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BURNS

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(morbidity and mortality of burn clients is related to a lack of ... Debridement. Mechanical. Enzymatic. Cleaning. Stimulating granulation and revascularization ... – PowerPoint PPT presentation

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Title: BURNS


1
BURNS
2
Pathophysiology of Burn Injury
  • Tissue destruction can lead to
  • Fluid/protein losses
  • Sepsis
  • Multiple system disturbances
  • Metabolic
  • Endocrine
  • Respiratory
  • Cardiac
  • Hematologic
  • Immune

3
Pathophysiology of Burn Injury
  • Extent of local and systemic disruption depends
    on
  • Age
  • General health status
  • Extent of injury
  • Depth of injury
  • Area of body injured
  • (morbidity and mortality of burn clients is
    related to a lack of or delay in healing)

4
Burn Classifications
  • Superficial
  • Least destruction
  • Only epidermis injured
  • Partial-thickness
  • Epidermis destroyed
  • Varying depths of dermis damaged/destroyed
  • Superficial partial-thickness
  • Erythematous and moist with vesicles
  • painful
  • Deep partial-thickness
  • Red and waxy without blisters
  • Moderate edema, lesser degree of pain
  • Hypoxia and ischemia can cause extension of wound

5
Burn Classifications
  • Full-thickness
  • Entire epidermis and dermis involved
  • No viable epithelial cells, grafts required
  • Hard, dry leathery eschar
  • Deep full-thickness
  • Extend beyond skin into underlying fascia and
    tissues
  • Muscle, bone and tendon damage with exposure to
    surface
  • Blackened and depressed, little or no sensation
  • Early excision and grafting beneficial

6
Illustrations of Burns
  • Superficial partial-thickness
  • Deep partial-thickness

7
Illustration of Burns
  • Full Thickness
  • Deep Full Thickness

8
Vascular Changes
  • Fluid Shift
  • Period of inflammatory response
  • Vessels adjacent to burn injury dilate ? ?
    capillary hydrostatic pressure and ? capillary
    permeability
  • Continuous leak of plasma from intravascular
    space into interstitial space
  • Associated imbalances of fluids, electrolytes and
    acid-base occur
  • Hemoconcentration
  • Lasts 24-36 hours

9
Vascular Changes
  • Fluid remobilization
  • Capillary leak ceases and fluid shifts back into
    the circulation
  • Restores fluid balance and renal perfusion
  • Increased urine formation and diuresis
  • Continued electrolyte imbalances
  • Hyponatremia
  • Hypokalemia
  • Hemodilution

10
Other System Changes
  • Cardiac
  • Decreased cardiac output
  • Need fluid resuscitation and support with O2
  • Pulmonary
  • Respiratory insufficiency as a secondary process
  • Can progress to respiratory failure
  • Aggressive pulmonary toilet and oxygenation
  • Gastrointestinal
  • Decreased or absent motility (may need NG tube)
  • Curlings ulcer formation
  • H2 histamine blockers, mucoprotectants and
    enteral nutrition

11
Other System Changes
  • Metabolic
  • Hypermetabolic state
  • Increased oxygen and calorie requirements
  • Increase in core body temperature
  • Immunologic
  • Loss of protective barrier
  • Increased risk of infection
  • Suppression of humoral and cell-mediated immune
    responses

12
Compensatory Responses
  • Inflammatory Compensation
  • Initiates healing
  • Contributes to fluid shift (? capillary
    permeability)
  • Local tissue reaction due to release of chemicals
    by wbcs
  • Sympathetic Nervous System Compensation
  • Stress Response (Figure 68-8, p. 1562)

13
Phases of Burn Injury
  • Emergent
  • First 48 hours
  • Acute
  • Approximately 48 hours after injury to complete
    wound closure
  • Rehabilitative
  • Begins with wound closure and ends when client
    returns to highest possible level of functioning

14
Emergent Phase
  • Goals
  • Maintain open airway
  • Ensure adequate breathing/circulation
  • Limit extent of injury
  • Maintain function of vital organs
  • Prevent potential complications

15
Acute Phase
  • Interventions aimed at
  • Maintenance of cardiovascular/respiratory system
  • Nutritional status
  • Burn wound care
  • Pain control
  • Psychosocial interventions

16
Rehabilitative Phase
  • Emphasis
  • Psychological adjustment of client
  • Prevention of scars and contractures
  • Resumption of pre-burn activity
  • Work
  • Family
  • Social

17
Clinical Manifestations of Burns
  • Respiratory
  • Direct airway injury
  • Carbon monoxide poisoning
  • Thermal injury
  • Smoke poisoning
  • Pulmonary fluid overload
  • External factors
  • Cardiovascular
  • Hypovolemic shock and ? cardiac output
  • Impaired circulation/tissue perfusion
  • Potential for ECG changes

18
Clinical Manifestations
  • Renal/urinary
  • Changes R/T ? renal perfusion and debris
  • Fluid shift ? GFR and urine output
  • Fluid remobilization-- ? GFR and diuresis
  • Tubular blockage from myoglobin and uric acid
  • Fluid resuscitation should maintain output at
    30-50 mL/hour
  • Integumentary
  • Size of injury is important to diagnosis and
    prognosis
  • Rule of Nines
  • Lund-Browder method
  • Specific treatments dependent upon depth of injury

19
Burns Nursing Diagnoses
  • Decreased cardiac output
  • Deficient fluid volume
  • Ineffective tissue perfusion (multiple)
  • Ineffective breathing pattern
  • Pain
  • Excess fluid volume
  • Risk of ineffective thermoregulation
  • Disturbed sensory perception

20
Decreased CO, Deficient Fluid Volume,
Ineffective Tissue Perfusion
  • Interventions
  • Non-surgical
  • IV fluid therapy
  • Plasma exchange
  • Drug therapy
  • Surgical
  • Escharotomy

21
Ineffective Breathing Pattern
  • Interventions
  • Non-surgical
  • Airway maintenance
  • Promotion of ventilation
  • Monitoring gas exchange
  • Oxygen therapy
  • Drug therapy
  • Positioning and deep breathing
  • Surgical
  • Tracheostomy
  • Chest tubes
  • escharotomy

22
Acute Pain
  • Interventions
  • Non-surgical
  • Drug therapy (opioids) (anesthetic agents)
  • Complimentary/alternative therapies
  • Environmental manipulation
  • Surgical
  • Early surgical excision of burn wound

23
Impaired Skin IntegrityWound Care Management
  • Non-surgical
  • Debridement
  • Mechanical
  • Enzymatic
  • Cleaning
  • Stimulating granulation and revascularization
  • Dressings

24
Dressings
  • Standard
  • Multiple gauze layers over topical agent or
    antibiotic
  • Biologic (Table 68-8, p. 1578)
  • Homograft (allograft) from cadaver
  • Heterograft (xenograft) from animal (pig)
  • Amniotic membrane
  • Cultured skin
  • Artificial skin
  • Two-layer product which creates an artificial
    dermis
  • Synthetic dressing
  • Solid silicone and plastic membrane
  • Can see through to monitor wound status

25
Impaired Skin IntegrityWound Care Management
  • Surgical management
  • Surgical excision
  • Treatment of choice for deep partial-thickness
    wounds
  • Wound coverings
  • Permanent skin coverage by autograft
  • Split thickness
  • Successive reharvesting
  • Meshing of split thickness graft

26
Risk for Infection
  • Non-surgical management
  • Drug therapy
  • Tetanus Toxoid and Topical Antimicrobials
  • Organism specific drugs
  • Isolation
  • Environmental manipulation
  • Secondary prevention/early detection
  • Surgical management
  • Aggressive surgical incision of infected wound

27
Additional Interventions
  • Imbalanced Nutrition
  • Calculate calorie needs and provide adequate
    calories and nutrients
  • Calorie requirements can exceed 5000 per day
  • Impaired Mobility
  • Interventions to maintain pre-burn ROM and
    prevent contractures
  • Disturbed Body Image
  • Grief counseling
  • Encouraging independence
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