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GALL BLADDER

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gall bladder by dr. hayder m. abdulnabi md, cabs anatomy pear-shaped, 7.5-12.5 cm normal capacity- 50 ml fundus, body, neck (terminates in a narrow infunbibulum ... – PowerPoint PPT presentation

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Title: GALL BLADDER


1
GALL BLADDER
  • BY
  • DR.
  • HAYDER M. ABDULNABI
  • MD, CABS

2
ANATOMY
  • PEAR-SHAPED, 7.5-12.5 CM
  • NORMAL CAPACITY- 50 ML
  • FUNDUS, BODY, NECK (TERMINATES IN A NARROW
    INFUNBIBULUM)
  • ( HARTMANNS POUCH- A DILATATION IN THE NECK DUE
    TO AN IMACTED STONE)
  • CRISS-CROSS MUSCLE COAT (WELL DEVELOPED IN THE
    NECK)
  • GLANDULAR MUCOUS MEMBRANE WITH CRYPTS OF LUSCHA

3
  • THE CYSTIC DUCT 2.5 CM (CONTAINS THE SPIRAL VALVE
    OF HEISTER)
  • THE COMMON HEPATIC DUCT 2.5CM (UNION OF RT AND LT
    HEPATIC DUCTS)
  • THE COMMON BILE DUCT 7.5CM (JUNCTION OF CHD AND
    THE CYSTIC DUCT), OF 4 PARTS

4
  • 1- SUPRADUODENAL 2.5CM (RUNS IN THE FREE EDGE OF
    LESSER OMENTUM
  • 2- RETRODUODENAL
  • 3- INFRADUODENAL
  • 4- INTRADUODENAL (PASSES OBLIQUELY THROUGH 2ND
    PART OF DUODENUM, SURROUNDED BY THE SPHINCTER OF
    ODDI, OPENS AT THE SUMMIT OF THE PAPILLA OF VATER

5
THE ARTERIAL SUPPLY OF THE GALL BLADDER
  • THE CYSTIC ARTERY (BRANCH OF THE RT HEPATIC
    ARTERY), USUALLY BEHIND THE CBD
  • ACCESSORY CYSTIC ARTERY (OCCASIONAL)(BRANCH OF
    THE GASTRODUODENAL ARTERY)

6
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7
LYMPHATICS
  • SUBSEROSAL AND SUBMUCOSAL DRAIN INTO THE CYSTIC
    LYMPH NODE OF LUND (SENTINEL LN) THEN TO THE
    HILUM OF THE LIVER TO THE COELIAC LYMPH NODES
  • SUBSEROSAL LYMPHATICS CONNECT WITH THE
    SUBCAPSULAR LYMPHATICS OF THE LIVER (FREQUENT
    SPREAD OF GALL BLADDER CA TO THE LIVER)

8
FUNCTIONS OF THE GALL BLADDER
  • BILE IS COMPOSED OF 97 WATER, 1-2 BILE SALTS,
    1 PIGMENTS, CHOLESTEROL AND FATTY ACIDS
  • LIVER EXCRETION RATE IS 40 ML/HOUR
  • 1- RESERVOIR (FASTING CAUSE RESISTANCE INCREASE
    IN SPHINCTER OF ODDI) (FEEDING DECREASE THE
    RESISTANCE AND THE GALL BLADDER CONTRACTS BY THE
    ACTION OF CHOLECYSTOKININ RELEASED BY UPPER
    INTESTINAL MUCOSA IN RESPONSE TO FOOD
    PARTICULARLY FAT)

9
  • 2- CONCENTRATION OF BILE 5-10 TIMES ( BY ACTIVE
    ABSORBTION OF WATER, SOD. CHLORIDE, AND
    BICARBONATE) WITH INCREASE IN THE PROPORTION OF
    BILE SALTS, PIGMENTS, CHOLESTEROL AND CALCIUM
  • 3- MUCIN SECRETION, 20ML/HOUR

10
INVESTIGATIONS OF THE BILIARY TRACT
  • 1- PLAIN RADIOGRAPH-- (RADIO-OPAQUE STONE 10,
    PORCLAIN GALL BLADDER, LIMEY BILE, AIR)
  • 2- ORAL CHOLECYSTOGRAPHY-- (A CONTROL X-RAY IS
    TAKEN THE DAY BEFORE AND IOPANOIC ACID CONTRAST
    MEDIUM TABLETS IS TAKEN ORALLY AT NIGHT, THE NEXT
    DAY ERRECT AND SUPINE X-RAY IS TAKEN TO THE RT
    HYPOCHONDRIUM AND X-RAY REPEATED TO OBSERVE GALL
    BLADDER CONTRACTION(

11
RADIO-OPAQUE STONES
PLAIN X- RAY
12
PLAIN X-RAY
PORCLAIN GB
13
AIR
PLAIN X-RAY
14
ORAL CHOLECYSTOGRAM
STONES
15
  • NONVISUALIZATION (NONFUNCTIONING) GALL BLADDER IS
    DUE TO-- FAILURE OF THE PATIENT TO TAKE THE
    TABLETS, VOMITING, MALABSORBTION, IMPAIRED LIVER
    FUNCTION, BLOCKED CYSTIC DUCT,SEVERE GALL BLADDER
    DISEASE (FAILURE OF CONCENTRATION)

16
  • 3- INTRAVENOUS CHOLANGIOGRAM USING INTRAVENOUS
    RADIO-OPAQUE MEDIUM TO SHOW THE BILE DUCTS, MAY
    BE USED WITH ORAL CHOLECYSTOGRAM OR TOMOGRAPHY (A
    METHOD TO PUT ONE GIVEN PLANE INTO SHARP FOCUS
    WHILE BLURRING OTHERS)

17
  • 4- ULTRASONOGRAPHY (NONINVASIVE)
  • AND SHOWS BILIARY CALCULI, DILATION OF BILIARY
    TREE,CA HEAD PANCREAS, WALL THICKNESS, GALL
    BLADDER SIZE, HALLO SIGN
  • 5- RADIOISOTOP SCANNING USING RADIOACTIVE
    IODINE(131) OR Tc(99)
  • 6- COMPUTED TOMOGRAPHY IN OBESE OR PATIENTS WITH
    GASEOUS DISTENTION THAT MAKE ULTRASONOGRAPHY
    DIFFICULT

18
GB
STONE
ACOSTIC SHADOW
US
19
STONE
CBD
ACOSTIC SHADOW
ULTRASONOGRAPHY
20
  • 7- ENDOSCOPIC RETROGRADE CHOLAGIOPANCREATOGRAPHY
  • (ERCP) BY CANNULATION OF THE AMPULLA OF VATER
    USING FIBEROPTIC DUODENOSCOPE AND INJECTION OF
    CONTRAST MEDIUM ,TO TAKE SAMPLE FOR CULTURE AND
    BRUSHING FOR CYTOLOGY. ITS USE CAN BE EXTENDED TO
    DO PAPILLOTOMY TO EXTRACT STONES, PASSING
    CATHETER OR DORMIA BASKET, AND STENT PLACING
    THROUGH STRICTURES.
  • IT MAY CAUSE ASCENDING BILIARY INFECTION, SO
    SHOULD BE DONE UNDER ANTIBIOTICS COVER

21
DUCT OF WIRSUNG
CATHETER IN THE AMPULLA
ERCP
22
  • 8- PERCUTANEOUS TRANSHEPATIC CHOLANGIOGRAPHY-
    INJECTION OF CONTRAST MEDIUM THROUGH A CHIBA OR
    OKUDA NEEDLE (15CM LONG , 0.7MM IN DIAMETER) INTO
    THE LIVER THROUGH THE 8TH INTERCOSTAL SPACE IN
    THE MIDAXILLARY LINE.
  • IT CAN BE USED TO PUT A CATHETER FOR DRAINAGE
    OR STENT FOR ANTEGRADE DRAINAGE.
  • BLEEDING TENDENCY IS A CONTRA INDICATION AND
    THE PROCDURE SHOULD BE DONE UNDER ANTIBIOTICS
    COVER

23
CHIBA NEEDLE
PER CUTANEOUS TRANSHEPATIC CHOLANGIOGRAM
24
  • 9- PEROPERATIVE CHOLANGIOGRAPHY BY TAKING X-RAY
    DURING OPERATION AFTER INJECTING THE CONTRAST BY
    A POLYTHENE CATHETER INTRODUCED INTO THE CBD
    THROUGH AN OPENING IN THE CYSTIC DUCT TO DETECT
    ANY STONE IN THE CBD BEFORE EXPLORATION.
  • FAILURE OF THE CONTRAST TO ENTER THE DUODENUM
    MAY BE ALSO DUE TO SPHINCTER SPASM AND HERE
    SUCCINYLCHOLINE IS GIVEN TO EXCLUDE THIS
    POSSIBILITY
  • 20 OF CASES THE MEDIUM ENTER THE DUCT OF
    WIRSUNG AND IT IS NOT NECESSARILY PATHOLOGICAL

25
CATHETER
PER- LAPAROSCOPIC CHOLANGIOGRAPHY
26
CATHETER
CBD
DUODENUM
PER-OPERATIVE CHOLANGIOGRAM
27
  • 10- OPERATIVE BILIARY ENDOSCOPY (CHOLEDOCHOSCOPY)
  • 11- PEROPERATIVE POSTEXPLORATORY CHOLANGIOGRAPHY
    (THROUGH THE T- TUBE)
  • 12- POSTOPERATIVE CHOLANGIOGRAPHY (T-TUBE), 10-14
    DAYS AFTER CHOLEDOCHOTOMY

28
STONE IN CBD
PER-OPERATIVE CHOLANGIOGRAPH
29
Rt HEPATIC DUCT
Lt HEPATIC DUCT
PER-OPERATIVE CHOLEDOCHOSCOPE
30
STONE IN COMMON HEPATIC DUCT
T-TUBE
T-TUBE CHOLANGIOGRAM
31
CONGENITAL ANOMALIES OF THE GALL BLADDER AND BILE
DUCTS
  • 1. ANOMALIES OF THE GALL BLADDER- ABSENCE
  • PHRYGIAN CAP (HAT OF THE PEOPLE OF PHRYGIA IN
    ANCIENT ASIA MINOR)
  • (FRENCH REVOLUTION LIBERTE CAP)
  • FLOATING GALL BLADDERTORTION
  • DOUBLE GALL BLADDER

32
  • 2. ANOMALIES OF THE DUCTS-
  • ABSENCE
  • ATRESIA
  • CONGENITAL DILATATION OF INTRAHEPATIC DUCTS
  • CHOLEDOCHAL CYST
  • LOW INSERTION OF CYSTIC DUCT
  • ACCESSORY CHOLECYSTOHEPATIC DUCT

33
  • 3. ANOMALIES OF THE ARTERIES-
  • RT HEPATIC ARTERY AND OR CYSTIC ARTERY CROSS
    IN FRONT OF THE CHD
  • HEPATIC ARTERY TAKE A TORTOUS COARSE IN FRONT
    OF THE ORIGIN OF THE CYSTIC DUCT
  • RT HEPATIC ARTERY IS TORTOUS AND THE CYSTIC
    ARTERY IS SHORT (CATERPILLAR TURN)
  • ACCESSORY CYSTIC ARTERY

34
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35
GALL STONES(CHOLELITHIASIS)
  • MIXED STONES- 90, CHOLESTEROL IS THE MAJOR
    COMPONENT, Ca CARBONATE, Ca PHOSPHATE, Ca
    PALMITATE AND PROTEIN (USUALLY MULTIPLE AND
    FACETED)
  • 2. CHOLESTEROL STONES- (CHOLESTEROL SOLITAIRE)
  • 3. PIGMENT STONES- (SMALL, BLACK, MULTIPLE)

36
MIXED STONES
37
MIXED STONES
38
CHOLESTEROL STONES
39
PIGMENTSTONES
40
  • LIMEY BILE- OCCUR WHEN THERE IS GRADUAL
    OBSTRUCTION TO THE CYSTIC DUCT OR THE CBD
    (CHRONIC PANCREATITIS, CA PANCREAS)
  • THE GALL BLADDER WILL BE OPAQUE IN A PLAIN X-RAY
    (FILLED BY Ca CARBONATE AND Ca PHOSPHATE) WHICH
    IS THE COMPONENTS OF TOOTH PASTE

41
  • CHOLESTEROL IS HELD IN SOLUTION BY THE DETRERGENT
    EFFECT OF BILE SALTS AND PHOSPHOLIPID
    (LECITHINE)TO FORM MICELLES.
  • ANY CHANGE IN THE EQUILIBRIUM BETWEEN THESE
    THREE ELEMENTS WILL LEAD TO GALL STONE FORMATION

42
HYDROPLYLIC END
HYDROPHOBIC END (CHOLESTEROL)
BILE SALT MICELLE
43
PATHOGENESIS OF GALL STONE FORMATION
  • METABOLIC- INCREASE CHOLESTEROL LEVEL IN
    BILE(SUPERSATURATED OR LITHOGENIC BILE), WITH
    AGE, FEMALE ( CONTRCEPTIVE PILLS), OBESITY,
    PATIENTS ON CLOFIBRATE
  • BILE SALTS DECREASE BY INTERRUPTION OF
    ENTERO-HEPATIC CIRCULATION( ILEAL DISEASSE,
    RESECTION, BYPASS SURGERY, CHOLESTYRAMINE)
  • ESTROGEN DECREASE CONCENTRATION OF BILE SALT IN
    THE BILE(CCP)

44
CHOLESTEROL SOLUBILITY STATUS
45
  • 2. INFECTION- NIDUS
  • 3. BILE STASIS- GALL BLADDER CONTRACTILITY
    DECREASE IN PREGNANCY, BY ESTROGEN(CCP), AFTER
    TRUNCAL VAGOTOMY, PATIENTS ON TPN ( LACK OF GOOD
    ORAL INTAKE) CAUSE DECREASE IN CHOLYCYSTOKININ
    SECRETION

46
  • 4. PIGMENT STONES OCCUR WITH HEMOLYSIS(
    HEREDITARY SPHEROCYTOSIS, SICKLE CELL ANEMIA,
    THALASSEMIA, MALARIA)
  • WHERE BILIRUBIN PRODUCTION WILL INCREASE.
  • PIGMENT STONES ALSO INCEASE WITH BENIGN AND
    MALIGNANT STRICTURES AND WITH PARASITE
    INFESTATION OF THE BILIARY DUCTS( ASCARIS
    LUMBRICOIDES, CHLONORCHIS SINENSIS)

47
INCIDENCE OF GALL STONES
  • FAT, FERTILE, FLATULENT, FEMALE, FIFTY- IS THE
    USUAL SUFFERER OF GALL STONES
  • IT CAN OCCUR AT ANY AGE AND IN BOTH SEXES
  • TOW THIRD ARE ASYMPTOMATIC
  • SAINTS TRIAD- GALL STONES
  • DIVERTICULOSIS
  • HIATUS HERNIA

48
COMPLICATIONS OF GALL STONES
  • 1.IN THE GB- SILENT( NO INDICATION FOR OPERATION)
  • CH CHOLECYSTITIS
  • AC CHOLECYSTITIS
  • GANGRENE
  • PERFORATION
  • EMPYEMA
  • MUCOCELE
  • CARCINOMA
  • 2. IN THE BILE DUCTS-
  • OBSTRUCTIVE JAUNDICE
  • CHOLANGITIS
  • ACUTE PANCREATITIS
  • 3. IN THE INTESTINE-
  • ACUTE INTESTINAL
    OBSTRUCTION (GALL STONE ILEUS)

49
CHRONIC CALCULOUS CHOLECYSTITIS
  • THICK, FIBROTIC WALL, BACTERIA ISOLATED IN LESS
    THAN 30 OF CASES FROM THE BILE AND SUGGESTS A
    CHEMICAL IRRITANTS IN THE BILE RATHER THAN
    BACTERIAL AS A CAUSE IN THE OTHER CASES

50
CHRONIC CHOLECYSTITIS
51
SIGNS AND SYMPTOMS
  • Rt HYPOCHONDRIAL PAIN-
  • DISCOMFORT TO EXCRUTIATING PAIN(BILIARY COLIC)
  • RIADITES TO THE Rt SHOULDER
  • PRESIPITATED BY FATTY MEAL
  • ASSOCIATED BY NAUSEA AND VOMITING
  • TENDERNESS IN THE Rt HYPOCHONDRIUM
  • MURPHYS SIGN MAY BE POSITIVE
  • (IF PAIN LASTS MORE THAN 12 HOURS, TEPERATURE
    INCREASE, AND WBC INCREASE, CONSIDER THE
    DIAGNOSIS OF AC CHOLECYSTITIS)

52
DIAGNOSIS
  • ULTRASONOGRAPHY IS USUALLY THE ONLY INVESTIGATION
    REQUIRED
  • TREATMENT
  • ANALGESICS INCLUDING OPIATES (SIMULTANEOUS
    INJECTION OF HYOSCINE BUTYLBROMIDE IS NEEDED TO
    ENCOUNTER THE EFFECT OF OPIATES ON THE SPHINCTER
    OF ODDI)
  • ANTIEMETICS
  • LOW FAT DIET UNTIL------
  • CHOLECYSTECTOMY
  • (DISSOLUTION OF GALL STONES HAS NO LONGER A ROLE
    IN THE TREATMENT OF GALL STONES)

53
ACUTE CALCULOUS CHOLECYSTITIS
  • THE GALL BLADDER OFTEN ALREADY AFFECTED BY
    CHRONIC CHOLECYSTITIS
  • 95 OF CASES THE STON IS IMPACTED IN THE
    HARTMANNS POUCH OR OBSTRUCTING THE CYSTIC DUCT
  • MICRO-ORGANISMS CAN BE ISOLATED IN MOST OF THE
    CASES FROM THE BILE OR GB WALL
  • (E.COLI, STRTEP.FECALIS, BACTEROIDES, RARELY
    CLOSTRIDIA AND TYPHOID)

54
ACUTE CHOLECYSTITIS
55
SEQUELAE OF ACUTE CHOLECYSTITIS
  • RESOLUTION- BY BACK SLIPPING OF THE STONE(MUCOUS
    MEMBRANE LIFTING), AND RELEASE OF MUCOID OR
    MUCOPURULENT CONTENT
  • 2. EMPYEMA(PYOCELE)- WHEN THE OBSTRUCTION
    PERSISTS
  • 3. PERFORATION- LEADS TO LOCAL ABSCESS OR
    GENERALIZED PERITONITIS
  • (FUNDUS AND NECK)

56
SIGNS AND SYMPTOMS
  • PAIN
  • NAUSEA AND VOMITING
  • PYREXIA(38C OR MORE)
  • TENDERNESS
  • MURPHYS SIGN
  • PALPABLE GB
  • BOASS SIGN

57
DIAGNOSIS
  • ULTRASONOGRAPHY
  • DIFFERENTIAL DIAGNOSIS
  • APPENDICITIS
  • PERFORATED PEPTIC ULCER
  • ACUTE PANCREATITIS
  • ACUTE PYELONEPHRITIS (Rt)
  • MYOCARDIAL INFARCTION
  • BASAL PNEUMONIA (Rt)

58
TREATMENT
  • 1.CONSERVATIVE TREATMENT FOLLOWED BY
    CHOLYCYSTECTOMY
  • (90 OF CASES WILL SUBSIDE) BY
  • A. NASOGASTRIC ASPIRATION
  • B. I V FLUID
  • C. ANALGESIA
  • D. ANTIBIOTICS (AGAINST GRAM -NEGATIVE AEROBES)
  • C. INTERVAL CHOLECYSTECTOMY (4-6 MONTHS)
  • AFTER THE ACUTE EPISODE HAS RESOLVED

59
  • 2. EARLY CHOLECYSTECTOMY SHOULD BE DONE WITH IN
    72 HOURS FROM THE ONSET OF ACUTE SYMPTOMS (GOLDEN
    PEROID)
  • 3. EMERGENCY CHOLECYSTECTOMY- DONE AT ANY TIME
    NEEDED, WHEN DIAGNOSIS IS DOUBTFUL(ACUTE HIGH
    RETROCAECAL APPENDICITIS)
  • OR WHEN THERE IS PERFORATION

60
MUCOCELE AND EMPYEMA
  • MUCOCELE- THE BILE IS ABSORBED AND REPLACED BY
    MUCIN SECRETION(STERILE BLADDER NECK OBSTRUCTION
    BY A STONE OR MALIGNANCY)
  • EMPYEMA- GALL BLADDER FILLED WITH PUS EITHER AS
    A SEQUELE OF AC CHOLECYSTITIS OR A MUCOCELE
    BECOME INFECTED

61
MUCOCELE OF THE GB
62
MUCOCELE OF THE GB WITH STONE IN THE HART. POUCH
63
ACALCULOUS CHOLECYSTITIS
  • CHOLECYSTOSIS
  • NOT UNCOMMON GROUP OF CHRONIC INFLAMATION AND
    HYPERPLASIA OF ALL TISSUE ELEMENT-
  • CHOLESTEROSIS(STRAWBERRY GB)- WITH A STRAWBERRY
    INTERIOR AND YELLOW SPECKS (SEEDS OF CHOLESTEROL
    CRYSTALS)
  • CHOLESTEROL POLYPS- MUCH LESS NUMEROUS AND LARGER
    THAN THE YELLOW SEEDS
  • CHOLYCYSTITIS GLANDULARIS PROLIFERANS-
  • (POLYPS, ADENOMYOMATOSIS, INTRAMURAL
    DIVERTICULOSIS)

64
NEW TECHNIQUES FOR GALL STONES
  1. LITHOTRIPSY- EXTRACORPORIAL SHOCK WAVE
  2. PERCUTANEOUS CHOLECYSTOLITHOTOMY- USING A
    NEPHROSCOPE UNDER US CONTROL
  3. LAPAROSCOPIC CHOLECYSTECTOMY
  4. MINICHOLECYSTECTOMY

65
INDICATIONS FOR CHOLEDOCHOTOMY AT CHOLECYSTECTOMY
  1. STONES FELT IN THE CBD
  2. THERE IS JAUNDICE OR HISTORY OF JAUNDICE OR
    RIGOR(CHOLANGITIS)
  3. DILATED CBD(10mm OR MORE)
  4. ABNORMAL LFT IN PARTICULAR A RAISED ALKALINE
    PHOSPHATASE
  5. PRESENCE OF SINGLE FACTED STONE IN THE GALL
    BLADDER

66
POSTCHOLECYSTECTOMY SNDROME
  • PERSISTENCE OF SYMPTOMS AFTER GALL BLADDER
    REMOVAL DUE TO-
  • DISEASES OTHER THAN THE BILIARY TRACT(HIATUS
    HERNIA, PEPTIC ULCER, PANCREATITIS,
    DIVERTICULITIS OR IRRITABLE BOWWEL SYNDROME)
  • 2. BILIARY CAUSES- A- RETAINED STONE IN THE
    CBD
  • B- LONG CYSTIC
    DUCT STUMP IS LEFT
  • C- CBD OPERATIVE
    DAMAGE (STRICTURE FORMATION)

67
STONES IN THE COMMON BILE DUCT
  • EITHER SECONDARY DUE TO PASSAGE OF STONES FROM
    THE GALL BLADDER OR RARELY PRIMARY STONES OCCUR
    WITH IFESTATION OF THE BILIARY TREE BY ASCARIS
    LUMBRICOIDES AND CLONORCHIS SINUNSIS.
  • THESE STONES EITHER LEAD TO OBSTRUCTION OR
    INFECTION)CHOLANGITIS)

68
SIGNS AND SYMPTOMS
  • ASYMPTYMATIC
  • PAIN
  • JAUNDICE (INTERMITTENT OR PERSISTENT)(DARK
    URINE,PALE STOOL, PRURITIS)
  • FEVER AND RIGOR (CHOLANGITIS)
  • (CHARCOTS TRIAD)
  • TENDERNESS
  • IMPALPABLE GB (FIBROTIC AND INCOMPLETE
    OBSTRUCTION)
  • COURVOISIERS LAW

69
DIFFERENTIAL DIAGNOSIS
  • PANCREATIC CA
  • VIRAL HEPATITIS
  • DRUG INDUCES
  • PRIMARY BILIARY CIRRHOSIS
  • DIAGNOSIS
  • US, ERCP, PTC
  • COMPLICTIONS
  • BILIARY CIRRHOSIS
  • SUPPURATIVE CHOLANGITIS (LIVER ABSCESSES,
    SEPTICAEMIA)

70
PRE-OPERATIVE MANAGEMENT OF OBSTRUCTIVE JAUNDICE
  • 1. HIGH INTAKE OF GLUCOSE (BUILD UP LIVER
    GLYCOGEN STORE)
  • 2. VITAMIN K (FAT SOLUBLE), 10mg IV OR IM
  • 3. ANTIBIOTICS (BROAD SPECTURUM)
  • 4. HYDRATION (PEVENT RENAL FAILURE) (5 DEXTROSE
    TO ENSURE 30 ml/HOUR URINE FLOW)

71
SURGICAL PROCDURES
  • 1. ENDOSCOPIC PAPILLOTOMY (DORMIA BASKET, BALLOON
    CATHETER)(STENT TO RELIEVE SYMPTOMS)
  • 2. PERCUTANEOUS REMOVAL OF STONES BY BURHENNE
    METHOD (T- TUBE LEFT FOR SIX WEEKS AND THEN
    REMOVED, DILATION OF THE MATURE TRACT, STEERABLE
    CATHETER, AND THEN STONE BASKET)
  • 3. PERCUTANEOUS BILIARY DRAINAGE (PTC), IN THE
    VERY ILL
  • 4. SUPRADUODENAL CHOLEDOCHOTOMY WITH OR WITH OUT
    TRANSDUODENAL SPHINCTEROTOMY OR
    CHOLEDOCHODUODENOSTOMY

72
EXPLORATION OF THE CBD
73
DILATED CBD
DORMIA BASKET
ERCP
74
STRICTURE OF THE CBD
  • BENIGN POSTOPERATIVE 80
  • INFLAMMATORY
  • MALIGNANT
  • POSTOPERATIVE STRICTURE
  • DUE TO TEQUNICHAL ERROR DURING CHOLECYSTECTOMY(
    15 ONLY RECOGNIZED DURING SURGERY)

75
  • CAUSES- 1. BLIND HAEMOSTAT APPLICATION IN AN
    EFFORT TO STOP UNEXPECTED BLEEDING ( PRINGLES
    MANOEUVRE )
  • 2. TOO MUCH TRACTION ON THE GB
    3. FAILURE TO IDENTIFY CALOTS TRIANGLE(MUCH
    INFLAMMATION)
  • 4. IGNORANCE OF THE ANATOMICAL
    ANOMALIES
  • 5. LACERATION OF CBD (DURING
    EXPLORATION)
  • PRESENTED EITHER AS A- PROFUSE BILIARY FISTULA OR
    BILIARY PERITONITIS (DRIN OR NO DRAIN)
    B- DEEPENING JAUNDICE (BY
    SUSEQUENT FIBROSIS)

76
INVESTIGATION
  • US, T-TUBE CHOLANGIOGRPHY, ERCP, PTC
  • TREATMENT
  • IMMEDIATE ROUX EN Y CHOLEDOCHOJEJUNOSTOMY IS THE
    BEST FOR BENIGN STRICTURES AND COMPLETE CBD
    TRANSECTION
  • IN DEBILITATING PATIENTS, AN EXTERNAL DRAINAGE
    CATHETER OR BALLOON DILATION AND A STENT
  • FOR MALIGNANT STRICTURES CHOLECYSTOJEJUNOSTOMY
  • CHOLEDOCHOJEJUNOSTOMY
  • STENTING

77
CARCINOMA OF THE BG
  • IT IS RARE AND FOUND IN LESS THAN 1 OF GB
    OPERATIONS, GALL STONES FOUND IN OVER 90 OF
    CASES, PATIENTS USUALLY IN THEIR 70S, FEMALEMALE
    RATIO OF 51
  • THE USUAL TYPE IS SCIRRHOUS CA, BUT SEQUAMOUS OR
    MIXED SEQUAMOUS-ADENOCARCINOMA MAY BE FOUND
  • SPREAD BY DIRECT INVASION OF THE LIVER AND TO THE
    PORTA HEPATIS
  • DISTANT METASTASES ARE UNCOMMON

78
SIGNS AND SYMOTOMS
  • IT MAY BE FOUND DURING CHOLECYSTECTOMY
  • MASS DUE TO THE TUMOUR OR OBSTRUCTION OF CYSTIC
    DUCT WHICH LEADS TO MUCOCELE
  • CHOLECYSTITIS(OBSTRUCTION OF THE CYSTIC DUCT)
  • JAUNDICE IN MORE THAN 50 OF CASES

79
TREATMENT
  • RESECTION OF THE GB WITH THE ADGACENT PART OF THE
    LIVER
  • PALLATION TO RELIEVE JAUNDICE(STENT)
  • 5 - YEAR SURVIVAL RATE IS 2-5, BUT IF THE TUMOUR
    FOUND DURING CHOLECYSTECTOMY, IT WILL REACH MORE
    THAN 50

80
CHOLANGIOCARCINOMA(BILE DUCT CARCINOMA)
  • IT IS MORE COMMON THAN GB CARCINOMA
  • STONES PRESENT IN LESS THAN 30 OF CASES
  • MALE ARE SLIGHTLY MORE THAN FEMALE
  • USUALLY ADENOCARCINOMA
  • THE PATIENTS ARE OLD AND PRESENTS LATER

81
TRATMENT
  • HILAR LESIONS RARELY RESECTABLE,
  • AND MAY NEED EXTERNAL DRAINAGE FOLLOWED BY
    RADIOTHERAPY
  • TUMOURS OF THE LOWER END MAY BE TREATED BY
    WHIPPLES OPERATION, OR STENTING

82
BILIARY FISTULAS
  • EXTERNAL AND INTERNAL
  • 1 .EXTERNAL FISTULAS- NEARLY ALL FOLLOW BILIARY
    OPERATION ON THE BILIARY TRACT OR DUODENUM, FROM
    INJURY OR LEAKINK ANASTOMOSIS
  • IT MAY PERSIST IF THERE IS DISTAL OBSTRUCTION
  • CAN BE ASSESSED BY SINOGRAM OR ERCP
  • 2. INTERNAL FISTULAS- WHEN A GALL STONE ULCERATE
    THROUGH THE GB INTO THE STOMACH, DUODENUM, OR
    COLON
  • IT MAY CAUSE AIR TO BE SEEN IN PLAIN RADIOGRAPH
  • IF LARGE ENOUGH, IT MAY LEAD TO SMALL BOWEL
    OBSTRUCTION
  • OBSTRUCTION OF THE COLON GIVES THE SUSPITION OF
    UNDERLYING CARCINOMA CAUSING NARROWING OF THE
    LUMEN

83
LAPAROSCOPIC CHOLECYSTECTOMY
  • THE INDICTION ARE THE SAME AS FOR OPEN
    CHOLECYSTECTOMY
  • ADVANTAGES
  • 1. LESS POST-OPERATIVE PAIN
  • 2. SMALLER INCISIONS
  • 3. BETTER COSMESIS
  • 4. SHORTER HOSPITALIZATION
  • 5. EARLIER RETURN TO FULL ACTIVITY
  • 6. DECREASED TOTAL COSTS

84
DISADVANTAGES
  • 1. LACK OF DEPTH PERCEPTION
  • 2. VIEW IS CONTROLLED BY CAMERA
  • 3. MORE DIFFICULT TO CNTROL BLEEDING
  • 4. DECREASD TACTILE DISCRIMINATION
  • 5. POTENTIAL CO2 INSUFFLATION COMPLICATIONS
  • 6. ADHESIONS AND INFLAMMATION LIMIT ITS USE
  • 7. SLIGHT INCREASE IN BILE DUCT INJURY

85
COMPLICATIONS OF LC
  • A. GENERAL- 1. HEMORRHAGE
  • 2. BILE DUCT INJURY
  • 3. BILE LEAK
  • 4. RETAINED STONES
  • 5. PANCREATITIS
  • 6. WOUND INFECTION

86
  • B. PNEUMOPERITONEUM RELATED
  • 1. C02 EMBOLISM
  • 2. VASO-VAGAL RFLEX
  • 3. CARDIAC ARRYTHMIAS
  • 4. HYPERCARBIC ACIDOSIS
  • C. TROCAR RELATED
  • 1. ABDOMINAL WALL
    BLEEDING, HEMATOMA
  • 2. VISCERAL INJURY
  • 3. VASCULAR INJURY

87
LC THEATRE
88
VERES NEEDLE
89
TELESCOPE
90
DISSECTING CALOTS TRIANGLE
91
GB DISSEC. BY DIATHERMY
92
GB RETRIEVAL BAG
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