Angina Pectoris - PowerPoint PPT Presentation

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Angina Pectoris

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* Type of angina Variant angina or prinzmetals angina : pain at rest with reversable ST-segment elevation, caused by coronary artery vasospasm. – PowerPoint PPT presentation

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Title: Angina Pectoris


1
Angina Pectoris
  • Prepared by
  • Ansam Sharef
  • Ahmad Aswad

2
Angina Pectoris
  • Definition
  • Angina pectoris is a clinical syndrome usually
    characterized by episodes of pain or pressure in
    the anterior chest . The cause is usually
    insufficient coronary blood flow which results in
    a decreased oxygen supply to meet an increased
    myocardial demand for oxygen in response to
    physical exertion or emotional stress.

3
Pathophysiology
  • Myocardial ischemia can result from
  • A reduction of coronary blood flow caused by
    fixed \or dynamic epicardial artery stenosis.
  • Abnormal constriction or deficient relaxation of
    coronary artery.
  • Reduce O2-carrying capacity of the blood .

4
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5
Factors are associated with typical angina pain
  • Physical exertion (increase myocardial o2
    demand).
  • Exposure to cold ( cause elevation of BP increase
    oxygen demand )
  • Eating heavy meals
  • Stress or any emotion-provoking situation

6
Type of angina
  • Stable angina predictable consistent pain that
    occurs on exertion and relieved by rest.
  • Unstable angina or preinfarction or cresendo
    angina symptoms occur more frequently and longer
    than stable angina.
  • Intractable or refractory angina sever
    incapacitating chest pain.

7
Type of angina
  • Variant angina or prinzmetals angina pain at
    rest with reversable ST-segment elevation, caused
    by coronary artery vasospasm.
  • Silent ischemia objective evidence of ischemia
    (as ECG changes with a test), but pt report no
    symptom.

8
Risk Factors for atherosclerosis
  1. Family history of premature coronary artery
    disease.
  2. DM, systemic HTN.
  3. Cigarette smoking.
  4. Hypercholesterolemia.
  5. Others as obesity, increase levels of
    lipoprotein,fibrinogen, s.triglycerides.

9
Clinical manifestations
  1. May produce pain vary in severity from feeling
    of indigestion to chocking in retrosternal area ,
    radiate to neck , jaw shoulders , inner aspects
    of upper arms
  2. Feeling of weakness or numbness in the arms ,
    wrists and the hands
  3. Shortness of breath

10
Clinical manifestations
  1. Pallor, Diaphoresis
  2. Dizziness or lightheadedness
  3. Nausea and vomiting

11
Diagnostic findings
  • Often made by evaluating the clinical
    manifestations of ischemia and the pts history
  • 12-Lead ECG and blood laboratory
    values help in making diagnosis
  • C-reactive protein ( CRP) is a marker for
    inflammation of vascular endothelium which caused
    by CAD

12
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13
Diagnostic findings
  1. An elevated blood level of homocysteine ,an amino
    acid proposed as an independent risk
    factor for cardiovascular disease , but no
    studies supported the relationship between
    elevation of homocysteine and
    atherosclarosis

14
Medical management for the pt with angina pectoris
15
  • The objective is to decrease oxygen demand of
    myocardium and to increase oxygen supply
  • 1.Nitroglycerin administered to reduce
    myocardial oxygen consumption

16
  • 2.Beta-adrenergic blocking agents
  • ( propranolol ,metoprolol , atenolol) appear
    to reduce myocardial oxygen consumption, result
    in a reduction in heart rate, decreased BP , and
    reduced myocardial contractility .

17
  • 3-Calcium channel blocking agents some decrease
    sinoatrial node automaticity and node conduction
    ,resulting in slower heart rate

18
4- Antiplatelet and anticoagulent medications
  • Aspirin prevents platelet activation and
    reduces the incidence of MI and death with CAD
  • Heparin prevents the formation of new blood
    clots

19
  • Oxygen administration
  • usually initiated at the onset of chest
    pain to increase the amount of oxygen delivered
    to the myocardium and to decrease the pain.

20
Prevention
  • Self care action plan changing habits.
  • Stop smoking
  • Increase level of exercise
  • Cut down on fatty foods
  • Eat more oats, which decrease cholesterol

21
  • Lose wt if u DR. thinks you are overweight.
  • Make sure your BP is not high by regular check
  • Consider another method of contraceptive if you
    take pill

22
Nsg process for pt with Angina pectoris
  • Assessment
  • The nurse gather information about the pts
    symptoms and activities . The nurse may ask about
    the period that angina last , and if any
    medication relieve the angina.

23
Diagnosis
  • Ineffective myocardial tissue
  • perfusion secondary to CAD as
  • evidence by chest pain or equivalent
  • symptoms
  • Anxiety related to fear of death

24
Diagnosis
  • Noncompliance , ineffective management of
    therapeutic regimen related to failure to
    accept necessary lifestyle changes

25
Planning and goals
  • The major goal include immediate treatment when
    angina occur , preventing of angina , reduction
    of anxiety and absence of complications

26
NSG interventions
  • Treating angina when pt experiences angina the
    nurse should direct pt to stop activities and sit
    or rest in bed in semi-fowler position
  • Reducing anxiety The nurse should explore and
    implicate that the diagnosis has for the pt
    providing information about the illness ,
    treatment and methods of preventing its
    progression

27
  • Preventing pain when the pt has pain with
    minimal activity , the nurse alternates the pts
    activities with rest periods

28
Evaluation
  • expected pts outcomes may include
  • Reports that pain is relieved promptly
  • Reports decreased anxiety

29
  • Understands ways to avoid complications and
    demonstrates freedom from complications
  • Adheres to self-care program

30
THANK YOU
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