohol and Other Drug related concerns. These
    services are anonymous and confidential (within
    confidentiality limitations).

50
24/7 Services
  • DirectLine 1800 888 236 for consumers and
    significant others who are experiencing Alcohol
    and Other Drugs related concerns.
  • DACAS 1800 812 804 Drug and Alcohol Clinical
    Advisory Service for Health Professionals.
  • This service has 24/7 Addiction Specialist
    Medical Consultants.

51
24/7 Services cont
  • CounsellingOnline www.counsellingonline.org.au A
    web-Based Counselling, Information, Referral and
    Support service 24/7 for consumers and
    significant others specifically related to
    Alcohol and Other Drug concerns. This is a
    National Service.
  • All services are staffed by Professional
    Counsellors
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