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APPLIED ANATOMY OF PARA NASAL SINUSES BY DR. SAIMA

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Title: APPLIED ANATOMY OF PARA NASAL SINUSES BY DR. SAIMA


1
.APPLIED ANATOMY OF PARA NASAL SINUSES
  • .MICRO ANATOMY WITH FUNCTION
  • PRESENTOR- DR. SAIMA TABASSUM SAROORI
  • RESIDENT Ist year,
  • DEPARTMENT OF ENT-MMIMSR

2
APPLIED ANATOMY OF MAXILLARY SINUS
3
  • FLOOR
  • OROANTRAL FISTULA-MAXILLARY TEETH ARE IN
    DIRECT RELATION TO MAXILLARY SINUS FLOOR. TH E
    DISTANCE BETWEEN THE APICAL END OF MAXILLARY POST
    TOOTH WITH THE FLOOR OF MAXILLARY SINUS IS
    1-1.5CM IN ADULTS. THIS DISTANCE IS MAX IN
    PAEDIATRIC GROUP lt 15 YEARS OF AGE SO CHANCE OF
    ORO ANTRAL FISTULA IS LESS COMMOM IN THEM

4
  • CALDWELL LUC SURGERY WALL OF MAXILLARY
    SINUS IS VERY THIN IN CANINE FOSSA REGION SO THIS
    IS AN IDEAL SITE FOR CALD WELL LUC SURGERY USED
    IN
  • 1. CHRONIC MAXILLARY SINUSITIS WITH
    IRREVERSIBLE CHANGES IN SINUS MUCOSA
  • 2.SUSPECTED NEOPLASM IN THE ANTRUM ITS
    BIOPSY
  • 3. REMOVAL OF FOREIGN BODY OR ROOT OF TOOTH
  • 4. ORO ANTRAL FISTULA DENTAL CYST
  • 5. RECURRENT A.C POLYP
  • 6. OF MAXILLA OR BLOW OUT OF ORBIT
  • 7. LIGATION OF MAXILLARY ARTERY

5
OROANTRAL FISTULA
6
  • MAXILLARY SINUSITIS
  • DENTAL CARIES , HALLUTOSIS, GUM BLEEDING MUST
    BE EXAMINED PROPERLY IN THE PATIENT HAVING
    MAXILLARY SINUSITIS AS DENTAL CARRIES OF
    MAXILLARY MOLARS MAY CAUSE ASCENDING INFECTION IN
    THE MAXILLARY SINUS
  • MAXILLARY TENDERNESS
  • IS ELICITED BY PUTTING GENTLE PRESSURE
    OVER MAXILLARY CANINES.

7
  • ROOF
  • TRAUMA/ BLOW OUT FRACTURE ?
  • ORBITAL WALL OF M.S IS VERY FRAGILE SO ITS
    VULNENARABLE TO TRAUMA. IN SUCH CASES ORBITAL
    FAT MAY PROLAPSE IN SIDE THE MAXILLARY SINUS
    CAVITY CAUSE ENOPTHALOUS,RESTRICTED EYE
    MOVEMENTS,LOSS OF SENSATION OVER I.O REGION,
    PERIORBITAL ECHYMOSIS DIPLOPIA GLAUCOMA
  • EROSION MAXILLARY SINUS TUMOR MAY ENLARGE
    IN SIZE ERODE THE ORBITAL WALL LEADING TO
    EXOPTHALMOUS, CHEMOSIS MAY HAMPER THE VISION

8
BLOW OUT OF LEFT ORBIT
9
  • INFRA ORBITAL NERVE BLOCK? INFRAORBITAL
    FORAMEN TRANSMITIING I.O NERVE VESSELS LIES IN
    THE ORBITAL WALL/ ROOF OF MAXILLARY SINUS. BEING
    VERY FRAGILE IS VULNERABLE TO GET INJURED IN
    ANY ORBITAL/MAXILLARY TRAUMA
  • I.O NERVE IS USED TO ACCOMPALISH REGIONAL
    ANAESTHESIA FOR FACE

10
INFRA ORBITAL NERVE BLOCK
11
ANTERIOR WALL
  • OSTEOMYLIETIS ANTERIOR WALL OF MAXILLARY
    SINUS IS VULNERABLE TO DEVELOP OSTEOMYELITIS
    IS MORE OFTEN SEEN IN INFANTS CHILDREN THAN
    ADULTS COZ OF PRESENCE OF SPONGY BONE IN ANTERIOR
    WALL OF SINUS. INFECTION ASCENDS FROM THE DENTAL
    SAC AND LESS OFTEN ITS PRIMARY INFECTION

12
  • THEN SPREADS TO SUBPERIOSTEAL
    REGION?FISTLA(I.O REGION,ALVEOLUS, PALATE OR IN
    ZYGOMA?SEQUESTERISATION BONE DEFORMITY

13
  • TO ELICIT THE TENDERNESS OF MAXILLARY SINUS?
  • BY TAPPING OVER THE LATERAL ASPECT OF
    ANTERIOR WALL I,e PROMINANCE OF CHEECK BONE
    AND INTRA ORAL PALPATION BETWEEN CANNINE FOSSA
    ZYGOMATIC BUTTRESS.

14
PALPATION OF MAX SINUS
15
  • POSTERIOR WALL
  • LIGATION OF MAXILLARY ARTERY ?
  • IN CASE OF EPISTAXIS WHENCONSERVATIVE
    PROCEDURES ARE NOT HELPFUL ,LIGATION OF MAXILLARY
    ARTERY IS REQUIRED. WHICH CAN BE APPROCHED
    THROUGH INTRA NASAL PATH INTO THE MAXILLARY SINUS
    WHERE WE CREATE A WINDOW IN ITS POSTERIOR WALL TO
    REACH MAXILLARY ARTERY
  • TUMOR OF INFRATEMPORAL SPACE ?
  • MAY ERODE POSTERIOR WALL OF MAXILLARY SINUS
    INFILTERATE PARASYMPATHETIC GANGLION (S.P
    GANGLION)

16
  • PT MAY PRESENT WITH DEEP FACIAL PAIN, HARD
    PALATE INSENSITIVITY DECREASED LACRIMATIOM

17
LIGATION OF MAXILLARY ARTERY
18
  • MAXILLARY CAVITY
  • 1.PROOF PUNCTURE
  • 2. HALLER CELLS
  • 3. KARTAGENERS SYNDROME

19
KARTAGENERS SYNDROME
20
ANTRAL LAVAGE
21
HALLER CELLS
22
APPLIED ANATOMY OF FRONTAL SINUS

23
  • DOMINANCE OF ONE SINUS
  • SINUS IS RELATED TO ANT ETHMOIDAL CELLS BOTH
    EMBRYOLOGICALLY ITS ANATOMY. RT LT FRONTAL
    SINUS DEVELOPS INDEPENDENTLY ARE MORE OFTEN
    ASYMETRICAL LEADING TO DOMINANCE OF ONE SINUS.
  • EROSION OF POST WALL
  • FRONTAL SINUS LIES BETWEEN THE ANTERIOR AND
    POSTERIOR FRONTAL TABLE. ANTERIOR WALL IS THICK
    AS COMPARED TO POST WALL SO EROSION OF POSTERIOR
    WALL IS MORE COMMON.

24
  • INTERSINUS CELL
  • THE INTERVENING INTERSINUS SEPTUM CAN
    SOMETIME GET PNEUMATISED FORMING INTERSINUS CELL.
  • MUCOCELE
  • FLOOR OF FRONTAL SINUS CORRESPONDS TO THE ANT
    ROOF OF ORBIT WHICH IS A THIN BONE CAN BE EASILY
    ERODED BY MUCOCELE

25
  • FRONTAL OSTIUM
  • FRONTAL SINUS DRAINAGE PATHWAY HAS AN
    HOURGLASS SHAPE OPENS IN NOSE AT THE LEVEL OF
    FRONTAL RECESS. NARROWEST PART OF THIS TRACT IS
    FRONTAL INFUNDIBULUM OR OSTIA WHICH IS LOCATED AT
    MOST I.M ASPECT OF SINUS AND IS BOUNDED BY
    VARIOUS IMP STRUCTURES. BESIDE DIFF ANT ETH CELL
    GROUPS FRONTAL OSTIUM IS MOST CHALLENGING TO
    ACCESS( SMALL DIA,ANT LOCATION,RELATION WD ORBIT)

26
  • UNCAPPING OF EGG
  • DEPENDING ON THE SUPERIOR ATTACHEMENT OF
    U.P, F.R WILL DRAIN MEDIAL OR LATERAL TO THE
    U.P? RECESSES TERMINALIS?UNCAPPING OF EGG
  • SUPRAORBITAL CELLS
  • SOMETIMES THE SUPRAORBITAL CELLS ARE PRESENT
    IN FRONTAL RECESS? CAN BE SEEN AS TWO OPENINGS.
    F.S OPENING LIES MEDIOALLY LATERAL OPENING IS
    FOR S.O CELLS

27
  • FRONTAL RECESS FRONTAL SINUS?
  • WHILE DISSECTING FRONTAL RECESS SURGEON MAY
    THINK HAS ENTERED F.S BUT HE MAY BE WITH IN THE
    FRONTAL CELL
  • OSTEOMYELITIS/POTTS PUFFY TUMOR?
  • OF F.B NOT VERY UNCOMMON. THE MUCOSAL LINING
    OF F.S, MARROW CAVITY FRONTAL BONE SHARE COMMON
    VENOUS DRAINAGE THROUGH VALVELESS DIPLOEIC VEINS.
    THIS VENOUS DRAINGAE PERMITS SPREAD OF INFECTION
    FROM F.S INTO F.BONE.

28
POTTS PUFFY TUMOUR
29
  • LOCAL SUPPORATION OF FRONTAL BONE PROPOGATES
    TO HAVERSIAN SYSTEM OF INNER OUTER TABLE OF
    SKULL
  • DEMINERALISATION,NECROSIS
  • PERFORATION OF ANT TABLE OF F.S
  • SUBPERIOSTEAL COLLECTION OF PUS
  • GRANULATION TISSUE FORMATION
  • POTTS PUFFY TUMOR PT MAY PRESENT WITH
    ANT WALL BULGE FLUCTUANT TENDER
    ERYTHEMATOUS SWELLING OF F.S IS PATHOGNOMIC

30
  • OSTEOPLASTIC BONE FLAP?
  • MOST OF THE FRONTAL SINUS SURGERY IS CARRIED
    OUT THROUGH INTRA NASAL APPROACH . ONLY 5 OF
    ALL FRONTAL SINUS OPERATIONS ARE EXTERNAL. THIS
    APPROACH IS CONSIDERED AS PREFFERED ONE IN
    PROBLEM F.S WHERE COMPLETE MICRO ENDOSCOPIC
    REMOVAL OF MUCOSA IS DONE OBLITERATION OF SINUS
    IS DONE WITH ABDOMINAL FAT

31
  • THUMB TEST ?FRONTAL TENDERNESS IS EXAMINED BY
    THUMB TEST? PR ON F.S FLOOR CLEARLY BEHIND
    SUPRA ORBITAL RIM
  • PATIENT MAY PRESENT WITH PAINFUL PERCUSSION OVER
    F.B ?ACUTE FRONTAL SINUSITIS,PYOCELE
  • PAINLESS PERCUSSION WITH BULGING OF ANT WALL?
    MUCOCELE, TUMOR , PNEMATOSINOUS DILATANS OF F.S

32
THUMB TEST
33
APPLIED ANATOMY OF SPHENOID SINUS

34
  • DOMINANCE OF S.S?
  • RT LT SPHENOID SINUSES ARE SO ASYMETRICAL
    THAT IT LEADS TO DOMINANCE OF 1 SPHENOID COZ OF
    THIS ASYMETRY, THE INTERSINUS SEPTUM GOES OFF THE
    MIDLINE CAN HAVE A POSTERIOR INSERTION ON BONY
    CAROTID CANAL IN THE LAT WALL OF SPHENOID?BRISK
    AVULSION?CAROTID RUPTURE
  • RELATION WITH PITUTARY GLAND?
  • SUPERIOR WALL OF S.S USUALLY REPRESENT THE
    FLOOR OF SELLA TURCICA. DEPENDING ON EXTENT OF
    PNEMATISATION IS CLASSIFIED IN 3 TYPES. SELLAR
    TYPE IS IN CLOSE RELATION WITH FLOOR OF PITUTARY
    FOSSA, MAKING PITUARY GLAND MORE VULNERABLE TO
    INJURY WHILE APPROACHING S.SIS

35
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36
  • LATERAL WALL OF S.S?
  • SHOWS VARIOUS PROMINANCE, MOST IMP BEING
    CAROTID OPTIC CANAL. ICA IS MOST MEDIAL ST IN
    CAVERNOUS SINUS RESTS AGAINST LATERAL SURFACE
    OF SPHENOID BONE. ITS PROMINANCE VARIES FROM A
    FOCAL BULGE TO SERPENTIGIOUS ELEVATION MAKING
    THE FULL COURSE OF INTRACAVERNOUS PART OF CAROTID
    ARTERY WD AN INTERVENING CLEFT . BONE OVER THESE
    ST ARE VERY THIN LEADING TO DEHISCENCE .

37
STRUCTURES IN RELATION TO S.S
38
  • WHILE APPROACHING S.S, SURGEON MAY GET CONFUSED
    BETWEEN THE P.E.C THE SPHENOID SINUS, WHICH CAN
    BE DIFFERENTIATED ON THE BASIS OF?SHAPE, OPENING
    OF SINUS IN RELATION TO MAXILLARY RIDGE POST
    CHOANA POST END OF NASAL SEPTUM

39
  • IMP. LANDMARK OF SP. OSTIUM?
  • S.S DRAINS THROUGH A SINGLE OSTIUM INTO SER. THIS
    OS IS CLASICALLY LOCATED 7cm FROM COLUMEELA AT
    AN ANGLE OF 30 DEGREE WITH THE FLOOR OF NOSE IN
    PARA SAGITAL PLANE THIS CORRESPONDS TO A
    LOCATION HALF WAY UP THE ANT WALL OF S.S
  • ENDOSCOPICALLY THE POSTEROSUPERIOR END OF SUP
    TURBINATE POINTS SUPERIORLY MEDIALLY TOWARDS
    THE OSTIUM THUS REPRESENTS A VERY IMP LANDMARK
    OF IT.

40
APPLIED ANATOMY OF ETHMOID SINUS

41
CSF RHINORRHOEA
  • THE REGION WHERE ANT ETHMOIDAL ARTERY PERFORATES
    THE LATERAL LAMELLA OF CRIBRIFORM PLATE IS
    THINNEST OF ANT PART OF BASE OF SKULL MEASURES
    0.05mm WITH THE UNDERLYING DURA ADHERANT TO IT SO
    THIS REGION IS HIGHLY VULNERABLE TO TRAUMA
    LEADING TO C.S.F LEAKAGE.
  • PATIENT WITH DEEP OLFACTORY FOSSA(KEROS TYPE 3)
    ARE AT HIGH RISK

42
APPROACH TO ETHMOIDAL AIR CELLS
  • FOR AN APPROACH TO ETHMOID AIR CELLS,SURGEON
    SHOULD MAKE A PERFORATION IN GROUND LAMELLA TO
    ENTER THE POST ETHMOIDAL CELLS, ONCE HE REACHES
    THE POST ETHMOID CELLS, HE MUST CHANGE THE
    DIRECTION OF PROBE INFEROMEDIALLY TO ACCESS S.S.
    IF HE CONTINUES TO MOVE POSTERIORL HE LL ENTER
    THE CRANIAL CAVITY

43
CHANGES IN ETHMOIDAL POLYP
  • IN CASE OF ETHMOIDAL POLYP WHICH IS USUALLY
    BILATERAL ,IT MAY DISTORT THE NORMAL ANATOMY OF
    SINUS THE ETHMOID SINUS WILL EXPAND WHICH LEAD
    TO INCREASE IN INTER CANTHUS DISTANCE MIMICKING
    OCULAR FEATURE OF DOWNS SYNDROME

44
MICROANATOMY OF PNS
45
  • HISTOLOGICAL FEATURES?
  • CONISISTS OF 3 LAYERS-
  • 1.SUPERFICIAL LAYER
  • .EPITHELIAL
    LININIG
  • .MUCUS BLANKET
  • 2.LAMINA PROPRIA
  • .SEROUS GLAND
  • .BLOOD
    VESSELS NERV
  • 3. CARTILAGENOUS LAYER

46
HISTOLOGICAL FEATURE OF PNS
47
  • EPITHELIAL LINING OF PNS?
  • PSUEDOSTRAITIFIED CILIATED COLUMNAR
    EPITHELIUM
  • OTHER CELLS IN SUP LAYER?GOBLET CELLS,
    BASAL CELL, NON CILIATEDCELLS
  • MUCUS LAYER?
  • 1.INNER AQ/SOL/SEROUS LAYER (CONT,LESS
    VISCOUS,SURROUNDS SHAFT OF CILIA)
  • 2.OUTER GEL/SUP. LAYER (DISCONT,HIGH
    VISCOSITY,ALONG TIP OF CILIA,CONTAINS AB,ENZYM)

48
  • CILIARY MOVEMENT OF PNS?
  • CILIA OF PNS BEATS IN A SPIRAL MANNER TO PROPEL
    THE MUCUS OUT OF SINUS IN TO THERE RESPECTIVE
    OSTIUM..
  • BEAT FREQUENCY OF CILIA IS BETWEEN 10-20 Hz AT
    BODY TEMP WD A MAX OF AROUND 14Hz . IT REMAINS
    FAIRLY CONSTANT BTW 32-40 DEG CELSIUS

49
  • EACH BEAT CONSISTS OF A RAPID PROPULSIVE STROKE
    FOLLOWED BY A SLOW RECOVERY PHASE
  • DURING PROPULSION PHASE CILIA IS STRAIGHT THE
    TIP ENGAGES IN VISCOUS LAYER OF MUCUS BLANKET
    WHERE AS DURING RECOVERY PHASE THE CILIA BENT
    OVER LIES IN AQ LAYER.

50
PATTERN OF CILIARY BEAT
51
  • COMPOSITION OF MUCUS?
  • WATER IONS FROM TRANSUDATION
  • GLYCOPROTEINSIALO.SULPHO FUCOMUCINS
  • ENZYMESLYSOZYME,LACTOFFERIN
  • CIRCULATORY PROTEINCOMPLEMENT,ALPHA 2
    MACROGLOBIN, C REACTIVE PROTEIN
  • Ig VARIOUS CELLS E.G BASOPHIL EIOSINOPHIL N
    LEUKOCYTES

52
  • FUNCTIONS OF PARA NASAL SINUSES

53
  • PLAY ROLE IN CONDITIONING THE INSPIRED AIR
  • THEY ADD RESONANCE TO LARYNGEAL VOICE
  • LIGHTEN THE SKULL
  • REGULATION OF INTRANASAL PRESSURE
  • ACTS AS SHOCK TEMP BUFFERS
  • CONTRIBUTE TO FACIAL GROWTH
  • INCREASES THE SURFACE AREA FOR OLFACTION
  • SINUS MUCOSA MAY ACT AS A DONOR SITE FOR
    RECONSTRUCTIVE PROCEDURES E.G SUBGLOTTIC STENOSIS
    IMPLANTATION OF MAX SINUS MUCOSA INTO NASAL
    CAVITY IN ATROPHIC RHINITIS

54
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