CHAPTER 24 SPECIAL CONCERNS OF THE PEDIATRIC PATIENT ROTATIONAL DEFORMITIES - PowerPoint PPT Presentation

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CHAPTER 24 SPECIAL CONCERNS OF THE PEDIATRIC PATIENT ROTATIONAL DEFORMITIES

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CHAPTER 24 SPECIAL CONCERNS OF THE PEDIATRIC PATIENT ROTATIONAL DEFORMITIES Intoeing pigeon toed , common. Usually spontaneously corrects. – PowerPoint PPT presentation

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Title: CHAPTER 24 SPECIAL CONCERNS OF THE PEDIATRIC PATIENT ROTATIONAL DEFORMITIES


1
CHAPTER 24SPECIAL CONCERNS OF THE PEDIATRIC
PATIENTROTATIONAL DEFORMITIES
  • Intoeing pigeon toed, common. Usually
    spontaneously corrects.
  • Metatarsus adductus (packaging defect)
    stretching and casting (associated with hip
    dysplasia.
  • Talipes Equinovarus.

2
CHAPTER 24SPECIAL CONCERNS OF THE PEDIATRIC
PATIENT (Contd.)
  • Club Foot
  • Metatarsus adductus
  • Equinus (foot flexion)
  • Always check hips

3
ANGULAR DEFORMITIES
  • Genu varum (bow legged)
  • Genu valgum (knock-kneed)
  • Normal Exam
  • 2-3 years old, bow legged.
  • 3 years old, knock-kneed.
  • 7 years old, slightly knock-kneed.
  • Pathologic if unilateral, painful or asymmetric.
  • Consider rickets (vitamin D), renal disease,
    dysplasias, (dwarfism)

4
ANGULAR DEFORMITIES (Contd)
  • INFANTILE BLOUNTS DISEASE Unknown etiology
  • Medial tibial physis ceases to function
    appropriately.
  • Leads to relative overgrowth laterally.
  • Genu varum.
  • Black females.
  • Large kids.
  • Early walkers lt11 months.
  • Treatment Surgery.

5
FOOT DEFORMITIES
  • CLUB FOOT
  • 1 in1,000 live births, half are bilateral.
  • 2.5x more common in males.
  • Inheritance multi-factorial.
  • Metatarsus adductus.
  • Equinus and heel varus.
  • Not packaging defect.
  • Always screen for hip dysplasia.
  • Treatment Casting for 3 months, then surgery.

6
FLAT FEET
  • Pes Planus
  • Absent arch which reappears when up on tip toes.
  • Treatment Reassure family.
  • Rigid flat foot
  • Tarsal coalition (calcaneus, talus, navicular may
    fuse abnormally). Can cause decreased motion and
    increasing pain.
  • Treatment Surgery.

7
HIP DISORDERSDEVELOPMENTAL DYSPLASIA OF THE HIP
(DDH)
  • Genetic and can arise during development.
  • 1 in 1,000 live births.
  • Female.
  • First born.
  • Breech position.
  • Family history.
  • Allis sign (abnormal skin folds).

8
HIP DISORDERSDEVELOPMENTAL DYSPLASIA OF THE HIP
(DDH) (Contd.)
  • Galeazzi sign (decreased height of affected
    knee).
  • 2 provocative tests
  • Ortolani maneuver relocates hip.
  • Barlow maneuver dislocates hip.
  • X-rays not helpful until after age 4 months.
    Pelvis/hips not ossified at birth.
  • Ultrasound better after 2 weeks of age. 5 are
    missed by ultrasound. Must repeat tests for 1
    year.
  • If untreated, leads to arthritis.

9
SLIPPED CAPITAL FEMORAL EPIPHYSIS (SCFE)
  • Displacement or slipping of part of femoral head
    through growth plate.
  • 11-13 years old for girls.
  • 13-15 years old for boys.
  • Related to hormonal disorders (chubby, short,
    hypogonadism).
  • More common in Blacks.

10
SLIPPED CAPITAL FEMORAL EPIPHYSIS (SCFE) Contd.
  • Complaint of knee or hip pain (obturator nerve
    referral pain).
  • Limp, painful internal rotation of hip.
  • Treatment Surgery (pinning).

11
LEGG-CALVE PERTHES DISEASE
  • Idiopathic necrosis of femoral head.
  • Usually 4-8 year old males, small for age,
    active.
  • Limited abduction and external rotation.
  • Disease course takes 2 years.
  • Treatment Involves maintaining femoral head in
    socket. Usually unilateral.

12
GROWTH PLATE FRACTURES
  • Unlike adults, children rarely injure ligaments
  • because the physis is weaker.
  • SALTER-HARRIS CLASSIFICATIONS
  • Type I
  • Fracture goes straight through growth plate.
  • X-rays within normal limits.
  • Type II
  • Fracture goes through physis and metaphysis.
  • Most common.
  • Good prognosis.

13
GROWTH PLATE FRACTURES (Contd.)
  • Type III
  • Fracture goes through physis and epiphysis.
  • Intraarticular.
  • Will require surgery.
  • If left untreated, leads to growth arrest.
  • TYPE IV
  • Fracture goes through epiphysis, growth plate and
    metaphysis.
  • Surgery.
  • High complication of growth arrest.

14
GROWTH PLATE FRACTURES (Contd.)
  • Type V
  • Rare injury.
  • Compression injury or crush injury to the growth
    plate.
  • Leads to growth arrest.

15
NEUROMUSCULAR DISORDERSCEREBRAL PALSY
  • Brain lesion which leads to non-progressive
  • Neurologic condition.
  • Perinatal.
  • 3.5 per 1,000 live births.
  • Classifications
  • Quadriplegic all four extremities.
  • Diplegic lower extremities.
  • Hemiplegic one side of body.
  • Spasticity high muscle tone.

16
SPINA BIFIDA
  • Describes variety of neural tube defects.
  • Severity depends on which level is affected.
  • 1 in 1,000 live births.
  • Meningocele Vertebral arches unfused.
    Meningeal sac is visible.
  • Myelomeningocele Neural elements exposed
    without sac.
  • Rachischisis Neural elements exposed without
    sac.

17
SPINA BIFIDA (Contd.)
  • Occurs in embryologic development.
  • Women must have Folate gt 400 units per day.
  • No hot baths or saunas during first trimester.
  • Diagnosis by 16 weeks gestation with ultrasound.
  • Amniocentesis confirms diagnosis (increased Alpha
    Feta protein).
  • Treatment Immediate closure of defect.

18
SPINA BIFIDA (Contd.)
  • Thoracic level causes spine and hip problems.
  • Lumbar and sacral levels cause knee and foot
    problems.
  • L4 gives quadriceps which allows ambulation.

19
SCOLIOSIS
  • Three-dimensional curvature of the spine.
  • IDIOPATHIC
  • Detected around age 10-12.
  • Only 10 severe enough to warrant surgery.
  • Forward bend test. Rib hump on clinical exam.
  • Less than 25 degrees observe.
  • 25-45 degrees brace.
  • Greater than 45 degrees surgery (fusing spine).

20
SCOLIOSIS (Contd.)
  • CONGENITAL
  • Look at heart and kidney abnormalities.
  • NEUROMUSCULAR
  • Cerebral palsy, spina bifida, muscular dystrophy,
    spinal cord injuries.

21
CHILD ABUSE
  • Non-accidental injuring of a child.
  • Mandatory reporting laws for physicians in all 50
    states.
  • 1,000 deaths per year.
  • Types of child abuse
  • Emotional.
  • Medical neglect.
  • Sexual.
  • Physical.

22
CHILD ABUSE (Contd.)
  • PHYSICAL ABUSE
  • Multiple fractures with various stages of
    healing.
  • Posterior rib fracture.
  • Bilateral acute long bone fractures.
  • Complex skull fracture.
  • Long bone fracture in non-ambulatory children
    (spiral fracture of long bones no longer
    pathopneumonic for child abuse).
  • Skeletal survey.

23
INFECTION
  • OSTEOMYELITIS Infection of bone.
  • Osteomyelitis generally spreads hematogenously.
    Dissemination of bacteria in blood stream.
  • In children, structures of blood vessels of
    metaphysical region predisposes them to
    infection.

24
INFECTION (Contd.)
  • SUBPERIOSTEAL ABSCESS
  • Staph aureus most common in all ages.
  • Streptococcus less than 4 years of age.
  • E-coli neonates.
  • Sickle cell anemia staph aureus, salmonella.
  • Pseudomonas stepping on a nail while wearing
    sneakers.
  • Labs CBC with differential, sed rate, CRP,
    blood cultures, x-rays and bone scan, aspiration.
  • Treatment Six weeks IV antibiotics, rarely
    surgery.

25
INFECTION (Contd.)
  • SEPTIC ARTHRITIS Infection in joint.
  • Bacteria invade joint synovium.
  • Usually sicker than patients with osteomyelitis.
  • Treatment Emergent surgical drainage.
  • Differential diagnosis Juvenile rheumatoid
    arthritis.

26
INFECTION (Contd.)
  • TOXIC SYNOVITIS Acute non-bacterial joint
  • Inflammation.
  • Self-limiting.
  • Normal sed rate and C-reactive protein.
  • No abnormal joint fluid.
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