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Respiratory System part 2 second years student Nursing Collage

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Title: Respiratory System part 2 second years student Nursing Collage


1
Respiratory Systempart 2second years
studentNursing Collage
  • Iman Al Shaweesh
  • Sept. 2008
  • Al Najah Univesity

2
Health History
  • The reason that pt. is seeking health care is,
    dyspnea (shortness of breath), hemoptysis (blood
    spit up from respiratory tract), odema, cough,
    general fatigue, weakness.
  • Nurse must identify chief complain , when started
    problem ,how long, duration, severity, assess
    risk factors, identify the impact of ss on the
    patient ability to perform daily activitie.

3
  • Major ss are dyspnea, cough, wheezing, sputum
    production, chest pain, clubbing of fingers,
    hemoptysis, cyanisis. This SS are related to
    duration severity of disease.

4
1-Dyspnea
  • difficult or labored breathing, shortness of
    breath. Symptom common to many pulmonary
    cardiac disorders, particularly when there is
    decrease lung compliance or increase airway
    resistance.
  • Rt. Ventricle will affect by pulmonary disease
    because it must pump blood through the lung
    against greater resistance.

5
  • Sudden dyspnea in health person indicates.
  • Pneumothorax (air in pleural cavity).
  • Pulmonary embolism
  • RDS.
  • Acute respiratory obstruction.

6
Orthopnea
  • (inability to breath easily except in an upright
    position). Found in pt. with COPD, heart disease.
  • Dyspnea with an expiratory wheeze occurs with
    COPD.
  • Noisy breathing result from narrowing of the
    airway or localized obstruction of major bronchus
    by tumor or foreign body.

7
Relieve measures
  • high fowlers position, 02 in sever case.
  • Ask pt. circumstance that produces dyspnea.
  • How much exertion triggers shortness of breath?
  • Cough?
  • Time of day or night? Occur at rest.
  • Shortness sudden or gradual.

8
2-Cough
  • Result from irritation of the mucous membranes
    any where in the respiratory tract.stimulus many
    arise from an infectious process or irritant as
    smoke, dust, gass.its protect against
    accumulation of secretion in the bronchi
    bronchioles.

9
Clinical manifestation
  • Cough may indicate serious pulmonary disease.
  • Nurse must evaluate character of cough, dry,
    loose, sever, brassy, hacking.
  • A dry, irritant cough in characteristics of an
    URTI of viral origin or may be S.E of (ACE)
    inhibitor therapy.
  • Laryngotracheitis cause an irritative high
    pitched cough.

10
  • Tracheal lesions produce a brassy cough.
  • A sever or changing cough may indicate
    bronchogenic carcinoma.
  • A cough in the morning with sputum production may
    indicate bronchitis.
  • A cough that worsen when the pt. is in supine
    position suggest postnasal drip (sinusitis).
  • Coughing after food intake may indicate
    aspiration of material into the tracheobronchial
    trea.

11
3-sputum production
  • pt. who coughs long enough produce sputum.
  • Violent coughing cause bronchial spasm
    obstruction irritation of bronchi and may
    result in scope (funting).
  • Uncontrolled cough that is non productive is
    exhausting potentially harmful.

12
Clinical manifestation
  • Thick, yellow, green..Indicate bacterial
    infection.
  • Thin, mucoid sputum......Indicate viral.
  • Pink tinged mucoid sputum Indicate lung
    tumor.
  • Profuse, frothy, pink material ...Indicate
    pulmonary odema.
  • Foul-smelling sputum bad breath .Indicate
    lung abscess, infection from anaerobic organism.

13
Relieve measure
  • Adequate hydration (water).
  • Inhalation of nebulizer.
  • Stop smoking, because it causes inflammation
    hyperplasia of mucous, and decrease production of
    surfactant.
  • If smoking stop sputum will decrease, encourage
    pt. to drink juices to change sputum taste
    select of food that increase the appetite.

14
4-chest pain
  • may associated with pulmonary or cardiac disease.
  • Chest pain with pulmonary may be sharp stabbing
    intermittent or dull, aching persistant
  • Pain may refer else where- neck, back, abdomen.

15
Clinical manifestation
  • Chest pain may occur with pneumonia, pulmonary
    embolism with lung infarction pleurisity.
  • It also late symptom of bronchogenic carcinoma.
  • Lung disease not always produce pain because lung
    visceral pleura lack sensory nerves
    insensitive to pain stimuli partial pleura has
    rich supply of sensory nerves.

16
  • Pleuritic pain from irritation of partial pleura
    is sharp, pt. describe it as (like the stabbing
    of knife). Pt. comfortable when sleep on affected
    part.
  • Nurse must assess quality, intensity, radiation
    of pain, relationship of pain to inspiratory
    expiratory.

17
  • Relieve measures
  • Analgesic medication but not to depress the
    respiratory center or productive cough.
  • NSAID for pleurituic pain.
  • Regional anesthetic block may be performed to
    decrease extreme pain.

18
5-wheesing
  • Is often a major finding in pt. with
    bronchoconstriction or airway narrowing.
  • Heard with stethoscope depend on location.
  • Wheezing is a high pitched, musical sound heard
    mainly on expiration.
  • Relieve measure Oral or inhaled bronchodilator.

19
6-clubbing of the fingers
  • is singe of lung disease found in pt. with
    chronic hypoxic condition, chronic lung
    infections, CA of lung.
  • Initially manifested as sponginess the nail bed
    loss of nail bed angle

20
7-Hemoptysis
  • Expectoration of blood from respiratory tract.
  • Is symptom of both pulmonary cardiac disorders.
  • Onset is sudden, may be intermittent or
    continious.
  • Common cause 1- pulmonary infection.
  • 2- CA of lung.
  • 3- Abnormalities of
    heart
  • blood vesserls.

21
  • 4- Pulmonary artery or vein
    abnormalities.
  • 5- Pulmonary emboli infarction.
  • DX. Evaluation
  • Chest x-ray.
  • Chest angiography.
  • Bronchoscopy
  • Full history physical examination

22
  • Inspection of blood, small amount or massive
    hemorrhage. Source of bleeding gums( blood
    appearing in noise).
  • Lung (bright red, frothy salty taste haemoptesis.
  • Stomach(haemoptesis dark blood).

23
8-Cyanosis
  • Is a bluish coloring of skin, very late indicator
    of hypoxia.
  • Presence or absence of cyanosis is determined by
    amount of unoxygenated hemoglobin in blood. When
    there is 5g/dl of unoxygenated HG. A pt. with
    15g/dl HG will not demonstrate cyanosis until
    5g/dl of HG become unoxygenated.
  • Cyanosis is not reliable singe of hypoxia.
    Because anemic pt. rarely manifest cyanosis.

24
  • In the present of pulmonary condition cyanosis
    assess by tongue lips.
  • Peripheral cyanosis results from decrease blood
    flow to certain area not indicate central
    problem.

25
Physical assessment of upper respiratory tract
  • Noise sinuses
  • Inspect external nose for lesion, asymmetry or
    inflammation .ask pt. to tilt head background,
    gently pushing tip of nose inspect mucosa for
    color, swelling, bleeding.nasal Deviation,
    Perforation.
  • Nurse inspect inferior middle turbinates then
    nurse palpate frontal maxillary sinuses for
    tenderness using thumb gently pressure. Frontal
    maxillary sinuses can be inspected by
    (transilliumination). If light fails to generate,
    the cavitycantain fluid.

26
  • Pharynx mouth
  • Ask pt to open mouth take breath inspect for
    color, symmetry, ulceration or enlargement.
  • Trachea.(position, mobility of trachea).

27
Assessment of lower respiratory structures
breathing
  • Thorax Inspection of thorax provide information
    about musculoskeletal structure, nutritional
    status respiratory system. Note symmetry.
  • Chest configuration. Normal ratio of
    anterioposterior diameter to lateral diameter is
    12. Four deformities of chest associated with
    respiratory disaease.

28
  • A-Barrel chest.
  • Result of overinflation of the lungs
  • Increase in anterioposterior diameter of thorax
  • Pt. with emphysema, ribs are more wildely spaced
    intercostals spaces tend to blug on expiration.

29
Barrel chest
30
  • B-Funnel chest (pectus excavatum)
  • Occur where is depression in lower portion of
    sterum this may compress heart great vessels
    resulting in murmers.
  • May occur with rickets or marfans syndrom

31
  • C-Pigeon chest (pectus carinatum).
  • Occur as result of displacement of sternum.
  • Increase anterioposterior diameter.
  • Rickets , marfans syndrome, sever hyposkoliosis.

32
  • D-kyphoscoliosis.
  • Is characterized by elevated of scapula
    corresponding S- shape spine.
  • This limit lung expantion.
  • Occure with osteoporosis.

33
Breathing pattern respiratory rates
  • Normal breath 12-18 b/m, regular in depth
    rythem. This descripe as eupnea.
  • Bradycardia slow breathing associated with
    increase ICP , brain injury, drug overdose.
  • Tachypnea- rapid breathing. Pt with pneumonia,
    metabolic acidosis, pulmonary odema, septicemia.
  • Hyperventilation shallow, irregular breathing.
    hyperventilation associated with sever acidosis
    is called ( kussmauls respiration

34
  • Hyperpnea increase depth of respiration.
  • Hyperventilation increase in rate depth .
    inspiration expiration are equal in duration.
  • 1-Thoracic palpation
  • The nurse palpates the thorax for tenderness,
    masses, lesion, vocal fremitus, and respiratory
    excursion. Nurse performs direct palpation with
    finger tips or ball of the hand (for deeper
    masses).

35
  • A-Respiratory excursion
  • Is an estimation of thoracic expansion may
    dissolve significant information about thoracic
    movement during breathimg.
  • Pt. instructed to inhale deeply while the
    movement of nurse thumbs. This movement is
    normally symmetry. Asymmetric excursion due to
    fracture rib, trauma, unilateral bronchial
    obstruction.
  • Decrease chest excursion due to chronic fibrotic
    disease.

36
  • B-Tactile fremitus
  • Sound generated by the larynx travels distally a
    lone the bronchial tree to test the chest wall in
    resonant motion. Pt asks to repeat 99.
  • Pt. with emphesema- rupture of aloveoli
    trapping of air, exhibit no tactile fremitus.
  • Tactile fremitus increase over lob affected with
    pneumonia.

37
  • C-Thoracic percussion
  • Nurse use percussion to determine a whether
    underlying tissues are filled with air, fluid or
    solid material.also used to estimate size,
    location certain structure with thorax. (heart.
    Diaphragm).

38
  • D-diaghragmatic excusion
  • Normal resonance of the lung stops at the
    diaphragm, the position of diaphragm is different
    in inspection expiration.
  • Assess position motion of diaphragm by
    ask pt. to take breath hold it, nurse
  • marked with pen. Distance between the two
    markings indicates range of motion.
  • 8-10cm in healthy, 5-7 most people. The
    diaphragm is about 2cm. higher in Rt.
  • Because position of heart.

39
DX.Evaluation
  • Pulmonary function test. Routinely used in pt.
    with chronic respiratory disorders, test should
    measurements of lung volume, ventilatory function
    mechanism of breathing diffusion gas
    exchange.
  • Arterial blood gas study. Aid in assessing the
    ability of the lungs to provide adequate o2
    remove co2. the ability of kidney to reabsorb
    excrete bicarbonate ions to maintain body normal
    PH. ABGs are obtained through an arterial
    puncture at radial, brachial, femoral, or
    indwelling arterial catheter.

40
  • Pulse oximetry. Non invasive method of
    continuously monitoring o2 sat. of hemoglin.
    Aprobe or sensor is attached to the fingertip
    forehead, earlobe, bridge of nose. Normal
    95-100, decrease 85 indicate that tissue are not
    receiving enough o2.
  • Culture. Throat culture to identify pathogenic
    organism, drug sensitivity testing. Specimen to
    lab must be within 2 hr.s (overgrowth of
    organisim). Specimen taken at morning.

41
  • Imagining studies. Include X- ray, CT, MRI
    (magnetic resonance imagimg, radioscopic
    diagnostic scans.
  • 1-Chest x-ray normal pulmonary tissue is
    radiolucent, there for densties produced by
    fluid, tumors, foreign bodies pathogenic
    condition can be detected by x-ray.
  • 2-CT used to identify pulmonary nodules small
    tumors that are not visible on routine chest
    x-ray.

42
  • 3-MRI are more diagnostic image than CT..to
    characterized pulmonary bodules.
  • 4-Fluoroscopic studies used to assess with
    invasive procedures as chest needle biopsy. To
    study movement of chest wall, heartr,
    diaphragm.To detect , diaphragm paralysis lung
    masses.

43
  • 5-Pulmonary angiography used to investigate
    thromboembolic disease of the lung as congenital
    abnormalities of pulmonary vascular tree.( it
    involve rapid injection of radiopaque agent (
    from femoral vein, arm vein) into the vascular of
    the lungs for radiographic study of pulmonary
    vessels

44
  • 6-Radioisotope DX. Procedures (lung
    scan).Ventilation- perfusion lung scan is first
    performed by injecting a radioactive agent into a
    peripheral vein then obtaining a scan of the
    chest into detects radiation.Used clinically to
    measure the integrity pf pulmonary vessels
    relative to blood flow to evaluate blood flow
    abnormalities as seen in pulmonary emboli. It
    takes 20-40 minute.

45
VI.Endoscopic procedure
  • Bronchoscopy is direct inspection examination
    of the larynx, trachea, bronchi through either
    a flexible fiberoptic scope or rigidf
    bronchoscope.
  • Purpose of DX. Bronchoscopy.
  • To examine tissue or collect secreations.
  • To determine location obtain tissue.
  • To determine if tumor can be resected surgically.
  • To DX. Bleeding site.

46
  • Thepaputic bronchoscopy is used to
  • Remove foreign bodies.
  • Remove recreation obstruction the
    tracheobronchial tree.
  • Treat post operative atelactasis.
  • Destroy excise lesion.

47
  • Complication
  • Reaction to local anesthesia.
  • Infection
  • Aspiration
  • Bronchoscopy
  • Hypoxemia

48
  • NSG intervention
  • consent form, NPO, explain procedure,
    preoperative medication ( atropine,
    sedation)..as prescribed to inhibit vagal
    stimulation, suppress cough. Also pt. must remove
    dentures.
  • Post op. pt. must be NPO until the cough reflex
    returns. Ice-chips fluid given.
  • Observe v/s, hypoxia, bleeding, hypotention,
    tachycardia, dysarythmia.

49
Thoracoscopy
  • (DX. Treat biobsy).
  • Is DX procedure in which the pleural cavity is
    examined with an endoscope. Small incisions are
    made into the pleural cavity.
  • Chest tube may be inserted pleural cavity is
    drained by negative- pressure water seal drainage.

50
  • NSG intervention
  • Monitor shortness of breath (which may indicate
    pneumothorax).
  • Monitor chest drainage if chest tube is in place.
  • Monitor activity restriction.

51
Thoracenteses.
  • A thin layer of pleural fluid normally remains in
    the pleural space. An accumulation.
  • may occur in some disorders. Sample can obtain by
    (aspiration of pleural fluid for DX. To
    therapeutic purposes).
  • Needle biobsy can be performed at same time.
    Study includes grams stain culture sensitivity,
    acid- fast staining, PH..

52
Biopsy
  • The excision of small amount of tissue, may be
    performed to permit examination of cells from the
    pharynx, larynx.
  • Pleural biopsy is accomplished by needle biopsy
    of the pleura or by pleuroscipy. Visual
    exploration through a fiberoptic bronchoscopy.
  • Lung biobsy procedures is performed to obtain
    lung tissue for examination to identify the
    nature of lesion.

53
  • There are several non-surgical technique as
  • transcantheter bronchial brushing
  • trans brachial lung biopsy
  • percutanous.
  • NSG intervention same as bronchoscopy, monitor
    shortness of breath, bleeding, infection, report
    pain, redness of biopsy, site, pus..
  • Lymph node biopsy scalene node biopsy may
    performed to detect lymph spread of pulmonary
    disease as hodgkins carcinoma.

54
Management of pt.s with upper respiratory tract
disorder
  • 1-Rhinitis is a group of disorder characterizes
    by inflammation irritation of the mucous
    membranes of the nose. It may be classified as
    non allergic allergic.
  • Patghophysiolopgy
  • Non allergic rhinitis may be caused by a variety
    of factors including.
  • Environmental factors such as change temp.
    Humidity odors, foods, infection, age, systemic
    disease, drugs (cocaine). Or prescribed
    medication, foreign body.

55
  • SS
  • Rinorrhea (excessive nasal drainage, runny
    nose), nasal congestion, discharge itching,
    sneezing, and headach.
  • RX.
  • Depend on cause if viral, medication given to
    reduce symptoms.
  • In allergic rhinitis tests may perform,
    corticosteroid desensitizing immunization may
    require.

56
  • If bacterial infection- antimicrobial agent.
  • Antihistamine for allergy for sneezing, itching,
    rhinrrhea.
  • Oral decongestant agent.

57
  • NSG Manegement.
  • Avoid or decrease exposure to allergens
    irritants.
  • Controlling environment.
  • Technique of administer nasal medication.
  • Hygiene, blow the nose before medication.
  • Treat symptom.
  • In elderly, nurse discusses value of vaccine in
    the fall to achieve immunity prior the beginning
    of flu season.

58
2-Viral rhinitis (common cold).
  • Is used when referring to an URTI is self-
    limited caused by a virus (viral rhinituis).
  • nasal congestion, rhinorrhea, sneezing, sore
    throat general malaise characterize it.
  • Cold are highly contagious because virus is shed
    for about two days befot the symptoms appear.

59
  • The 6 viruses known to produce SS of viral
    rhinitis are rhiniuvirus, parainfluenza virus,
    corona virus, resp. syncytial virus influenza,
    adenovirus. Each one have multiple strain.(e.g
    100 strains of rhinivirus, which account of 50
    of al colds.

60
  • SS
  • Nasal congestion, runny nose, sneezing, nasal
    discharge, nasal itching, tearing watery eyes,
    scratchy or sore throat, general malaise.
  • Low grade fever, chills, headache, muscle aches.
  • In some people it exacerbates to herpes simplex.
  • Symptom last 1-2 wk.s.

61
Treatment
  • Consist of symptomatic therapy, no specific
    treatment.
  • Adequate fluid intake.
  • Rest, increase intake of vitamin C, use
    expectorant.
  • Warm salt water gargles
  • NSAID as aspirin, ibuprofen relieves the aches,
    pain, fever.

62
  • Some research suggests Zink lozenges, may
    decrease duration of cold symptoms if taken
    within 1st 24 hr.s of onset.
  • Antibiotic should not be used.
  • NSG Manegement
  • Teach pt. how to break the chain of infection,
    hand washing (most effective).
  • Teach method to treat symptom prevent measure.

63
3-Acute sinusitis
  • The sinuses, mucus lined cavities filled with air
    that drain normally into the nose, are involved
    in a high proportion of URTI.
  • If opening to nasal are clear, the infection
    resolve promptly.
  • Some individual are more prone to sinusitis
    because of their occupation as paint.

64
  • Patghophysiolopgy
  • Acute sinusitis is an infection of the Para nasal
    sinuses.
  • It frequently developa as aresult of an URTI.
  • Nasal congestion caused by inflammation, odema,
    transudation of fluid, lead to obstraction of the
    sinus cavity.
  • This provide an excellent modium for bacterial
    growth.
  • .

65
  • Bacterial organism account for 60 of acute
    sinusitis as streptococcus pneumonia, hemophilus
    influenza, moraxella catarrbalis.
  • Dental infection associated with acute sinusitis

66
  • SS
  • Facial pain, nasal obstruction, fatigue,
    purulent nasal discharge, fever, headache, facial
    ear pain fullness, dental pain, cough, decrease
    sense pf smell, sore throat, eyelid edema.

67
Assessment DX finding
  • History physical examination.
  • Tenderness over the infected sinus
    area.transilluminated (decrease transmission of
    air).
  • Sinus x-ray. (Fluid level, mucisal thikining).
  • Computed tomograghy scanning is most effective DX
    tool.

68
  • Complication
  • If not treated lead to meningitis brain abscess,
    ischemic infarction, osteomyelitis, sever orbital
    cellulites.
  • RX.
  • Goal to treat infection, shrink the nasal mucosa
    relieve pain.
  • Antimicrobial, first lineamoxicillin,
    erythromycin, bactrim.

69
  • Mucolytic agent, decease nasal congestion.
  • Antihistamine
  • If pt. continues to have symptom 7-10 days, the
    sinuses may need to be irrigated hospital may
    be required.
  • NSG Management
  • Teach method to promot drasinage as inhaling
    system, increase fluid intake, local heat.

70
  • Inform pt. about S.E of nasal spray body
    receptors depened on spray to keep nasal passage
    open. (rebond congestion).
  • Teach early signe of a sinus infection
    recommended preventive measures as following
    health practices, avoid contact with people have
    URTI.
  • Explain the fever nuchal rigidity, sever headach
    as signe of potential complication.

71
Chronic Sinusitis
  • Is an inflammation of sinuses that persists for
    more than 3 wk.s in adult 2 wk.s in children.
    Estimated that 32 millions develop/year.
  • Is an inflammation of sinuses that persists for
    more than 3 wk.s in adult 2 wk.s in children.
    Estimated that 32 millions develop/year.
  • Blockage that persist for greater 3 wk. may occur
    because of infection, allergy, or structural
    abnormalities.

72
  • This result in stagnant recreation, an ideal
    medium for infection.
  • Organism as acute immunocompremized at risk of
    fungal.

73
Clinical Manifestation
  • Impaired mucocilliary clearance ventilation,
    cough, chronic hoarseness, chronic headach in
    periorbital area, facial pain.
  • Fatigue, nasal stuffness, decrease smell taste,
    fullness in the ears.
  • Symptoms are generally pronounced on awakening.

74
DX finding assessment
  • History, computed tomography scan or MRI if
    fungal suspected.
  • Rule out other systemic disorders as tumor.
  • Nasal endoscopy.
  • Complication
  • Uncommon.
  • sever orbital cellulites
  • meningitis, encephalitis
  • cavenous sinus thrombosis

75
Treatment of chronic sinusitis
  • As acute course 3-4 wk.s)
  • Surgical management when medical failed incision
    drainage sinus, removing tumor, correct
    structural deformities (excise fungal ball
    necrotic tissue drain sinus).
  • Topical corticosteroid, antimicrobial agents are
    administer before after surgery.

76
  • NSG Management
  • Nurse teaches pt. how to promote sinus drainage
    at home by increase envoromental humidity.
  • Increase fluid, follow medication regime.
  • Instrucr signe of sinus infection.

77
4-Acute Pharyngitis
  • Is an inflammation or infection in the throat
    usually causing symptoms of a sore throat.
  • Pathophysiology
  • Most cases of acute pharyngitis are caused by
    viral infection.
  • When group.A beta-hemolytic streptococcus- most
    common cause acute pharyngitis the condition know
    as strep throat.

78
  • Body response by triggering an inflammatory
    response in the pharynx, this results in pain,
    fever, vasodilation, edema, tissue damage,
    manifested by redness swelling in the tonsillar
    pillars, uvula soft palate, creamy exudates may
    be presented in tonsillar pairs.
  • If a beta- hemolytic streptococci is a more sever
    illness.

79
  • Complication include, sinasitus, OM,
    peritonsillar abscess, mastoiditis cervical
    adenitis.
  • In rare case meningitis, pneumonia.
  • SS
  • Afiery red pharyngitis membrane tonsils
    lymphoid follicles those are swollen flacked
    with white purple exudates. Enlarge tender
    cervical lumph nodes.
  • Fever, malaise, sore throat may present.

80
Assessment DX.
  • Latex agglutination (LA) / strep antigen
  • ELISA
  • Throat culture
  • Nasal swab blood culture
  • RX
  • Viralsupportive measures
  • Bacterial.. penicillin, erythromycin,
    cephalosporin (10 day).
  • analgesia, aspirin, acamol.
  • Antitussive med. Codian
  • Nutritional therapy, liquid, soft, in sever case
    IV

81
NSG Management
  • Stay in bed during febril stage.
  • Disposed of tissue to decrease spread infection.
  • Examin skin for rash, because pharungitis may
    precede some communicable disease as rubella.
  • Warm saline gargles or irrigation.
  • Ice collar can relieve sever throats.

82
  • Mouth care.
  • Full course of antibiotics, A beta-hemolytic
    strep. Infection possible develop complication as
    nephritis, RF, which may have onset 2-3 wk.s of
    pharyngitis.
  • Nurse must instruct the important of take full
    course of med.)

83
Chronic Pharyngitis
  • Is a persistent inflammation of the pharynx, its
    common in adults who work or live in dusty
    surrounding, use their voice to excess, suffer
    from chronic cough, use alcohol.
  • Three type of chronic pharyngitis
  • Hypertrophic general thickening congestion of
    the pharyngeal mucusemembrane.
  • B. Atrophic late stage of type one membrane is
    thin, whitish, glistening.

84
  • C. Chronic granular(clergymanas sore throat),
    characterized by numerous swollen lymph follicles
    on the pharynx.
  • SS
  • Pt. sense, constant sense of irritation or
    fullness on throat.
  • Mucous that collects in the throat can be
    expelled by coughing difficulty swallowing.

85
Treatment
  • Rx based on relieving symptoms avoid exposure to
    irritants.
  • Nasal congestion- nasal spray contain ephedrine
    sulfate
  • Aspirin or acetaminiphil.
  • Analgesic, antihistamin q 4-6 hr.

86
NSG Management
  • Avoid contact with other.
  • Alcohol, tobacco, smoke, exposure to cold are
    avoided.
  • Drink plenty of fluids
  • Gargelling with worm saline may relieve throat
    discomfort.
  • Lozenges will keep throat moistened.
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