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Peripheral Vascular Diseases

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Peripheral Vascular Diseases Arterial Manifestations: Diminished or absent pulses Smooth, shiny, dry skin, no hair No edema Round, regularly shaped painful ulcers on ... – PowerPoint PPT presentation

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Title: Peripheral Vascular Diseases


1
Peripheral Vascular Diseases
  • Arterial Manifestations
  • Diminished or absent pulses
  • Smooth, shiny, dry skin, no hair
  • No edema
  • Round, regularly shaped painful ulcers on distal
    foot, toes or webs of toes
  • Dependent rubor
  • Pallor and pain when legs elevated
  • Intermittent claudication
  • Brittle, thick nails
  • Venous Manifestations
  • Normal pulses
  • Brown patches of discoloration on lower legs
  • Dependent edema
  • Irregularly shaped, usually painless ulcers on
    lower legs and ankles
  • Dependent cyanosis and pain
  • Pain relief when legs elevated
  • No intermittent claudication
  • Normal nails

2
Peripheral Arterial Occlusive Disease (830)
  • Pathophysiology Narrowing and sclerosis of
    large arteries (femoral, iliac, popliteal)
    especially at bifurcations due to plaque
    formation
  • Risk factors smoking, obesity, sedentary
    lifestyle, HTN, DM, hyperlipidemia, Fa hx
  • S/S see previous slide. May also have bruit
    over femoral or popliteal doppler area
  • Dx Tests US, exercise testing (822), pulse
    volumes, angiography (823), Trendelenberg test
    (see Assessment text)

3
Treatment of PAOD
  • Meds-antiplatelets, antilipidemics, vasodilators,
    Trental (misc.)
  • Good skin and foot care-podiatrist for probs
  • Avoid standing for long periods, crossing legs,
    tobacco
  • Low fat diet, lose weight
  • Exercise to point of pain Buerger-Allen
    exercises
  • Surgery-fem-pop bypass, endardarectomy, amputation

4
Nursing Management of PAOD
  • Administer and monitor meds
  • Patient education
  • Meds
  • Good skin and foot care
  • Avoiding pressure, tobacco
  • Diet and exercise
  • S/S of acute occlusion
  • Pre and postop care if indicated

5
Nursing Management of Pt. with Fem-Pop Bypass
(832-3)
  • Preop Goal Prevent trauma and maintain
    circulation
  • Keep leg level or slightly dependent
  • Protect leg from trauma
  • Administer anticoagulants
  • Assess and monitor VS and NV status
  • Assess and monitor anxiety
  • Education on above and on what to expect after
    surgery

6
Nursing Management of Fem-pop Bypass contd
  • Postop Goal Maintain adequate circulation
    through graft (saphenous, umbilical, Gore-Tex)
  • ICU at first
  • Assess VS, pulse ox, IO, PT, PTT, lytes, BUN,
    creat
  • Monitor NV status (6 Ps) q1h x 8, then q2h x 24
    using doppler. Compare to other extremity.
  • Assess ankle-brachial index (ABI) q8h x 24
  • Administer anticoags, analgesics
  • TEDs for some surgeons, no leg crossing, or
    prolonged extremity dependency
  • Pt education on meds, activity, how to recognize
    vascular complications

7
Nursing Management contd
  • Notify surgeon immediately for
  • Absence of pulse
  • Abnormal ABI
  • Abnormal VS
  • Hemorrhage
  • Severe edema with pain and ltsensation (may
    indicate compartment syndrome)

8
Acute Arterial Occlusive Disease (arterial
embolism-840)
  • Pathophysiology blood clots from arteries, left
    ventricle, or trauma suddenly break loose and
    become free flowing, lodge in bifurcations,
    causing obstruction distally with acute and
    sudden symptoms
  • Assessment 6 Ps (pain, pallor, pulselessness,
    paresthesia, paralysis, poikilothermia), ABIlt1,
    US, MRI, or angiography

9
Management of Arterial Embolism
  • Medical
  • Anticoagulants-heparin bolus then 1000U/hr
  • Thrombolytics
  • Surgical (depends on occlusion time)
  • Embolectomy (840)
  • Bypass
  • Angioplasty with stent placement
  • Nursing
  • Administer and monitor anticoag or thrombolytic
    tx
  • If surgery, then monitor for postop angioplasty
    and stent placement, bypass, or embolectomy
    (similar to bypass except no ICU and hospital
    time is less).

10
Buergers Disease (thromboangiitis obliterans-834)
  • Pathophysiology obstructive and inflammatory
    disease of small and medium sized arteries and
    veins. Believed to be autoimmune. Has
    exacerbations and remissions. Smoking is very
    high risk factor.
  • Assessment pain and instep claudication,
    intense rubor, absence of distal pulses (pedal,
    radial, ulnar), paresthesias segmental limb
    blood pressures, US, angiography

11
Management of Buergers Disease
  • Medical/Surgical
  • Pain meds
  • Stop smoking
  • Treatment of infection and gangrene
  • Sympathectomy (removal of sympathetic ganglia or
    branches-causes permanent vasodilation
  • Amputation
  • Nursing
  • Support stopping smoking
  • Administer pain meds
  • Education regarding protection extremities from
    cold and trauma.

12
Raynauds Disease (841)
  • Pathophysiology arterial spasms of small
    cutaneous vessels of fingers and toes. May have
    too many alpha 2 receptors leading to
    vasoconstriction and not enough beta receptors.
    Aggravated by cold and stress.
  • Assessment classic tri-color symptoms-pallor,
    cyanosis, rubor, pain, and paresthesia.
    Bilateral and symmetric.

13
Management of Raynauds Disease
  • Medical/Surgical
  • Avoiding cold, stress, nicotine
  • Ca channel blockers (particularly nifedipine)
    especially for acute vasospasm
  • sympathectomy
  • Nursing
  • Avoid stress, take stress mgmt classes
  • Avoid cold and trauma
  • Teach about nifedipine (can cause orthostatic
    hypotension)

14
Hypertension (855)
  • Definitions and Etiology
  • SBP gt 140 and DBP gt 90 at least 3 times.
  • Affects 20-25 of population. 90-95 have
    primary or essential HTN (unknown etiology).
    Other 5-10 have secondary, meaning there is a
    disease process causing it (i.e., thyrotoxicosis,
    renal artery stenosis, pheochromocytoma).
    Hypertensive crisis-DBP gt 120. Malignant
    HTN-rises rapidly. White coat HTN-increased BP
    when patient goes to MD.
  • Risk factors are similar to CAD
  • Classifications p. 855, Table 32-1

15
Assessment of HTN
  • S/S
  • Usually absent unless severe or advanced
  • If symptoms they include HA, blurred vision,
    dizziness, nosebleeds
  • BP gt 140/90
  • S4 gallop rhythm
  • Dx Tests
  • BP readings
  • CBC, UA, lytes, lipids, glucose, renal and liver
    functions
  • ECG
  • CXR
  • Echo

16
Management of HTN
  • Monitoring of BP recommendations for F/U on p.
    856, Table 32-2.
  • Algorithim p. 859. Lifestyle changes and meds.
  • Lifestyle changes include wt reduction, heart
    healthy diet, no nicotine, regular exercise.
  • Meds 50-1 90-2. Stepped approach with
  • Diuretics
  • Beta and alpha blockers
  • Vasodilators
  • ACEIs and Angiotensin receptor blockers
  • Ca channel blockers

17
Management of HTN contd
  • Treat complications
  • Angina, MI
  • CHF-from LV hypertrophy
  • CRF
  • CVA
  • Retinal hemorrhages

18
Nursing Management of HTN
  • History assess for all risk factors
  • Physical assessment heart sounds, pulses, VS,
    lungs, carotid bruit, retina, thyroid, abd, neuro
  • Pt education lifestyle modifications, monitor
    BP and daily wts and keep a record, keep appts,
    safety r/t hot showers and environments,
    orthostatic BPs, first dose syncope, meds, dont
    stop meds suddenly, OTC meds, keep list of meds,
    interacting meds-BCPs, steroids, NSAIDs, some
    antidepressants, antihistamines, nasal
    decongestants
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