Alterations in Urinary Elimination - PowerPoint PPT Presentation

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Alterations in Urinary Elimination

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Title: Alterations in Urinary Elimination


1
Alterations in Urinary Elimination
  • Karen Ruffin RN, MSN Ed.

2
Urinary Elimination
3
Categorize the Issue
  • Pre-renal
  • Intra-renal
  • Post-renal
  • Failure

4
Pre-Renal
  • Chronic HTN
  • Left sided heart failure
  • Infection
  • glomerulonephritis

5
Chronic HTN
  • Overview
  • Because of the large number of small diameter
    blood vessels associated with the kidney, kidney
    function is extremely sensitive to high blood
    pressure. Elevated pressures can cause blood
    vessel damage as plasma leaks into the artery
    wall under pressure. This plasma infiltration
    begins an inflammatory response that thickens the
    artery walls with resultant renal ischemia. Renal
    ischemia, in turn, can further damage the kidney
    through the renin-angiotensin response and its
    resultant exacerbation of hypertension.

6
Pathophysiology and Etiology
7
Risk Factors
8
Clinical Manifestation
9
Collaboration
10
Diagnostic test
11
Pharmacologic Therapies
12
Clinical Theripies
13
Nursing Process
  • Assessment
  • Health History
  • Physical Examination
  • Nursing Diagnosis
  • Plan
  • Implantation
  • Evaluation

14
Left sided Heartfailure
  • Overview
  • Recall the definition of heart failure being any
    condition in which cardiac output is insufficient
    to meet body needs. One of those needs in
    systemic circulation is adequate kidney perfusion
    to keep filtration function and waste removal
    within normal limits. Admittedly, with left CHF
    blood backs up into the lungs causing respiratory
    problems. But, it is the diminished output into
    the aorta from the failing heart that lowers
    kidney perfusion downstream and can result in
    renal failure with its associated low urine
    output and accumulation of wastes in circulating
    blood and body fluids.

15
Pathophysiology and Etiology
16
Risk Factors
17
Clinical Manifestation
18
Collaboration
19
Diagnostic test
20
Pharmacologic Therapies
21
Clinical Theripies
22
Nursing Process
  • Assessment
  • Health History
  • Physical Examination
  • Nursing Diagnosis
  • Plan
  • Implantation
  • Evaluation

23
Infection
  • Overview
  • In this condition, antibody complexes resulting
    from a recent infection collect on the glomerular
    membrane on the circulatory side and cause
    a,secondary glomerular inflammation. This
    glomerular inflammation can cause permanent
    nephron damage by fibrous connective tissue
    infiltration which interferes with the glomerular
    filtration process. Streptococcal infections are
    notorious as causative agents of acute
    glomerulonephritis. Consequently, something as
    simple as a strep throat can have serious
    consequences.

24
Pathophysiology and Etiology
25
Risk Factors
26
Clinical Manifestation
27
Collaboration
28
Diagnostic test
29
Pharmacologic Therapies
30
Clinical Theripies
31
Nursing Process
  • Assessment
  • Health History
  • Physical Examination
  • Nursing Diagnosis
  • Plan
  • Implantation
  • Evaluation

32
Intra-Renal
  • Posions
  • Toxins
  • Infections
  • Tumors/Stones
  • Acute Tubular Necrosis

33
Overview
  • With intrarenal disorders, the problem arises
    from within the nephron tubules themselves, at
    some point from the proximal tubule to the
    collecting duct. It is difficult to imagine
    damage so specific as to damage the inner nephron
    without affecting either end unless attention is
    given to bacterial toxins and accidentally
    ingested materials, such as pesticides,
    herbicides, heavy metals, antifreeze, certain
    cleaning fluids, and nephrotoxic drugs.

34
Poison, Toxins, ATN
  • Overview
  • With intrarenal disorders, the problem arises
    from within the nephron tubules themselves, at
    some point from the proximal tubule to the
    collecting duct. It is difficult to imagine
    damage so specific as to damage the inner nephron
    without affecting either end unless attention is
    given to bacterial toxins and accidentally
    ingested materials, such as pesticides,
    herbicides, heavy metals, antifreeze, certain
    cleaning fluids, and nephrotoxic drugs.

35
Pathophysiology and Etiology
36
Risk Factors
37
Clinical Manifestation
38
Collaboration
39
Diagnostic test
40
Pharmacologic Therapies
41
Clinical Theripies
42
Nursing Process
  • Assessment
  • Health History
  • Physical Examination
  • Nursing Diagnosis
  • Plan
  • Implantation
  • Evaluation

43
Post Renal
  • Renal Calyx (Kidney Stones)
  • Infection
  • Postrenal disorders are those that originate in
    the urinary tract downstream from the nephrons
    of the kidney. These disorders can involve the
    renal pelvis, the ureters or the bladder and
    urethra.

44
Overview
  • Renal Calyx (Kidney Stone). Kidney stones result
    from crystalline materials that occur in urine in
    concentrations sufficient to cause aggregate
    crystals that grow into stones within the renal
    pelvis. Once formed, these stones can move into
    the ureters and lodge causing intense pain until
    they are passed naturally or are removed
    surgically or disrupted by ultrasound treatments.
    A common kidney stone develops from calcium
    oxylate salts in people with high calcium and
    oxalic acid in their diets. Such stones are
    prevalent in people in the South. In fact,
    southerners have triple the incidence of other
    regions.

45
Overview
  • Calcium comes primarily from dairy products and
    leafy green vegetables, both of which are common
    in southern diets. Oxylates come from plant
    extracts (coffee, tea, and cola), which are also
    common in southern diets. Considering both of
    these factors, when combined with dehydration as
    is common in southern climates, it is not
    surprising that the rate among Southerners is so
    high.

46
Oveview
  • Pyelonephritis. Pyelonephritis is a condition
    which develops when infectious microorganisms
    establish in the urinary tract and migrate upward
    into kidney tissue. The incidence is particularly
    high in individuals who contaminate the urethra
    with fecal material containing E. coli as a
    result of poor hygiene or are unable to
    completely void the bladder for some reason. The
    urinary retention leads to excess microbial
    growth and eventual spread into the kidneys.

47
Pathophysiology and Etiology
48
Risk Factors
49
Clinical Manifestation
50
Collaboration
51
Diagnostic test
52
Pharmacologic Therapies
53
Clinical Theripies
54
Nursing Process
  • Assessment
  • Health History
  • Physical Examination
  • Nursing Diagnosis
  • Plan
  • Implantation
  • Evaluation

55
Renal Failure
  • No treatise of renal disease would be complete
    without a discussion of renal failure. Renal
    failures can be acute, lasting only for a short
    time, or chronic, developing over and continuing
    for an extended period of time. Chronic renal
    failures result when over 70 of nephrons are
    permanently lost and require dialysis to sustain
    life. From the point at which dialysis or
    transplantation is required to sustain life, the
    condition is termed end stage renal failure.

56
Describe the Differences
  • Acute
  • Chronic

57
Pathophysiology and Etiology
58
Risk Factors
59
Clinical Manifestation
60
Collaboration
61
Diagnostic test
62
Pharmacologic Therapies
63
Clinical Theripies
64
Nursing Process
  • Assessment
  • Health History
  • Physical Examination
  • Nursing Diagnosis
  • Plan
  • Implantation
  • Evaluation
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