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The Red Eye

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Title: The Red Eye


1
The Red Eye
  • ByCharlise A. Gunderson, M.D.Assistant
    ProfessorDepartment of Ophthalmology

2
Goals
  • Review the anatomy of the eye
  • Recognize common causes of the red eye
  • Be able to diagnose the causes of a red eye
  • Know when to refer a patient with a red eye to an
    ophthalmologist

3
  • Practioners are often confronted with a patient
    who presents with the red eye. The practioner
    must make a diagnosis and decide if referral to
    an ophthalmologist is necessary and whether or
    not the referral is urgent.

4
Review of Ocular AnatomyPicture taken from Basic
Ophthalmology for Medical Students and Primary
Care Residents published by the American Academy
of Ophthalmology
5
Eyelid anatomy

6
Lacrimal system and eye musculaturePicture taken
from Basic Ophthalmology for Medical Students and
Primary Care Residents published by the American
Academy of Ophthalmology
7
Useful tools to aid in diagnosis near vision
card, penlight with blue filter, topical
anesthetic, fluorescein strips
8
Possible Causes of a Red Eye
  • Trauma
  • Chemicals
  • Infection
  • Allergy
  • Systemic Infections

9
Symptoms can help determine the diagnosis
  • Symptom Cause
  • Itching allergy
  • Scratchiness/ burning lid, conjunctival, corneal
  • disorders, including
  • foreign body, trichiasis,
  • dry eye
  • Localized lid tenderness Hordeolum, Chalazion

10
Symptoms Contd
  • Symptom Cause
  • Deep, intense pain Corneal abrasions, scleritis
  • Iritis, acute glaucoma, sinusitis
  • Photophobia Corneal abrasions, iritis, acute
  • glaucoma
  • Halo Vision corneal edema (acute glaucoma,
  • contact lens overwear)

11
Diagnostic steps to evaluate the patient with the
red eye
  • Check visual acuity
  • Inspect pattern of redness
  • Detect presence or absence of conjunctival
    discharge and categorize as to amount (scant or
    profuse) and character (purulent, mucopurulent,
    or serous)
  • Inspect cornea for opacities or irregularities
  • Stain cornea with fluorescein

12
Diagnostic steps continued
  • Estimate depth of anterior chamber
  • Look for irregularities in pupil size or reaction
  • Look for proptosis (protrusion of the globe), lid
    malfunction or limitations of eye movement

13
How to interpret findings
  • Decreased visual acuity suggests a serious ocular
    disease. Not seen in simple conjunctivitis
    unless there is corneal involvement.
  • Blurred vision that improves with blinking
    suggests discharge or mucous on the ocular surface

14
Checking Vision
  • Checking visual acuity in the pediatric group can
    be very challenging and may not be practical in
    the pediatricians office for nonverbal children.
  • If the child is verbal and cooperative, several
    methods are available

15
Checking Vision Contd
  • Available methods
  • Snellen letters
  • Tumbling E
  • HOTV
  • Allen pictures

16
These are examples of Allen figures. It is not
important what the child calls the figure but
they must be consistent ie bird figure is often
called a dinosaur
17
Tumbling Es. Instruct the child to hold one hand
with the fingers pointing in the same direction
as the legs of the E or it may be easier to
describe it as the legs of the table.
18
Checking Vision Contd
  • Teach the child the tumbling E, HOTV, or Allens
    by allowing the child to look at the larger
    figures with both eyes open
  • Test each eye individually making sure that the
    other eye is completely occluded
  • Test the affected eye first to make sure that you
    have good attention and that the child does not
    tire

19
Pattern of Redness
20
Ciliary flush injection of deep conjunctival
vessels and episcleral vessels surrounding the
cornea. Seen in iritis (inflammation in the
anterior chamber) or acute glaucoma. Not seen in
simple conjunctivitisPicture taken from Basic
Ophthalmology for Medical Students and Primary
Care Residents published by the American Academy
of Ophthalmology
21
Conjunctival hyperemia engorgement of more
superficial vessels. Nonspecific sign.Picture
taken from Basic Ophthalmology for Medical
Students and Primary Care Residents published by
the American Academy of Ophthalmology
22
Corneal opacities
  • Three types of corneal opacities
  • Keratic precipitates
  • Diffuse haze
  • Localized opacities

23
Keratic precipitates are cellular deposits on the
corneal endothelium and result from iritis
(inflammation in the anterior chamber) Picture
taken from Basic Ophthalmology for Medical
Students and Primary Care Residents published by
the American Academy of Ophthalmology
24
Diffuse haze corneal edema or swelling,
frequently seen in angle closure glaucoma. Note
the indistinct margins of the corneal light
reflex. Picture taken from Basic Ophthalmology
for Medical Students and Primary Care Residents
published by the American Academy of Ophthalmology
25
Localized opacities may be due to keratitis
(corneal inflammation) or ulcer (localized
corneal infection) Picture taken from Basic
Ophthalmology for Medical Students and Primary
Care Residents published by the American Academy
of Ophthalmology
26
Methods of checking corneal epithelial disruption
  • Observe reflection from the cornea with single
    light source (ie penlight) as patient moves eye
    in various positions. Disruptions cause
    distortion and irregularity of reflection
  • Apply fluorescein to the eye and breaks in the
    epithelium will stain bright green when viewed
    with a cobalt blue light

27
Corneal epithelial defects outlined by
fluorescein when viewed with a cobalt blue light
(many penlights have a blue cap that can be
placed over them or some direct ophthalmoscopes
have a blue light). Picture taken from Basic
Ophthalmology for Medical Students and Primary
Care Residents published by the American Academy
of Ophthalmology
28
Pupillary abnormalities
  • In iritis spasm of the iris sphincter muscles may
    cause the pupil to be smaller in the affected eye
    or may be distorted due to inflammatory
    adhesions.
  • Pupil is fixed and mid-dilated in acute angle
    closure glaucoma
  • The pupil is unaffected in conjunctivitis

29
Anterior Chamber Depth EstimationPicture taken
from Basic Ophthalmology for Medical Students and
Primary Care Residents published by the American
Academy of Ophthalmology
30
  • Try to compare the anterior chamber depth of the
    two eyes
  • A narrow anterior chamber suggests angle closure
    glaucoma
  • Angle closure glaucoma is unusual in children,
    but may be seen in children with retinopathy of
    prematurity

31
Proptosis
  • Forward displacement of the globe
  • Sudden proptosis suggests serious orbital or
    cavernous sinus disease
  • In children, orbital infection or tumor must be
    ruled out
  • May be accompanied by conjunctival hyperemia or
    limitation of ocular movement

32
The proptotic eye appears larger than the normal
eye with more of the white sclera showing.

33
Red Eye Disorders An Anatomical Approach
  • Lids
  • Orbit
  • Lacrimal System
  • Conjunctivitis
  • Cornea
  • Anterior Chamber

34
Lid Disorders
  • Hordeolum/Chalazion
  • Blepharitis

35
Hordeolum/Chalazion
  • Usually begins as diffuse swelling followed by
    localization of a nodule to the lid margin
  • Hordeolum staphylococcal infection of the
    glands of Zeis
  • Chalazion obstruction of the meibomian glands

36
Hordeolum/Chalazion Treatment
  • In children surgical excision often requires a
    general anesthetic in the operating room
    therefore, extended trials of conservative
    therapy are warranted
  • Treatment includes warm compresses and topical
    antibiotic drops or ointment four times a day.
    Antibiotics should be continued for 3-4 days
    after spontaneous rupture to prevent recurrence

37
Hordeolum/Chalazion Treatment Contd
  • Lesions present for more than a month seldom
    resolve spontaneously and should be referred to
    an ophthalmologist on a non-urgent basis if no
    resolution with conservative management
  • Systemic antibiotics should only be used if the
    hordeolum or chalazion becomes secondarily
    infected

38
The nodule on the patients right upper lid is a
chalazion.
39
Blepharitis
  • Chronic inflammation of the lid margin
  • Types staphylococcal or seborrheic
  • Symptoms foreign-body sensation, burning,
    mattering
  • May predispose to chalazia, blepharoconjunctivitis
    , loss of lashes

40
Blepharitis note the crusting in the lashes and
the thickened lid margin
41
Blepharitis Treatment
  • Warm compresses
  • Lid scrubs with 50/50 mixture of nonirritating
    shampoo (Johnson and Johnsons baby shampoo) and
    water daily
  • Antibiotic ointment at bedtime for 2-3 weeks
    (Bacitracin or erythromycin)
  • Resistant cases can be referred to the
    ophthalmologist on a non-urgent basis

42
Blepharitis
  • In general, blepharitis is not curable only
    controllable and exacerbations are common

43
Orbital Disease
  • Preseptal cellulitis
  • Orbital cellulitis

44
  • Differentiation between preseptal and orbital
    cellulitis is important because treatment,
    prognosis, and complications are different

45
Preseptal Cellulitis
  • Infection of the eyelids and soft tissue
    structures anterior to the orbital septum
  • May be due to skin infection, trauma, upper
    respiratory illness or sinus infection

46
Preseptal Cellulitis - Symptoms
  • Mild to very severe eyelid edema
  • Eyelid erythema
  • Normal ocular motility
  • Normal pupil exam
  • Mild systemic signs (fever, preauricular and
    submandibular adenopathy)

47
Preseptal Cellulitis - Evaluation
  • Swab drainage if present for gram stain and
    culture
  • CBC
  • Blood cultures in more severe cases
  • CT scan of orbit to assess the paranasal sinuses,
    posterior extention into the orbit, and presence
    of subperiosteal or orbital abcesses

48
Preseptal Cellulitis - treatment
  • Systemic antibiotics
  • The younger the patient and the more severe the
    disease the more likely to initiate inpatient
    treatment (IV antibiotics)

49
Orbital Cellulits
  • Infectious process posterior to the orbital
    septum that affects orbital contents
  • Medical emergency !!!!
  • Requires combined efforts of pediatrician,
    ophthalmologist and often otolaryngologist for
    management

50
Orbital Cellulitis - Causes
  • Bacterial infection of the adjacent paranasal
    sinuses, particularly the ethmoids
  • Infants may develop secondary to dacryocysitis
    (infection of the nasolacrimal system)

51
Orbital Cellulitis Signs and Symptoms
  • Redness and swelling of lids
  • Impaired motility often with pain on eye movement
  • Proptosis
  • Decreased vision
  • Afferent pupillary defect
  • Optic disc edema

52
Orbital Cellulitis Note the marked lid swelling
and erythema
53
Orbital Cellulitis Note the periorbital edema
and erythema and the chemosis (conjunctival
swelling)Picture from Section 6 of the Basic
and Clinical Science Course published by the
Foundation of the American Academy of
Ophthalmology
54
Orbital Cellulitis Management
  • Hospitilization
  • Ophthalmology consult (urgent)
  • Blood culture
  • Orbital CT scan
  • IV antibiotics

55
Orbital Cellulitis Complications
  • Optic nerve damage (permanent visual loss)
  • Menititis in 1.9 of cases as infection may
    spread through the valveless orbital veins
  • Subperiosteal abcess
  • Cavernous sinus thrombosis

56
Subperiosteal abcess of the left orbit. Note the
dome shaped elevation of the periosteum along the
left medial orbital wall. Picture from Section 6
of the Basic and Clinical Science Course
published by the Foundation of the American
Academy of Ophthalmology
  • R L

57
Lacrimal System
  • Nasolacrimal duct obstruction
  • Dacryocystocele

58
Nasolacrimal Duct (NLD) ObstructionCongenital
  • Normal baseline lacrimation increases over the
    first 2 to 3 weeks of life therefore NLD
    obstructions may not be evident until the child
    is 3 weeks old
  • Usually due to failure of membranous valve of
    Hasner to regress
  • Up to 90 will spontaneously resolve without
    treatment (75 in the first six months of life)

59
Symptoms
  • One or both eyes appear moist
  • Tears overflow and stream down the cheek
  • Chronic or intermittent infections
  • Crusting of eyelashes
  • Periocular skin red and irritated

60
Treatment
  • Topical antibiotics (use prn yellow or green
    discharge, may use polytrim drops or erythromycin
    ointment)
  • Lacrimal sac massage (apply digital pressure over
    the lacrimal sac and then pull finger down the
    side of the nose)
  • Probe and irrigation
  • Attempt to rupture the membranous valve of Hasner
  • Silicone intubation
  • Recommended after no response to two probings or
    child over 1 year of age

61
When to refer
  • Children with suspected NLD obstructions should
    be referred to an ophthalmologist at 9 months of
    age if no resolution. Children under 1 year of
    age may be offered the option of an in office
    probing which can avoid general anesthesia.

62
NLD obstruction of the right eye. Note the
overflow tearing and the mucous on the lashes
without redness of the conjunctiva.Picture from
Section 6 of the Basic and Clinical Science
Course published by the Foundation of the
American Academy of Ophthalmology
63
Congenital Dacryocystocele
  • Blue, cyst like mass below medial canthal tendon
  • Nasolacrimal sac and duct distended with fluid
  • Upper and lower duct obstructions
  • Frequent secondary infections

64
Dacryosystocele treatment
  • Small percentage spontaneously decompress
  • Digital massage of lacrimal sac and topical
    antibiotics
  • Nasolacrimal duct probing with or without
    systemic antibiotics

65
Congenital Dacryocystocele of the right eye.
Note the elevation and bluish coloration of the
skin.Picture from Section 6 of the Basic and
Clinical Science Course published by the
Foundation of the American Academy of
Ophthalmology
66
Dacryocystitis
67
Conjunctiva
  • Conjunctivitis
  • Ophthalmia neonatorum
  • Subconjunctival hemorrhage
  • Dry Eyes (keratoconjunctivitis sicca)

68
Conjunctivitis
  • Nonspecific term for inflammation and erythema of
    the conjunctiva.
  • Several causes
  • Bacterial
  • Viral
  • Allergic
  • Chemical

69
Conjunctivitis Contd
  • History and symptoms can help determine the
    etiology
  • Correct diagnosis has direct implications for
    treatment and possible spread to close contacts

70
Conjunctivitis Contd
  • History
  • Any recent contact with some one with a red eye
    (within the past 2-3 weeks)?
  • How did it start?
  • Has it spread from one eye to the other?
  • Any tearing or discharge?
  • Any changes in vision?
  • Does it itch?
  • Has the child been rubbing their eyes?

71
Conjunctivitis - Discharge
  • Discharge Cause
  • Purulent Bacteria
  • Clear Viral
  • White mucous Allergies

72
Bacterial Conjunctivitis
  • Common causes
  • Staphylococcus
  • Streptococcus
  • Hemophilus
  • Pneumococcus

73
Bacterial Conjunctivitis
  • Erythema of conjunctiva
  • Purulent discharge
  • May be monocular (one eye) or binocular (both
    eyes)
  • Hemophilis may cause hemorrhage on the conjuctiva
    and occasionally the lids

74
Bacterial conjunctivitis note the purulent
discharge and conjunctival hyperemia Picture
taken from Basic Ophthalmology for Medical
Students and Primary Care Residents published by
the American Academy of Ophthalmology
75
Bacterial Conjunctivitis - treatment
  • Broad spectrum topical antibiotics
  • Polytrim, Ocuflox, Ciloxan
  • Warm compresses
  • Children may return to school once antibiotic
    therapy is instituted
  • Refer if not markedly improved within 4 days

76
Viral Conjunctivitis
  • Adenovirus
  • May be associated with systemic viral infections
  • Herpetic
  • Picornavirus and enterovirus type 70 cause a
    hemorrhagic conjunctivitis

77
Viral Conjunctivitis (non-herpetic)
  • HIGHLY CONTAGIOUS
  • Usually starts in one eye and progresses to the
    second eye
  • Often a history of recent contact with another
    person with a red eye or pink eye
  • Children must be kept out of school until tearing
    stops (up to two weeks)

78
Viral conjunctivitis - symptoms
  • Often bilateral
  • Often with diffuse, marked hyperemia
  • Watery discharge
  • Chemosis ( swelling of conjunctiva)
  • Some itching and foreign body sensation
  • Preauricular adenopathy
  • URI, sore throat, fever common

79
Viral conjunctivitis note the diffuse redness
and watery discharge
80
Viral conjunctivitis - treatment
  • Cold compresses
  • Good hygiene wash hands, do not share wash
    cloths, pillows, towels etc.
  • Topical treatment for symptom relief only (will
    not shorten the course of the disease)
  • Patanol, Zaditor, Acular, Artificial tears
  • No role for topical antibiotics

81
Viral conjunctivitis - complications
  • Usually resolves without sequelae
  • May be associated with corneal infiltrates that
    can decrease vision
  • Pseudomembranes on conjunctival surfaces of lids
    seem with eversion of lids and require removal
    with a dry Q-tip. May refer to ophthalmologist
    for this urgently if uncomfortable doing this in
    the office

82
Viral Conjunctivitis - Herpetic
  • Profuse watery discharge
  • May have eyelid margin ulcers and vesicles
  • Corneal involvement may result in permanent
    scarring and visual loss
  • Urgent referral to ophthalmologist for treatment
    with topical antivirals

83
Herpetic lid lesions from Herpes Simplex virus
Picture from Section 6 of the Basic and Clinical
Science Course published by the Foundation of the
American Academy of Ophthalmology
84
Typical herpetic corneal lesion stained with rose
bengal. Note the branching (dendritic) pattern.
Picture from Section 6 of the Basic and Clinical
Science Course published by the Foundation of the
American Academy of Ophthalmology
85
Allergic Conjunctivitis
  • Associated with hay fever, asthma, eczema
  • Often bilateral and seasonal
  • Milder conjunctival hyperemia
  • Chemosis
  • Itching (primary symptom)
  • Not contagious, children may return to school

86
Allergic conjunctivitis note the conjunctival
erythema but no watery discharge
87
Allergic conjunctivitis - treatment
  • Cold compresses
  • Topical antihistamines (Livostin)
  • Topical non-steroidals (Acular)
  • Topical mast cell stabilizers (Alomide)
  • Not effective until after one week of use

88
Ophthalmia Neonatorum
  • Chemical
  • Gonococcal
  • Chlamydial
  • Herpetic

89
Chemical conjunctivitis
  • Onset first 24 hours
  • Cause silver nitrate (90)
  • Signs Sxs bilateral, mild eyelid edema, clear
    discharge, conjunctival injection
  • Treatment supportive, spontaneous resolution in
    a few days

90
Gonococcal conjunctivitis
  • Onset 48 hours
  • Cause Neisseria gonorrhea via birth canal
  • Signs Sxs severe, purulent discharge,
    chemosis, eyelid edema
  • Dx gram stain
  • Treatment systemic cefriaxone or Pen G, topical
    erythromycin and irrigation

91
Gonococcal conjunctivitis note the copious
amounts of purulent dischargePicture from
Section 6 of the Basic and Clinical Science
Course published by the Foundation of the
American Academy of Ophthalmology
92
Chlamydial conjunctivitis
  • Onset 4 to 7 days
  • Cause
  • Signs Sxs more indolent, eyelid edema,
    pseudomembrane formation
  • Dx Giemsa-stained conj swabbings, fluorescent
    antibody staining
  • Treament topical and oral erythromycin
  • Treat parents as well

93
Herpetic conjunctivitis
  • Onset 1 2 weeks
  • Cause HSV 2 via birth canal
  • Signs Sxs serous discharge,conj injection and
    geographic keratitis
  • Dx Gram stain (multinucleated giant cells),
    Papanicolaou stain, viral cultures
  • Treatment topical antiviral trifluorothymidine
    and systemic acyclovir

94
Subconjunctival hemorrhage
  • Bleeding into the potential space between the
    conjunctiva and sclera
  • Usually resolve without sequelae and require no
    treatment
  • May be due to trauma, associated with
    conjunctivitis, coughing, sneezing
  • No need for referral

95
Subconjunctival hemorrhage
  • If associated with trauma inspect globe carefully
    to rule out other injuries
  • Corneal abrasions (discussed later)
  • Open globe (emergency requiring immediate
    referral to ophthalmologist)
  • Hyphema (discussed later)

96
Subconjunctival hemorrhage
97
Dry Eyes
  • Unusual in children
  • Symptoms
  • Burning, foreign body sensation, reflex tearing,
    mild if any conjuncitival hyperemia

98
Dry Eyes
  • Associated with
  • Aging
  • Rheumatoid arthritis
  • Stevens-Johnson syndrome
  • Systemic medications

99
Dry eyes - treatment
  • Artificial tear drops may be used as needed
  • May refer to an ophthalmologist on non-urgent
    basis if no relief

100
Cornea
  • Corneal Abrasions
  • Corneal Ulcers
  • Herpetic Keratitis
  • Chemical Burns

101
Corneal Abrasions
  • Often a history of trauma or getting something in
    the eye or contact lens wear
  • Symptoms
  • Pain, photophobia (light sensitivity), redness,
    tearing, blurred vision
  • Usually monocular

102
Corneal Abrasions - Diagnosis
  • Application of fluorescien dye into the eye and
    viewing with a cobalt blue light. Abrasion
    will appear green.
  • Application of a topical anesthetic (Alcaine)
    will aid with exam if available

103
Corneal Abrasions - treatment
  • Small abrasions will heal within 24 hours, larger
    abrasions take longer
  • May patch with a topical antibiotic ointment for
    24 hours (patch aids for comfort so that lid does
    not constantly pass across abrasion, not
    practical in younger children)
  • Prescribe topical antibiotic ointment or drop
  • Patient should be followed daily or every other
    day until healed
  • May refer to ophthalmologist for the next day
    follow up

104
Patching technique
  • Instill either an antibiotic ointment or drop
    into the eye
  • Instruct the patient to close both eyes
  • Place two eye pads over the affected eye (may
    fold the bottom pad in half to apply more
    pressure)
  • Tape firmly in place so that patient can not open
    lids beneath patch
  • The patch should be removed in 24 hours

105
Pressure patch applied to left eyePicture taken
from Basic Ophthalmology for Medical Students and
Primary Care Residents published by the American
Academy of Ophthalmology
106
Corneal Ulcer
  • A localized infection of the cornea
  • Usually bacterial, but may be fungal or protozoan
    (ameoba)
  • Requires emergent referral to an opthalmologist

107
Corneal Ulcer Signs/Symptoms
  • Pain
  • Photophobia
  • Foreign body sensation
  • Conjunctival hypermia
  • White opacity on the cornea
  • Anterior chamber inflammation (iritis)
  • May have associated hypopyon (pus in the anterior
    chamber)

108
Corneal Ulcer
  • Patient may have history of trauma or contact
    lens wear
  • Always suspect fungal infection if trauma is with
    vegetative matter i.e. tree branch

109
Corneal Ulcer note the white lesion on the
central cornea, the hypopyon (pus in the anterior
chamber), and the conjunctival hyperemiaPicture
taken from Basic Ophthalmology for Medical
Students and Primary Care Residents published by
the American Academy of Ophthalmology
110
Corneal Ulcer treatment
  • If ulcer severe, patient monocular (only has one
    seeing eye), or patient young may require
    hospitialization
  • Intensive topical antibiotic therapy with broad
    spectrum antibiotic (i.e. Ocuflox, Ciloxan,
    fortified Keflex)
  • Corneal cultures and gram stain

111
Corneal Ulcers complications
  • corneal scarring and permanent visual loss
  • corneal perforation requiring emergent surgical
    intervention

112
Herpetic Keratitis
  • Due to herpes simplex virus
  • Corneal involvement usually preceeded by
    conjunctival involvement
  • Refer to an ophthalmologist within 24 hours so
    that topical antiviral treatment may be started

113
Typical dendritic lesion of herpetic keratitis
stained with fluorescein
114
Herpetic Keratitis complications and prognosis
  • Recurrent process
  • Corneal scarring is common and leads to visual
    loss

115
Chemical Injury
  • Range from mild inflammation to severe damage
    with loss of the eye
  • Most important chemicals are strong acids and
    bases

116
Acid Injuries
  • Acid burns produce denaturation and coagulation
    of protein. Acid damage often limited by
    nuetralization of the buffering action of the
    tissues
  • Damage limited to area of contamination
  • Sulfuric and Nitric acids most common
  • Usually industrial, but may result from
    automobile battery explosions

117
Alkaline Injuries
  • Penetrate ocular tissues rapidly and produce
    intense ocular reactions
  • Damage widespread, uncontrolled, and progressive
  • Often results in epithelial loss, corneal
    opacification, scarring, severe dry eye,
    cataract, glaucoma and blindness

118
Chemical Injury Treatment
  • The single most important step in management is
    complete and copious irrigation of the eye
  • Treatment should be instituted within minutes
  • A true ocular emergency!!!!

119
Ocular Irrigation
  • Instill a drop of topical anesthetic if available
    (proparicaine)
  • Use eye irrigation solutions and normal saline IV
    drip
  • Squeeze copious amounts of solution into the eye
    and direct towards the temple, away from the
    unaffected eye
  • Irrigate under the lids

120
Chemical Injury Treatment
  • After several minutes of irrigation, check the pH
    of the eye by placing litmus paper into the
    inferior fornix
  • If the pH is not neutral resume irrigation until
    pH neutralized
  • Recheck pH 30 minutes after neurtralization as pH
    can rise again after irrigation stopped

121
Chemical Injury Treatment
  • Remove any visible particulate matter
  • Requires emergent referral to an ophthalmologist
    however, commence irrigation prior to calling the
    ophthalmologist

122
Anterior Chamber
  • Iritis
  • Hyphema

123
Iritis
  • Inflammation of the anterior segment of the eye
  • May be idiopathic, secondary to trauma, or
    associated with a systemic disease

124
Iritis signs/symptoms
  • Ciliary flush
  • Photophobia (light sensitivity)
  • Miotic pupil (pupil is smaller on affected side)
  • Keratic precipitates
  • Usually not associated with tearing or discharge

125
Iritis - treatment
  • Steroids may be topical, injected below the
    conjunctiva or tenons, or oral depending on
    cause and severity of iritis
  • Cycloplegia use of cycloplegic drop to dilate
    pupil. This will decrease movement of iris thus
    aiding with pain and help prevent scarring of
    iris to the lens

126
Iritis - referral
  • Should be referred on an urgent basis to an
    ophthalmologist for treatment and follow-up

127
Hyphema
  • Blood in the anterior chamber
  • Usually associated with trauma
  • Requires emergent referral to an ophthalmologist
    for treatment

128
Hyphema note the layered blood in the anterior
chamberPicture taken from Basic Ophthalmology
for Medical Students and Primary Care Residents
published by the American Academy of Ophthalmology
129
Hyphema - treatment
  • Strict bedrest
  • Topical steroids
  • Topical cycloplegic agents
  • Admit to hospital if young or concerned about
    follow-up or compliance
  • Need daily exams for 5 days including measurement
    of intraocular pressure
  • Sickle-cell prep (patients with sickle cell trait
    need more aggressive management of elevated
    intraocular pressures)

130
Review
  • True emergency (therapy instituted within
    minutes)
  • Chemical Injuries

131
Review
  • Require same day referrals
  • Orbital cellulitis
  • Ophthalmia neonatorum (except chemical)
  • Iritis
  • Hyphema
  • Corneal Ulcers

132
Review
  • Refer in 1-2 days
  • Preseptal cellulitis
  • Dacryocystocele
  • Herpetic conjunctivitis
  • Herpetic keratitis
  • Corneal abrasions

133
Review
  • Refer if no response to conservative management
  • Hordeolum/Chalazion
  • Blepharitis
  • NLD obstruction
  • Viral conjunctivitis
  • Allergic conjunctivitis
  • Bacterial conjunctivitis (exept due to gonorrhea)
  • Dry Eyes
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