Title: The Foot in Diabetes
1The Foot in Diabetes
Start
2Feet in Diabetes
Economics of Diabetes
NICE Guidelines
Foot Assessments
Read Codes for Foot Assessments
3Why worry about Diabetic Feet?
- Counting the cost of diabetes
- NHS expenditure on diabetes projected on the
basis of nine per cent (Currie CJ et al 1997) of
NHS costs in 2000 5,185,314,000 - This is equivalent to
- 99,717,567 a week
- 14,245,367 a day
- 593,560 an hour
- 9,893 a minute
- 165 a second
4Diabetic Feet
- Foot ulceration 'diabetic foot' is the
commonest reason for people with diabetes to be
admitted to hospital in the UK. (Young MJ et al
1994) Diabetic Foot problems take up more bed
days than all the other complications put
together (Waugh NR 1988) - It is a result of nerve damage (neuropathy) and
lack of blood supply (ischaemia). If an ulcer
becomes infected and gangrenous this can
necessitate amputation. According to one widely
cited study, people with diabetes are 15 times
more likely to need amputation than people
without the condition. (Bild DE et al 1989)
5The NICE Guidelines
- The National Institute for Clinical Excellence
(NICE) has produced guidelines to help prevent a
person with diabetes develop the complications
that can lead to amputation and hospitalisation - A Foot assessment can also recognise the
complications affecting the feet and establish
protocols for education, review and onward
referral.
6The NICE Guidelines
- A General Management approach
- Decision making shared between patient and
professionals - Recall and annual review
- Feet Examination as part of Annual review by
trained personnel to assess risk - Examination Pulses sensation deformity and
footwear - Classify Risk low increased high ulcer.
7Low Risk Foot
- Foot Pulses are Present so no Ischaemia
- No Loss of Sensation so no Neuropathy
8Increased Risk Foot
- Either Pulses are Absent
- Or there is Neuropathy
- Or there is Foot deformity
9High Risk Foot
- Previous History of Foot Ulceration
- Or Pulses are Absent
- Or there is Neuropathy
- and there is Foot deformity, or other risk factor
10Read Codes used in Foot Assessments
- Diabetic Foot Risk Assessment 66AW
- Read Codes for Low Risk Feet
- Right Foot 2G5E
- Left Foot 2G5I
- Read Codes for Increased Risk Feet
- Right Foot 2G5F
- Left Foot 2G5J
- Read Codes for High Risk Feet
- Right Foot 2G5G
- Left Foot 2G5K
11Foot Assessment
- The assessment is vital to establish a level of
risk in the Diabetic Foot - This assessment is normally performed in a
primary care setting by Practice nurses, but may
also be performed by Podiatrists, GPs and
Consultant Physicians
12Foot Assessment
- The risk groups the assessment is designed to
place people in are - Low Risk
- Increased Risk
- High Risk
- Ulcerated
- (Ref NICE Guidelines 2005)
13Foot Assessment
- The assessment can be divided into three parts
- History
- Examination
- Investigations
- Remember all these can be done at the same time
in a busy clinic otherwise you will run behind
and people will start getting annoyed with you in
the waiting room!
14History
- Presenting complaint if there is one
- Past Foot History
- Diabetes History
- Past Medical History
- Family History
- Drug History
- Psychosocial History
15Presenting Foot Complaint
- Foot problem could be
- Skin breakdown swelling colour changes pain
callosity toenail problem or footwear problem - Ask questions
- Where is the problem?
- When did it start?
- How did it start?
- How has it been treated so far?
16Presenting Foot Complaint
- Consider who is the best team member to deal with
the current problem and refer when necessary. - Pain as a symptom may be due to localised trauma
infection ischaemia or neuropathy - The next slide will help you distinguish between
pain from ischaemia and neuropathy
17Ischaemic or Neuropathic Pain?
- Ischaemic Pain
- Persistent pain
- Worse on elevation
- Relieved by dependency
- Pain in calf on exercise (claudication) relieved
by rest
- Neuropathic Pain
- Burning pains
- Contact pains due to sheets or other touch
- Sharp short shooting pains
- Pain relieved by cold
- Pain worse during rest
18Past Foot History
- Note previous ulceration and treatments
- Note any previous amputations
- Major
- Minor
- Reasons for amputations
- Osteomyelitis Necrosis Trauma
- Peripheral Angioplasties
- Peripheral Arterial Bypasses
19Diabetes History
- Type of Diabetes
- Type 1
- Type 2
- Duration of Diabetes
- Treatment of Diabetes
- Insulin
- Oral Hypoglycaemics
20Diabetes History
- Complications
- Retinopathy
- Nephropathy
- Cardiovascular
- Angina, Heart Failure, Myocardial Infarction
- Coronary artery angioplasty or bypass
- Cerebrovascular
- Transient Ischaemic attack (TIA)
- Stroke (CVA)
21Past Medical History
- Severe systemic conditions
- Cancer, Rheumatoid Arthritis etc
- Neurological conditions
- Epilepsy, Parkinsons disease etc
- Accidents
- Injuries
- Hospital Admissions
- Operations
22Drug History
- Present Medication
- Steroids, Anti-coagulants etc.
- Known Allergies or sensitivities
- Antibiotics
- Medications
- Dressings
- Adhesive dressings
23Family History
- Familial History of Diabetes
- Other serious illness
- Cause of death of near relatives
- Obesity
24Psychosocial History
- Occupation
- Smoker?
- Number of cigarettes smoked daily
- Drink Alcohol?
- Number of units drunk daily
- Psychiatric illness
- Home circumstance
- Type of accommodation Lives alone?
25Foot Examination
- Explain what you are doing for each test
- Look at the feet and legs from a distance away
and note any differences between the right and
left in terms of Deformity, Colour and Volume - Ask the patient whether they have any worries
about their feet - Take a short history of any relevant operations,
injuries, medication, that may impact on their
feet. (See History)
26Foot Examination
- Feel with the back of the hand the temperature of
the skin surface from the distal part of the foot
up the leg and then compare to the other foot and
leg. - Are there any differences? Cold spots or areas of
heat? (May lead you to think about ischaemia or
infection) - Note the absence of hair growth on the toes and
legs (non conclusive sign of ischaemia)
27Foot Examination
- Note any colour differences
- Redness (erythema) may lead to conclusions about
presence of infection. - Oedema may or may not be present in infection
dependant on how ischaemic the foot is. - Any suspicion of infection should be referred on
to an appropriate team member to decide on the
necessity for antibiotic therapy - Now go on to check the foot pulses
28Check the Foot Pulses
- Dorsalis Pedis Pulse
- Use index, middle and ring fingers together and
palpate the dorsum of the foot over the bony
prominence of the tarsal bones 4-5 cm proximal to
the space between the first and second toe just
lateral to the Extensor Hallucis longus tendon - Posterior Tibial Pulse
- Found below and behind the medial malleolus
29Check the Foot Pulses
- If you palpate either of these pulses it is
unlikely there is any significant ischaemia in
the foot - If both pulses are not palpable then check the
popliteal and femoral pulses - ABPI (Ankle Brachial Pressure Index) can be
undertaken but calcification of arteries in
Diabetes can lead to falsely high ABPI readings
so toe pressures could be undertaken (TBPI) - Pulse Oximetry comparison between finger and toe
can also be very useful (Parameswaran et al Arch
Intern Med Feb 2005 Vol1654 442-446)
30Check for Neuropathy
- The method of choice is the 10gm monofilament
applied perpendicular to the foot and pressure
applied until it bends - Sites to test
- Apex of first, third, and fifth toes and the ball
of the foot (MTP joints) of the same toes, dorsum
of foot and heel - Vibration sense tested on dorsum of first toe and
a site further proximal such as the lateral
malleolus using a 128-Hz tuning fork
31Check for Neuropathy
- When testing get the person to close their eyes
- Repeat the test three times at each site
- One of those three tests should be a non test
where the foot is not touched - This is to ascertain whether the person being
tested is telling you what they feel you want
to hear
32Foot Examination
- Next examine each foot more closely
- Check each toe and in between each toe
- Check the Toenails
- Check the skin
- Check for areas of deformity or swelling
- Check joint mobility
- Check colour signs of necrosis or ischaemia
- Check Footwear
33Toes
- Toes should be checked for shoe pressures and
callosities - Toes should be checked for deformities and advice
given as appropriate on footwear
34Toenails
- Toenails should be checked for infections
- Fungal Nail infections
- Toenails should be checked for ulceration
- Ulceration under the nail
- (sub ungual ulceration)
35Toenails
- Toenails should be checked for thickening
(Onychogryphosis) - Check for ingrown Toenails
36Skin
- Skin should be checked for any wounds or entry
points for infections - Skin should be checked for callosity and signs of
pressure
37Deformity and Oedema
- Check deformities are not under pressure from
footwear - Be aware of any oedema and its possible causes
- venous insufficiency
- infection
38Joint Mobility
- Check joint mobility as this may impact on
possible pressure points - Other conditions complicating Diabetes such as
Rheumatoid arthritis can result in poor joint
mobility and gross deformity
39Necrosis and Ischaemia
- Buergers Test
- Elevate the limb above heart level Turns White
- Then lower the limb to dependency Turns Purple
- Is a Indicator for Ischaemia
- Find Pulse with doppler then raise leg if pulse
sound reduces or stops then ischaemia is indicated
40Necrosis and Ischaemia
- Wet necrosis
- Usually appears as yellow/grey slough with
grey/pale pink base - Dry Necrotic Lesions should be noted and left dry
but monitor demarcation lines for signs of
infection
41Examine Footwear
- Check Footwear for
- Length, width depth
- Template Test
- Internal seams or rough edges
- Fastenings
- Fastenings to keep shoe on
- No fastenings means shoe is too tight or person
has to claw toes to keep shoes on - Heels
- Wide stable heel
- Not higher than 2.5cm
42Footwear Template Test
- To check for length and width
- Place foot on thin card (cereal pack will do)
- Draw around barefoot
- Cut out template
- Place inside shoes
- This will show if any edges of the template need
to deform to get the shoe on - Repeat for the other foot
- Show the person your findings
43Stages of Diabetic Foot
- Stage 1 Normal or Low Risk Foot
- Stage 2 High-Risk Foot
- Stage 3 Ulcerated Foot
- Stage 4 Infected Foot
- Stage 5 Necrotic Foot
- Stage 6 Unsalvageable Foot
- (Stages of Diabetic Foot Ref Edmonds, Foster,
Sanders 2004)
44Normal Foot
- No risk factors present
- Foot sensation good
- Foot pulses palpable
- No foot deformities
- No pathological callus
- No swelling
- Foot Assessment
45High Risk Foot
- Neuropathy and Ischaemia
- are the main risk factors for ulceration.
- Deformity, oedema and callus
- are risk factors that will not necessarily lead
to ulceration unless either or both of the main
risk factors are present
46High Risk Foot
- The Foot has developed one or more or the
following risk factors for ulceration - Neuropathy
- Ischaemia
- Deformity
- Oedema
- Callus
47Ulcerated Foot
- Foot ulcers can be less than 1mm or cover most of
the foot surface - But all foot ulcers large or small should be
taken very seriously - As they can deteriorate rapidly
48Infected Foot
- Managing Infection is a vital in treating foot
ulceration - Tissue samples are better than swabs to send for
culture and sensitivity, but if swabs are used
take from the base of the ulcer.
Spreading erythema from L/4th toe would strongly
suggest infection
49Necrotic Foot
- Necrosis can be wet or dry
- Wet Necrosis has slough which is soft, yellow or
grey in colour - Dry Necrosis has slough which is hard and black
Wet Necrotic Slough
Dry Necrotic Slough
50Unsalvageable Foot
- Sometimes when there is a great risk of Spreading
Osteomyelitis and or necrosis or the patients
life is in danger from Septicaemia then
Amputation is the only recourse
Ulcer probes to bone Necrosis and osteomyelitis
present
Amputation was performed within days
51Investigations
- Laboratory Tests
- Full Blood Count (FBC) to detect anaemia or
polycythaemia - Serum Electrolytes, urea and creatinine to assess
Renal function - Serum bilirubin, alkaline phosphatase, gamma
glutamyl transferase, and aspartame transaminase
to assess Liver function - Blood Glucose HbA1c to assess diabetes control
- Serum cholesterol and triglycerides to assess
arterial disease risk factors
52Investigations
- Radiological determined by clinical presentation
and not always necessary - X-ray to detect
- Osteomyelitis
- Fracture
- Charcot Foot
- Gas in soft tissues
- Foreign Body
53Investigations
- Foot Pressures
- These techniques measure pressure distributions
of the foot either out of shoe by walking across
pressure plates or by in shoe pressure
templates which are placed in the shoes and then
foot pressures recorded over time.
54Foot Risk Factors
55Neuropathy
56Ischaemia
57Callus
- Callus is an indication that an area of skin is
working harder than it is designed for. - In poor tissue viability the callus can ulcerate.
- Try to remove the causes of the callus
- Footwear can be a major cause of callus
- So can deformities or poor gait patterns
58Foot Deformity
One month later ulcer improved and infection
treated with antibiotics
Ulcer over exostosis deformity with infection
59Foot or Toe Deformities
- Deformities under the foot can add to pressures
when the patient is ambulant - Deformities to the top or the sides of the foot
can add to pressures from footwear - If surgical treatment of the deformity is
unwanted then accommodate the deformity by
referring for therapeutic footwear
60Oedema
This patients Lymphoedema has caused pressure
from footwear on the lateral border of the foot
61Foot Ulceration
62Ischaemia
- Ischaemia is the result of poor arterial supply
to the foot and leg. - Without a good blood supply skin will not heal
well and can lead to necrosis (gangrene)
Necrosis of the apex of toe
63Foot Health Education
64Self Assessment Tests
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this module? - If not then press the button to go back to review
the material again - However if you feel ready to be questioned then
press this button instead
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66Neuropathy
- Diabetic Peripheral Neuropathy is the presence of
symptoms and/or signs of peripheral nerve
dysfunction in people with diabetes, after
exclusion of other causes e.g. Multiple Sclerosis
(Boulton et al 1998) - This can be sub divided into
- Motor Neuropathy
- Painful Neuropathy
- Autonomic Neuropathy
- Sensory Neuropathy
- Charcot Neuroarthropathy
67Motor Neuropathy
- Diabetic amyotrophy
- This is a rare and unusual manifestation which
results in poor motor control of the leg muscles
usually bilateral leading to muscle weakness and
muscle wasting - Usually affects the quadriceps (anterior thigh)
- May also affect hamstrings (posterior thigh)
- Not always associated with sensory loss
- (Ref Kaplan Abourizk 1981)
68Painful Neuropathy
- A small number of people with diabetes complain
of - Burning pains or Shooting pains in the legs
- Sensation of overtight skin
- Toes can feel larger than they actually are
- Heightened awareness of sensations
- Feeling of walking on stones
- Warm/cold sensations in the feet
69Painful Neuropathy
- This is a nasty complication of diabetes that can
be underestimated by healthcare professionals as
it presents with little sign that anything is
wrong. - However patients in pain do not sleep well become
disturbed, confused and depressed as constant
unexplained pain can be miserable to bear.
70Painful Neuropathy
- Can be provoked when insulin is first started and
gradually improves if tight blood sugars are
maintained - Distribution usually bilateral (both legs)
- Usually worse at night and is not improved with
foot dependency unlike ischaemic rest pain which
is improved if the foot is lowered out of the bed
71Painful Neuropathy
- Management is reassurance as this condition
usually resolves within 2 years but may be
replaced with numbness - Therapies include
- Topical therapy
- Glycaemic control
- Drug therapy
- Physical treatments
72Painful Neuropathy
- Topical Therapy
- Some patients find relief from burning pains or
contact dysaesthesia with opsite film dressings
or opsite sprays though clingfilm worn on the
unbroken skin of the legs at night may be an
alternative. - Capsaicin applied as a cream
73Painful Neuropathy
- Glycaemic Control
- High blood sugars (Hyperglycaemia) is known to
lower the threshold for pain - So with diabetic painful neuropathy due regard
should be taken to HbA1c levels noting the normal
range to be 3.8 to 6.4 - Diabetic therapies should then be optimised
74Painful Neuropathy
- Drug Therapy
- This consists of analgesics, hypnotics, tricyclic
antidepressants, anticonvulsants and
antiarrythmics (used rarely) - Burning pain Tricyclic antidepressants e.g.
Amitriptyline or Imipramine (but be aware of
postural hypotension) - Anticonvulsants e.g. Gabapentin
75Painful Neuropathy
- Physical Therapy
- Transcutaneous electrical nerve stimulation TENS
- Can be used to block the pain stimulus with
electrodes either side of painful area - Acupuncture
- Anecdotally reported as useful
76Autonomic Neuropathy
- This is very common in the diabetic foot as the
autonomic nervous system controls the sweat
glands. - This type of neuropathy produces dry skin which
is liable to fissure and could lead to infections
and/or ulceration
77Autonomic Neuropathy
- As we can see from this picture of a heel, dry
skin can easily lead to fissuring and ulceration
78Autonomic Neuropathy
- So people with autonomic neuropathy should be
encouraged to use emollients on a daily basis - A small amount daily is much better than a big
dollop once a week - Aqueous Cream B.P. is very good and generally
more affordable that branded products such as
E45,Vaseline Intensive Care or Atrixo
79Sensory Neuropathy
- This is the most common form of Neuropathy in
diabetes. - Leads to a numb, painless foot insensitive to one
or all of - light touch deep touch heat and cold pain
sensation and vibration sensation - This can lead to problems of patients being
unaware of trauma to the feet.
80Sensory Neuropathy
- This type of neuropathy can lead to problems of
ulceration caused by - Mechanical Trauma
- Chemical Trauma
- Thermal Injury
81Sensory Neuropathy
- Mechanical Trauma
- Skin can be subject to shearing and compressive
mechanical stresses leading to corns or callus - Further stresses can then lead to tissue
breakdown under the callosity
82Sensory Neuropathy
- Mechanical Trauma
- The pain and/or pressure response is absent and
the pressure causing the lesions continues
without rest and leads to ulceration beneath the
callosity
83Sensory Neuropathy
- Chemical Trauma
- People are tempted to use medicated corn and
callus plasters which contain salicylic acid
which breaks down the keratin molecules within
the skin and theoretically softening the
callosity unfortunately this acid varies in
concentration and can lead to ulceration
84Sensory Neuropathy
- Thermal Injury
- This example is of a person with diabetic sensory
neuropathy who burnt his toe on the metal side of
a radiant heater. - As he felt no pain he did not realise the damage
that was occurring. So as he watched television
the skin was burnt then blistered and took 7
months to heal
85Sensory Neuropathy
- Sensory Neuropathy when recognised as present in
a person with diabetes should lead to an
education of that person by healthcare
professionals in how to care for themselves. - The main point of that education is DAILY FOOT
EXAMINATIONS by the patient or their carer.
86Sensory Neuropathy
- Prevention and recognition of any foot problem is
the primary responsibility of the person with
diabetes and their carers. - The daily foot examination is vital in the high
risk foot as injuries may have occurred without
the patient realising so they must look for any
wounds, injuries or blistering which may give an
access point to opportunist infections. - Then a immediate regime of antiseptic dressings
should be instituted by the patient or carer with
daily wound checking - If any sign of infection is present or if the
wound is large an immediate GP appointment should
be sought for antibiotic therapy with support
from the community nursing team instituted
87Foot Risk Factors
Ischaemia
Neuropathy
Callus
Deformity
88Economics of Diabetes