ATRIAL SEPTAL DEFECT - PowerPoint PPT Presentation

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ATRIAL SEPTAL DEFECT

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ATRIAL SEPTAL DEFECT BY DR.ANAND Normally, oxygen-poor (blue) blood returns to the right atrium from the body, travels to the right ventricle, then is pumped into the ... – PowerPoint PPT presentation

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Title: ATRIAL SEPTAL DEFECT


1
ATRIAL SEPTAL DEFECT
  • BY DR.ANAND

2
  • Normally, oxygen-poor (blue) blood returns to the
    right atrium from the body, travels to the right
    ventricle, then is pumped into the lungs where it
    receives oxygen. Oxygen-rich (red) blood returns
    to the left atrium from the lungs, passes into
    the left ventricle, and then is pumped out to the
    body through the aorta.

3
  • An atrial septal defect allows oxygen-rich (red)
    blood to pass from the left atrium, through the
    opening in the septum, and then mix with
    oxygen-poor (blue) blood in the right atrium.

4
DEFINITION
  • An atrial septal defect is an opening in the
    atrial septum, or dividing wall between the two
    upper chambers of the heart known as the right
    and left atria..

5
GROSS SPECIMENS
6
EMBRYOLOGY
  • The heart is forming during the first 8 weeks of
    fetal development. It begins as a hollow tube,
    then partitions within the tube develop that
    eventually become the septa (or walls) dividing
    the right side of the heart from the left. Atrial
    septal defects occur when the partitioning
    process does not occur completely, leaving an
    opening in the atrial septum.

7
HEMODYNAMICS
  • RT.ATRIUM RECEIVES BLOOD FROM SUP. INF.VENA
    CAVA FROM LT. ATRIUM
  • RT.ATRIUM ENLARGES

8
HEMODYNAMICS
  • LARGE VOL OF BLOOD FROM RT.ATRIUM PASSES THRU
    NORMAL TRICUSPID VALVE PULMONARY VALVE
  • DELAYED DIASTOLIC MURMUR(LOW LT STERNAL BORDER)
  • RT.VENTRICLE ENLARGES
  • PULMONARY EJECTION MURMUR

9
HEMODYNAMICS
  • PULM. VALVE CLOSES LATE P2 IS DELAYED
  • RV IS FULLY LOADED,SO FURTHER RISE IN RV VOLUME
    CANNOT OCCUR
  • WIDELY SPLIT S2
  • FIXED SPLIT S2
  • ACCENTUATED S2

10
PRESENTATION
  • recurrent chest infections
  • fatigue
  • sweating
  • rapid breathing
  • shortness of breath
  • poor growth

11
ON EXAMINATION
  • INSPECTION
  • PARASTRNL IMPULSE
  • PALPATION
  • SYSTOLIC THRILL AT 2ND LT SPACE

12
AUSCULTATION
  • WIDE FIXED SPLIT S2
  • ACCENTUATED P2
  • ESM AT LT 2nd 3rd INTERSPACES
  • DELAYED DIASTOLIC MURMUR AT LOW LT INTERSPACE

13
CXR FINDINGS
  • MOD. CARDIOMEGALY
  • RA ENLARGEMENT
  • RV ENLARGEMENT
  • PROMINENT MAIN PULM ARTERY
  • PLETHORIC LUNG FIELDS

14
ECG CHANGES
  • RT AXIS DEVIATION
  • RT VENT HYPERTROPHY
  • rsR PATTERN IN V1

15
ECHO PICTURES
16
SEVERITY ASSESMENT
  • INTENSITY OF THE TWO MURMURS
  • THE HEART SIZE

17
COMPLICATION
  • PULMONARY HYPERTENSION(ABOVE 20 YEARS)
  • DISAPPEARANCE OF DIASTOLIC MURMUR
  • APPEARANCE OF PULM EJECN CLICK
  • LOUD PALPABLE P2
  • P2_STILL WIDELY SPLIT

18
MANAGEMENT
  • MEDICAL
  • ANTIBIOTICS FOR CHEST INFECTIONS
  • DIGOXIN TO INCREASE WORK OF HEART
  • DIURETICS TO REDUCE PRELOAD

19
SURGICAL REPAIRDEVICES
20
REPAIR
21
ROBO REPAIR
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