Title: Intermittent Compression Devices
1Intermittent CompressionDevices
- Jennifer Doherty-Restrepo, ATC, LAT
- Entry-Level Athletic Training Education Program
- PET 4995 Therapeutic Modalities
2Definitions
- Edema
- Abnormal amounts of fluid in extracellular tissue
spaces - Joint swelling
- Blood and/or fluid accumulation
- Appears and feels like a water balloon
- Lymphedema
- Accumulation of lymph in subcutaneous tissues
- Occurs over several hours following injury
3The Lymphatic System
- Pick up plasma and plasma proteins that escape
from small blood vessels and return them to blood
circulation - Acts as a safety valve for fluid overload
- Prevents edema formation
- Maintains homeostasis of extracellular
environment - Removes excess protein molecules and waste from
the interstitial fluid - Cleanses interstitial fluid
- Blocks thes spread of infection or malignant
cells
4The Lymphatic System
- Closed vascular system
- Parallels arterial system
- Lymphatic capillaries made of single
layered endothelial cells
5The Lymphatic System
- Lymphatic capillaries are surrounded by
interstitial fluid - Terminal lymphatics provide entry way into
lymphatic system for excess interstitial fluid
and plasma proteins
6The Lymphatic System
- Lymphatic capillaries form a network of lymphatic
vessels - Lead to larger Collecting Vessels in extremities
- Collecting vessels connect with thoracic duct or
right lymphatic duct, which join the venous
system
7The Lymphatic System
- Fluid moving into interstitial spaces pushes or
pulls on fibrils - Fibrils connective tissue fibers supporting
lymphatic capillaries - Endothelial cell junctions in lymphatic capillary
wall open - Allows entry of interstitial fluid,
cellular waste, large protein
molecules, plasma
proteins,
extracellular particles, and
cells into terminal
lymphatics
8The Lymphatic System
- Once in the terminal lymphatics, the interstitial
fluid, cellular waste, large protein molecules,
plasma proteins, extracellular particles, and
cells become lymph - Endothelial cell junctions in lymphatic
capillaries remain closed when there is - No increase in interstitial volume
- No tissue activity
- Movement of lymph is accomplished via muscle
activity, active and passive motion, elevation,
respiration, and blood vessel contraction
9Injury Edema Pitting Edema
- Local edema
- Formed by plasma, plasma proteins, and cellular
debris moving into interstitial spaces - Hormones released by injured cells stimulate
small anterioles, capillaries and venules to
vasodialate separating endothelial cells in
vessel wall - Increases permeability
10Injury Edema Pitting Edema
- Increased permeability allows more plasma, plasma
proteins, and cellular debris to escape into
local area - Results in gel-like fluid that is trapped by
collagen fibers - Gel-like fluid referred to as pitting edema
11Injury Edema Lymphedema
- Accumulation of edema causing overdistention of
lymph capillaries - Entry pores become ineffective and interstitial
fluid increases - Lymphedema results
- Constriction of lymph capillaries due to
increased pressure will also discourage lymph
flow and cause lymphedema formation
12Negative Effects of Edema
- Edema compounds injury by causing secondary
hypoxic cellular death in surrounding tissues - Other negative effects include
- Physical separation of torn tissue ends
- Pain
- Restricted joint range of motion
- Prolonged recovery times
- Interstitial fibrosis
- Reflex sympathetic dystrophy
13Treatment of Edema
- P.R.I.C.E.
- Minimize edema
formation - E-stim or AROM
exercises - Retard edema
accumulation - Any treatment that assists lymph flow will
decrease plasma protein content in interstitial
spaces and decrease edema
14Treatment of Edema Elevation
- Gravity used to assist lymph flow
- The higher the elevation, the greater the effect
on the lymph flow
15Treatment of Edema Compression
- Muscle contractions provide rhythmic internal
compression of lymph vessels to assist lymph flow - Isometrics, AROM exercise, or E-stim
- External compression assists lymph flow
- Massage, elastic compression, or intermittent
pressure devices - External compression spread interstitial edema
over a larger area, enabling more lymph
capillaries to become involved in removing plasma
proteins
16Treatment of Edema Weight-Bearing Exercise
- Activates a venous pump
- Not related to muscle activity
- Mediated by release of an endothelial-derived
relaxing factor (EDRF) - EDRF is liberated by sudden pressure changes and
it diffuses locally - Functions to relax smooth muscle and stimulate
blood flow in the veins
17Treatment of Edema Cryotherapy
- Cryotherapy used in conjunction with intermittent
compression has shown the best results in the
reduction of post-acute injury edema
18Intermittent Compression Treatment Parameters
- Inflation Pressure
- On/Off Time Sequence
- Total Treatment Time
19Inflation Pressure
- Loosely correlated with BP
- Most treatment protocols utilize a pressure
approximating diastolic BP
20Inflation Pressure
- Arterial capillary pressures 30 mmHg
- Any pressure that exceeds this should encourage
- Edema absorption, and
- Lymph flow
21Inflation Pressure
- Maximum pressure should correspond to diastolic
BP - More pressure is not necessarily better
- Enough pressure is needed to squeeze lymphatic
vessels and encourage lymph flow
22On/Off Time Sequence
- On/Off time sequences are variable
- Patient comfort should be a primary deciding
factor - 30s on, 30s off is effective and comfortable
- Lymphatic massage
- Shorter on/off time sequences may have an
advantage
23Total Treatment Time
- Clinical studies show significant limb volume
reduction after 30 minutes of compression - A treatment of 10 - 30 minutes may be adequate
unless edema is overwhelming in volume or is
resistant to treatment - Multiple treatment times per day may also be an
advantage in controlling and reducing edema
24Equipment Setup and Instructions
- Compression sleeves
- Half-leg, full-leg, half-arm, or full-arm
- Connect compression sleeve (deflated) to unit via
a rubber hose and connecting valve
25Equipment Setup and Instructions
- Assess BP and set inflation pressure
- Set On-time (between 30 to 120 seconds)
- Off-time is left at 0 until the compression
sleeve is inflated and treatment pressure is
reached - Then adjust off-time between 0 and 120 seconds
- During the off-time, instruct patient to move
extremity - Treatment should last between 20 - 30 minutes
26Cold and Compression Combination
- Combination of cold and compression is effective
in treating edema - Cold/compression unit
- Temperature adjustment ranges between 10 - 25oC
- Cooling accomplished by circulating cold water
through compression sleeve
27Compression and Electrical Stimulating Currents
- Combination of compression and e-stim
induced muscle pumping is effective in treating
edema - Facilitates reabsorption of injury byproducts by
lymphatic system
28Sequential Compression Pumps
- Sequentially inflate compression sleeves with
multiple compartments - Massage effect
- Applies pressure from distal to proximal
- Gradual decrease in pressure gradient
29Sequential Compression Pumps
- Highest pressure in distal compartment sleeve
- Determined by mean value of systolic to diastolic
BP - Middle compartment is set at 20 mm lower than the
distal cell - Proximal compartment pressure is reduced an
additional 20 mm
30Sequential Compression Pumps
- Each pressure cycle is 120s
- Distal compartment is pressurized initially and
continues pressurization for 90s - 20s later, the middle compartment inflates
- After another 20s, the proximal compartment
inflates - A final 30s period allows pressure in all three
compartments to return to 0 - Entire cycle is then repeated
31Indications
- Lymphedema
- Traumatic edema
- Chronic edema
- Stasis ulcers
- Intermittent claudications
- Postoperatively to reduce the possibility of
developing a deep vein thrombosis - Facilitate wound healing following surgery
32Contraindications
- Deep vein thrombosis
- Local superficial infection
- Congestive heart failure
- Acute pulmonary edema
- Displaced fractures
- Cancer