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MINI MED SCHOOL UNIVERSITY OF MARYLAND SCHOOL OF MEDICINE

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Title: MINI MED SCHOOL UNIVERSITY OF MARYLAND SCHOOL OF MEDICINE


1
MINI MED SCHOOLUNIVERSITY OF MARYLAND
SCHOOL OF MEDICINE
  • HIP DISEASE
  • COMMON IMPEDIMENTS TO WALKING ERECT
  • VINCENT D. PELLEGRINI, JR., M.D.
  • JAMES L. KERNAN PROFESSOR AND CHAIR,
  • DEPARTMENT OF ORTHOPAEDICS
  • email vpellegrini_at_umoa.umm.edu

2
HIP DISEASECOMMON IMPEDIMENTS TO WALKING
ERECT
  • FRACTURE
  • AVASCULAR NECROSIS
  • HIP ATTACK
  • ARTHRITIS

3
HIP DISEASECOMMON IMPEDIMENTS TO WALKING
ERECT
  • LEARNING OBJECTIVES
  • EPIDEMIOLOGY
  • CLINICAL SCENARIO
  • PATHOANATOMY
  • TREATMENT ALTERNATIVES

4
HIP FRACTUREEPIDEMIOLOGY
  • 250,000 HIP FRACTURES ANNUALLY IN USA
  • DOUBLE BY 2040 COURTESY OF BABY BOOMERS
  • 90 ARE LOW ENERGY
  • SIMPLE FALL
  • ELDERLY POPULATION
  • WITH OTHER MEDICAL ILLNESSES

5
HIP FRACTUREEPIDEMIOLOGY
  • US BUREAU OF CENSUS
  • 2030 ONE IN FIVE AMERICANS OVER 65
  • 21 OF FEMALES 19 OF MALES
  • HIP FRACTURE DOUBLES BY DECADE BEYOND 50
  • 1/3 WOMEN AND 1/6 MEN HIP FX BY AGE 90
  • CAUCASIAN FEMALES 2-3x gt HISPANIC AND A-A

6
HIP FRACTURERISK FACTORS
  • URBAN DWELLING
  • ETOH OR CAFFEINE OR TOBACCO
  • PHYSICAL INACTIVITY
  • PRIOR HIP FRACTURE
  • SENILE DEMENTIA
  • CONCURRENT ARTHRITIC DISEASE IS RARE
  • EXERTS A PROTECTIVE EFFECT !

7
HIP FRACTURERISK FACTORS
  • PSYCHOTROPIC MEDICATIONS
  • HYPNOTICS, ANTIDEPRESSANTS, ANTIPSYCHOTICS
  • BENZODIAZEPINES (VALIUM) -gt FALLS
  • WEIGHT LOSS gt 10 WHITE FEMALES OVER 50
  • IN NEED OF ASSISTIVE DEVICES FOR ADLs
  • HOME BOUND AMBULATORS

8
HIP FRACTURERELATED MORTALITY
  • BELLWETHER OF IMPENDING PHYSIOLOGIC DECLINE
  • INJURY - RELATED MORTALITY
  • PERIOPERATIVE EVENTS
  • ONE YEAR MORTALITY FOLLOWING HIP FX
  • 12 - 36
  • IN - HOSPITAL MORTALITY 1 - 2

9
HIP FRACTURERELATED MORTALITY
  • GREATEST RISK
  • INITIAL 4 - 6 MONTHS POSTOPERATIVE
  • MORTALITY RETURNS TO CONTROL AFTER 1 YEAR
  • LOWEST IN THOSE SENIORS WHO ARE
  • COGNITIVELY INTACT
  • INDEPENDENT COMMUNITY DWELLING

10
HIP FRACTURERELATED MORTALITY
  • OPERATIVE DELAY gt 2 DAYS
  • 2X RISK OF DEATH IN FIRST POSTOP YEAR
  • OTHER PREDICTORS OF FIRST YEAR SURVIVAL
  • NUTRITIONAL STATUS
  • GENERAL HEALTH

11
HIP FRACTURETREATMENT CONSIDERATIONS
  • FOR AMBULATORS AND THOSE
  • WITH REASONABLE AMBULATORY POTENTIAL
  • HIP FRACTURE IS AN
  • OPERATIVE DISEASE
  • MINIMIZE MORBIDITY AND MORTALITY ASSOCIATED
  • WITH BEDREST AND INACTIVITY
  • PNEUMONIA / BLOOD CLOTS (DVT AND EMBOLISM)

12
HIP FRACTURESPECIFIC TREATMENT GOALS
  • AMBULATORS
  • SOUND BONY UNION
  • RESTORATION OF NORMAL ANATOMY
  • NONAMBULATORS
  • PAIN - FREE FRACTURE SITE

13
HIP FRACTUREANATOMICAL TREATMENT DICTUM
  • THE LOCATION OF THE FRACTURE
  • RELATIVE TO THE CAPSULAR ATTACHMENTS
  • DETERMINES THE THREAT OF INJURY
  • TO THE BLOOD SUPPLY OF THE HIP,
  • THE METHOD OF TREATMENT,
  • AND ITS SUBSEQUENT LIKELIHOOD OF SUCCESS

14
ANATOMICAL BASIS OF TREATMENTTHE HIP CAPSULE
15
ANATOMICAL BASIS OF TREATMENTTHE HIP CAPSULE
16
HIP FRACTUREVASCULAR SUPPLY TO THE HIP
  • EXTRACAPSULAR
  • CIRCUMFERENTIAL RING OF VESSELS
  • AT THE INTERTROCHANTERIC LINE
  • MEDIAL AND LATERAL FEMORAL CIRCUMFLEX

17
ANATOMICAL BASIS OF TREATMENTBLOOD SUPPLY
TO THE HIP
18
HIP FRACTURETHE THREAT TO VASCULAR SUPPLY
  • RETINACULAR VESSELS
  • PRECARIOUS IN LOCATION ON THE FEMORAL NECK
  • ARE SUBJECT TO INJURY / INTERRUPTION
  • BY ANY INTRACAPSULAR HIP FRACTURE
  • DISPLACEMENT -gt RISK OF DISRUPTION / TAMPONADE

19
ANATOMICAL BASIS OF TREATMENTBLOOD SUPPLY
TO THE FEMORAL HEAD
20
HIP FRACTURESPECIFIC TREATMENT CONSIDERATIONS
  • EXTRACAPSULAR FRACTURE
  • WILL HEAL WITH NONOPERATIVE RX
  • CASTING OR TRACTION
  • BUT, MORBIDITY AND MORTALITY
  • ASSOCIATED WITH PROLONGED BEDREST
  • IS UNACCEPTABLE

21
HIP FRACTUREEXTRACAPSULAR - INTERTROCHANTERIC
  • VASCULAR SUPPLY TO THE HIP IS PRESERVED
  • GENERALLY TREATED BY INTERNAL FIXATION
    (REPAIR OF THE PARTS)
  • FAILURE TO HEAL IS RARE (1-2)

22
HIP FRACTURESPECIFIC TREATMENT CONSIDERATIONS
  • INTRACAPSULAR FRACTURE
  • HEAL ONLY INFREQUENTLY WITH NONOPERATIVE RX
  • DISADVANTAGED BIOMECHANICS
  • COMPROMISED VASCULAR SUPPLY
  • NONDISPLACED FX
  • TREAT BY REPAIR TO MOBILIZE THE PATIENT

23
HIP FRACTUREINTRACAPSULAR - FEMORAL NECK
  • NONDISPLACED FX
  • TREAT BY REPAIR TO MOBILIZE THE PATIENT
  • DISPLACED FX
  • REPLACE THE HIP
  • AVOID NONUNION / NECROSIS
  • SINGLE OPERATION

24
HIP FRACTUREDISPLACED INTRACAPSULAR
  • THE UNSOLVED FRACTURE
  • ?? REPAIR OR REPLACEMENT ??
  • UNDER AGE 60
  • REPAIR / PRESERVATION FEMORAL HEAD
  • OVER 60 gt PROSTHETIC REPLACEMENT
  • IN BETWEEN - CONTROVERSIAL

25
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26
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27
HIP FRACTUREINTRACAPSULAR FX COMPLICATIONS
  • VASCULAR SUPPLY TO THE HIP IS THREATENED
  • ASEPTIC NECROSIS 15 - 33
  • 1 IN 3 REQUIRE ADDITIONAL OPERATION
  • NONUNION / FAILURE OF FIXATION 10 - 30
  • VERTICAL FX PATTERN HIGH RISK
  • 75 REQUIRE REOPERATION

28
ASEPTIC NECROSIS OF THE HIP HIP ATTACK
  • DEATH OF BONE IN THE FEMORAL HEAD
  • SUPPORTING OVERLYING JOINT CARTILAGE
  • REPAIR REPLACEMENT OF THE DEAD BONE
  • TEMPORARY WEAKENED STATE
  • UNDERLYING BONE COLLAPSES HEAD FLATTENS
  • EVENTUAL ARTHRITIS

29
ANATOMICAL BASIS OF TREATMENTBLOOD SUPPLY
TO THE HIP
30
ASEPTIC NECROSIS OF THE HIP EPIDEMIOLOGY
  • TYPICALLY YOUNG PATIENTS lt 50 YRS
  • TWO THIRDS HAVE BOTH HIPS INVOLVED
  • TREATMENT DILEMMA
  • TOO YOUNG / WILL OUTLIVE HIP REPLACEMENT

31
ASEPTIC NECROSIS OF THE HIP RISK FACTORS
DISRUPTION OF CIRCULATION
  • FRACTURE
  • STEROID EXPOSURE
  • EXCESSIVE ALCOHOL CONSUMPTION
  • CHEMOTHERAPY
  • BONE MARROW DISORDERS
  • SICKLE CELL DISEASE
  • LUPUS
  • DEEP SEA DIVING - THE BENDS

32
ASEPTIC NECROSIS OF THE HIP PATHOANATOMY
  • BLOOD SLUDGING IN SMALL VESSELS
  • LOSS OF CIRCULATION ISCHEMIA
  • EDEMA / TISSUE FLUID
  • SWELLING IN SOFT TISSUES
  • INCREASE IN PRESSURE WITHIN THE BONE
  • PREVENTS BLOOD INFLOW VICIOUS CYCLE

33
ANATOMICAL BASIS OF TREATMENTBLOOD SUPPLY
TO THE FEMORAL HEAD
34
ASEPTIC NECROSIS OF THE HIP PATHOANATOMY
  • DEAD BONE MARROW FAT
  • EARLIEST DETECTION BY MRI
  • ANTERIOR, SUPERIOR AND LATERAL HEAD
  • OPPOSITE THE SITE OF ENTRY
  • OF PRIMARY BLOOD VESSELS TO HEAD

35
ASEPTIC NECROSIS OF THE HIP STEROIDS /
ASTHMA MRI
36
ASEPTIC NECROSIS OF THE HIP SYMPTOMS
  • PAIN
  • INCREASED PRESSURE IN BONE
  • DEAD BONE WEAK COLLAPSES WITH OVERLYING
    CARTILAGE
  • LATE ARTHRITIC DISEASE

37
ASEPTIC NECROSIS OF THE HIP TREATMENT
CONSIDERATIONS
  • GOALS
  • RELIEF OF PAIN
  • ENHANCE REPAIR / REVASCULARIZATION DEAD BONE
  • PREVENTION OF LATE ARTHRITIS

38
ASEPTIC NECROSIS OF THE HIP CLASSICAL
TREATMENT
  • CORE DECOMPRESSION
  • DRILL A HOLE IN FEMUR TO RELIEVE PRESSURE
  • RELIABLE RELIEF OF PAIN
  • UNCHANGED PROGRESSION OF DISEASE
  • EVENTUAL COLLAPSE OF DEAD BONE
  • LATE ARTHRITIS AND HIP REPLACEMENT
  • 10 - 30 RETENTION OF NORMAL HIP AT 5 YRS

39
ASEPTIC NECROSIS OF THE HIP CONTEMPORARY
TREATMENT
  • VASCULARIZED BONE GRAFT (FIBULA)
  • RELIEVE PRESSURE IN FEMUR
  • REDIRECT NEW CIRCULATION
  • PROVIDE SUPPORT TO PREVENT COLLAPSE
  • TWO SURGICAL TEAMS
  • ONE HARVEST FIBULA FROM SAME SIDE LEG
  • ONE PREPARE HIP AND MICROSURGERY

40
ASEPTIC NECROSIS OF THE HIP VASCULARIZED
FIBULA GRAFT
41
ASEPTIC NECROSIS OF THE HIP VASCULARIZED
FIBULA GRAFT
42
ASEPTIC NECROSIS OF THE HIP VASCULARIZED
FIBULA GRAFT
43

VASCULARIZED FIBULA FOR HIP AVN STEROIDS / MS
- 8 WEEKS
44
VASCULARIZED FIBULA FOR HIP AVN STEROIDS / MS
- 6 YEARS
45
ASEPTIC NECROSIS OF THE HIP TREATMENT
RESULTS
  • IF OPERATION BEFORE COLLAPSE OF HEAD
  • VASCULARIZED BONE GRAFT (FIBULA)
  • 80 - 85 RETENTION OF NORMAL HIP AT 5 YRS
  • 15 CONVERSION TO TOTAL HIP REPLACEMENT

46
VASCULARIZED FIBULA FOR HIP AVNMECHANISM OF
FAILURE
47
ARTHRITIS OF THE HIP EPIDEMIOLOGY
  • MANY DIFFERENT CAUSES
  • POST TRAUMA
  • OSTEOARTHRITIS (WEAR AND TEAR)
  • RHEUMATOID ARTHRITIS (INFLAMMATORY)
  • ASEPTIC NECROSIS
  • DEVELOPMENTAL / ADOLESCENT
  • CONGENITAL

48
ARTHRITIS OF THE HIP PRESENTATION
  • GROIN PAIN
  • REFERRED PAIN TO THIGH AND KNEE
  • UNABLE TO FULLY STRAIGHTEN / EXTEND
  • STOOPED PAINFUL GAIT
  • STIFFNESS / PARTICULARLY LOSS OF ROTATION
  • CONSTANT PAIN AT REST NIGHT

49
ARTHRITIS OF THE HIP TREATMENT
  • ANALGESIC MEDICATION
  • ANTI-INFLAMMATORY MEDICATION
  • CANE (OPPOSITE HAND)
  • WEIGHT LOSS
  • OSTEOTOMY (CUT REPOSITION HIP)
  • FUSION (STIFFENING)
  • TOTAL HIP REPLACEMENT

50
ANATOMICAL BASIS OF TREATMENTINSIDE THE HIP
JOINT - CARTILAGE WEAR
51
ARTHRITIS OF THE HIP XRAYS - LOSS OF
CARTILAGE SPACE
52
TOTAL HIP REPLACEMENT OUTCOMES
CEMENTLESS FIXATION LOOSENING RARE 90
SURVIVE 15 YRS MOST REVISIONS FOR SOCKET
WEAR
53
PROSTHETIC HIP REPLACEMENT MECHANISMS OF
FAILURE
INSTABILITY INFECTION WEAR OF THE BEARING
SURFACES BONE RESORPTION
54
PROSTHETIC HIP REPLACEMENT THE FUTURE
IMPROVED BEARING SURFACES NEWER
MATERIALS CERAMIC / METAL / PLASTIC REDUCED
REACTION TO WEAR DEBRIS PREVENT BONE
RESORPTION RESPONSE ? MINI INCISIONS ? NOT
LIKELY THE ANSWER
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