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Anorexia Nervosa (AN)

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Anorexia Nervosa (AN) Symptoms & Cause AQA A: Specifications states: cover only one eating disorder Dieting to be beautiful can go disastrously wrong – PowerPoint PPT presentation

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Title: Anorexia Nervosa (AN)


1
Anorexia Nervosa (AN)
Symptoms Cause
  • AQA A Specifications states cover only one
    eating disorder

Dieting to be beautiful can go disastrously
wrong
2
Clinical Characteristics of Anorexia Nervosa
(AN) DSM IV tr
  • Nervous loss of appetite.
  • Display an abnormal attitude towards food.
    (Eating Attitudes Test E.A.T.)
  • Primarily a female disorder, usually occurring
    during adolescence.
  • There is a refusal to maintain normal body
    weight.
  • Individuals need to weigh less than 85 of their
    normal body weight to be diagnosed as anorexic
    (Body Mass Index or BMI check online)
  • The distorted body image is not evident to
    anorexics themselves (Body Dismorphia Disorder or
    BDD)

Online http//psychcentral.com/quizzes/eat.htm
3
Secondary symptoms Anorexia causes a general
physical decline including
  • Cessation of menstruation (amenorrhoea)
  • Low blood pressure
  • Dry and cracking skin
  • Constipation
  • Insufficient sleep
  • Depression and low self-esteem
  • Up to 20 cases of Clinical AN are fatal (1-5)
  • A BMI of below 18.5 is an indicator 15 is
    clinical

4
When does it change from Diet into an
autonomous Disorder (DSM IVr)?
  • When the BMI (Body Mass Index) is equal to or
    less than 15 (below 85 expected weight by height
    and frame)
  • However the BMI is only an indicator, it must
    be accompanied by a distorted body image, an
    abnormal relationship with food, a morbid fear of
    gaining weight, cessation of periods (3 months)
    and denial that there is a problem

What does autonomous mean?
5
Why is 15 such an important figure
  • At puberty a 15 increase in body fat (puppy
    fat) is required to trigger the release of
    hormones necessary for the development of
    secondary sexual characteristics.
  • (Wider hips, breasts, periods, etc)

6
What happens if you drop below 15 BMI
  • Secondary sexual characteristic hormones are no
    longer produced and the body returns to pre-pubic
    child-like asexuality.
  • (Narrowing of hips, cessation of periods,
    breasts shrink, testicular atrophy (males) and
    these can be permanent!)
  • NB. The physiological effects of hormones are
    temporary and to maintain effect continuous
    production is necessary

7
Combined Causal Factors of AN (AO2)
  • The aetiology (cause progression) of AN is
    probably not singular, but more likely a
    combination of factors including
  • Biological
  • Psychological
  • Familial
  • Socio-cultural
  • Eg. The diathesis model
  • Genetic Predisposition Environmental Trigger
    Disorder

8
AN Biological explanations
  • (1) Genetic origin Familial studies have shown
    that first-degree relatives of AN have an
    increased risk (MZ 56 concordance) of
    developing an eating disorder (Holland et al.
    1988)
  • At age 11 there were no genetic influences on
    disordered eating. However, by age 17 the
    heritability of disordered eating was more than
    50 percent. The recent findings implicate puberty
    in the dramatic increase in genetic effects
    across time. (Meta-analysis from the Minnesota
    Twin Study Klump 2007)
  • So AO3 evaluate MZ Twin Studies and what does
    it suggest (eating behaviour is partially 50
    inherited/biologically determined).

9
(2) Set Weight or Point Theory The set-point
theory argues that an individual's metabolism
(metabolic hormones and fat cell enzymes.) will
adjust homeostatically to maintain a weight at
which the body is comfortable AO2 So our
weight/appearance is biologically controlled ?
inherited/biologically determined
10
  • (3) Hypothalamic dysfunction
  • An on and off command for eating
  • The lateral zone function as hunger centre
  • The ventromedial zone operates as satiety
    centre
  • AO2 So our eating (hungergt full-up) is
    biologically controlled ? inherited/biologically
    determined

11
Biological explanations cont...
  • (4) Imbalance of serotonin neurotransmitters
  • Increased Serotonergic activity Acts to suppress
    appetite
  • There is considerable evidence that increased
    serotonin activity in the brain is associated
    with appetite suppression. In fact, drugs which
    act on serotonin pathways in the brain are
    commonly used for the short-term management of
    obesity
  • (AO2) Therefore, inherited naturally high
    Serotonergic sensitivity would suppress eating
    and be a potential causal factor in the
    development and maintenance of AN

Link Increase in serotonin makes you happy and
suppresses hunger
12
Biological explanations summary... (AO2)
  • If anorexia can be shown to be genetic, then
    it must be inherited and we have little choice
    (Genetic Determinism). This then raises the
    question, what physical abnormality is passed on?
  • Hypothalamic abnormality?
  • Serotonergic abnormality?
  • But dont forget to be critical of twin studies
  • Studies are MZ and not MZa (reared apart),
    therefore they would share identical family
    environments (eg. shared learning from an
    anorectic mother?)
  • This coupled with small sample size brings
    the strength of evidence for genetic
    predisposition into question.
  • There must be other explanations (eg.
    psychological or triggers)

13
Some AO3 points to consider
  1. In all research, the researcher is trying to
    operationalise (IVs) a factor and claim it is
    the cause (DV) is this ok?
  2. Most research was performed in the Industrialised
    West. Is this ok?
  3. Are twin studies reliable?
  4. Are biological explanations the only ones
    available (isolationism)?
  5. Does the question of reductionism/determinism
    arise?

14
Psychological explanations of anorexia nervosa
  • Research into cause

15
Psychological explanations of AN
  • Behaviourist Explanations (AN as a Learned
    behaviour)
  • Classical conditioning (Learning by
    association)
  • Eating can be associated with anxiety since it
    can make people overweight
  • Losing weight ensures that the individual
  • reduces these feelings of anxiety ? Feel fat/ugly
    so diet and associate happiness with weight loss
    and unhappiness with weight gain

Psychological explanations of AN
16
  • Operant conditioning (Learning as a consequence
    of action)
  • The individual avoids food to gain a reward such
    as feeling positive about themselves
  • In early stages individuals can be admired or
    congratulated for losing weight and looking slim
    and healthy (positive regard)
  • Gain reward or satisfaction as a consequence of
    their actions (control of their food intake)
  • ? they associate their ACTION with happiness and
    failure (eating something) with unhappiness

17
  • SLT People imitate people they admire
    (Media/Peers etc) vicarious reinforcement
    (later reward for gaining the look)

They adjust behaviour to achieve the looks of
others and gain the rewards
18
Consider some simple questions ..
  • Q) Why do you like to look good when you go out?
  • Q) How do you know if you look good?
  • Q) What influences you when you go clothes
    shopping?
  • Q) Why are females more anxious about visiting a
    hair dresser than a dentist?

Jeggins
19
Psychological explanations of AN (AO2)
  • Behaviourist Explanations studies...
  • A review of 25 studies showed that a slender
    beauty ideal causes body dissatisfaction and
    contributes to E.D. (Groesz et al, 2001)
  • The effect was most marked in girls under 19
  • The slim ideal becomes equated with success and
    health whereas average weight or overweight
    becomes synonymous with failure, and this slowly
    becomes the dominant belief in society (Harrison,
    2001)

Psychological explanations of AN
19
20
Psychological explanations of AN
  • Behaviourist Explanations studies...
  • Women feel undue pressure on their appearance and
    reported that 27 of girls felt that media
    pressure them to strive to have a perfect body
    (Forehand, 2001)
  • Increase in eating disorders in Fiji with the
    introduction of American television programmes,
    which emphasise a westernised idealised body
    shape (Fearn, 1999)

20
21
Psychodynamic explanations (Freud)
  • Adolescents dont want to grow up and separate
    from their parents
  • They become fixated at the oral stage1 when
    they were completely dependent on their parents
  • Anorectics lose weight, lose secondary sexual
    characteristics, become childlike again (asexual)
    and return to the safety of being a little girl
    again (AO2 Gender Bias)
  • In Freudian terms, eating and sex are
    symbolically related2
  • A refusal to eat (the only control, they feel
    they have) represents a refusal of sexuality
  1. Stages of Psychosexual development
  2. Eating as manifest representation of sex
    (ego-defence)

21
22
Cognitive Williamson et al (1993) Distorted
Body Image
  • Two groups of participants
  • High risk AN (diagnosed)
  • Low risk AN (healthy No near relatives with AN)
  • Task Put stretched photographs of self back
    to correct size

23
Findings..
  • High Risk Group significantly over-estimated
    real size
  • Low Risk Group significantly under-estimated
    real size (flattered)
  • Conclusion
  • Anorectics cognitively misrepresent their own
    body image (Body Dismorphic false belief system
    BDD). Therefore no matter how much weight they
    lose, they still feel fat.

Anorectics can never reach their goal!
24
ED as an addiction
  • Smoking
  • Drinking
  • Dieting
  • Reversal Autonomy
  • (See article McCarthy, 2009)

So the dieting behaviour is learned, reinforced
and gains AUTONOMY and then the dieting controls
the person.
25
Some AO2/3 points to consider.
  • The Behaviourist Approach suggests that AN is a
    learned, reinforced product of involuntary
    associations (classical) or as a consequence of
    actions (operant) be critical (/-) of this
    approach.
  • SLT suggests we learn vicariously and model our
    behaviour on the consequences of observation be
    critical (/-) of this approach.
  • Psychodynamics (based on Freudian principles)
    suggests a dysfunction during the oral stage of
    psychosexual development be critical (/-) of
    this approach.
  • In all cases consider the approach, research
    methods (inc culture), isolationism and the
    reductionism/determinism debate.

NB The cognitive example supports one factor.
Addiction is an alternative way of looking at
EDs and 1 2 above are nowadays combined as
Social Cognitive Theory.
26
Assignment...
  • Outline symptoms of AN, then describe/evaluate
    two psychological and two biological
    explanations.
  • ( 25 marks)
  • Instructions
  • The essay style answer should have an
    Introduction (introduce/define). Then AO1 (9
    marks) and AO2/3 (16 marks) as sub-headed
    sections followed by a short conclusion
    (summary). You WILL need to focus on AO2/3 (check
    commentary and chapter summary pp 94-95
    textbook Cardwell Flanagan).
  • Ideally word processed (1 to 1.5 A4 or
    500/600 words).

Psychological explanations of AN
26
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