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Cardiovascular Physical Exam Pearls

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S2-opening snap (mitral stenosis) S2-pericardial knock (constrictive pericarditis) ... Aortic Stenosis. Three major causes. Congenital, Rheumatic, and ... – PowerPoint PPT presentation

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Title: Cardiovascular Physical Exam Pearls


1
Cardiovascular Physical Exam Pearls
  • Daniel J. ORourke, MD
  • Cardiology Symposium
  • December 2004

2
Valvular Abnormality
  • Cardiovascular Exam
  • JVP pressure pulsations
  • Carotid Pulse rate, rhythm, rate of rise,
    volume, compliance
  • Inspection
  • Palpation LV apical impulse, PMI
  • Heart Sounds intensity 2 components to S1 and
    S2. Gallops
  • Murmur

3
DDX of a 2 Component S1
  • S4-S1 (noncompliant LV)
  • S1-Ejection sound (valvular or aortic root
    dilatation)
  • S1-Click (MVP)
  • M1-T1 splitting (normal or pathologic - RBBB)

4
DDX of a 2 Component S2
  • Splitting of S2 (normal, narrow, wide,
    paradoxical)
  • S2-S3 (physiologic vs. pathologic normal LV,
    dilated LV)
  • S2-opening snap (mitral stenosis)
  • S2-pericardial knock (constrictive pericarditis)
  • S2-tumor plop (atrial myxoma)

5
Overview of Murmurs
  • Mid Systolic (SEM)
  • 1. Physiologic or flow murmur
  • 2. Aortic stenosis
  • 3. HOCM
  • Late Systolic
  • 1. Papillary muscle dysfxn
  • Holosystolic
  • 1. MR
  • 2. TR
  • 3. VSD
  • Early Diastolic
  • 1. Aortic regurgitation
  • 2. Pulmonic regurgitation
  • Mid Late Diastolic
  • 1. Mitral stenosis
  • 2. Tricuspid stenosis

6
Evaluation of Murmurs
  • Timing - Systolic vs. Diastolic
  • Intensity - Grade I-VI
  • Characteristics
  • Quality (blowing, harsh, musical)
  • Pitch (high or low)
  • Duration
  • Shape of murmur
  • Location
  • Radiation

7
Clinical ScenarioIn-hospital patient develops a
new murmur
  • Clinical question Endocarditis?
  • Murmurs associated with left-sided endocarditis
    AR, MR
  • Tachycardia
  • Soft S1 severe AR or moderate to severe MR
  • Pulmonary edema
  • Diastolic murmur (AR), systolic murmur (MR)
  • Classic findings of chronic AR are rarely present
  • Features of a physiologic or flow murmur

8
Physiologic Murmur(Flow, Functional)
  • Common in hyperkinetic states
  • Caused by rapid ejection of blood
  • Starts shortly after S1 and peaks by mid systole
  • Intensity related to velocity of blood flow -
    Never gt Grade III
  • Absence of concomitant cardiac pathology

9
Pathologic vs. Nonpathologic Murmurs
  • Always pathologic if
  • Diastolic murmur
  • Holosystolic or late systolic murmur
  • Continuous murmur
  • Grade 4-6 murmurs
  • Concomitant cardiac symptoms or exam findings

10
Clinical ScenarioKnown AS when do I need to
order a follow-up echo?
  • History angina, syncope (lightheadedness),
    heart failure (dyspnea)
  • Physical exam findings
  • Mean annual progression
  • AVA decreases by 0.1 cm2
  • Mean gradient increases by 10 mmHg
  • AVA Mean Gradient
  • Mild gt1.5 cm2 lt25 mmHg
  • Moderate 1.0-1.4 25-49 mmHg
  • Severe lt1.0 gt50 mmHg

11
Aortic Stenosis
  • Three major causes
  • Congenital, Rheumatic, and Degenerative
    (calcific)
  • Classic murmur
  • Harsh, crescendo-decrescendo SEM at the base
  • Radiates to the carotids and/or apex
    (Gallavardin)
  • Clues to assessing severity
  • Carotid upstroke
  • Duration of the murmur
  • Splitting of S2

12
Assessing AS Severity by Exam
  • Splitting of S2
  • Physiologic --gt Mild
  • Single --gt Mod/Severe
  • Paradoxical --gt Severe
  • Murmur Peak
  • Early-mid --gt Mild
  • Mid-late --gt Moderate
  • Late --gt Severe

Sustained LV apical impulse, S4 gallop Moderate
to severe
Delayed, diminished carotid upstroke - Severe
13
Obtaining an Echocardiogram Aortic Stenosis
  • Indications Class
  • Diagnosis and severity of AS I
  • Assessment of LV size, function, and/or
    hemodynamics I
  • Reevaluation of patients with known AS with
    I
  • changing symptoms or signs
  • Reassessment of asymptomatic patients with severe
    AS I

JACC 1998321486-1588.
14
Obtaining an Echocardiogram Aortic Stenosis
  • Indications Class
  • Reassessment of asymptomatic patients with
    IIa
  • mild to moderate AS and LV dysfunction or
  • hypertrophy
  • Reassessment of asymptomatic patients with stable
    III
  • exam findings and normal LV size and function

JACC 1998321486-1588.
15
Frequency of Surveillance Echo
  • Asymptomatic, clinically stable patients
  • Mild AS Every 5 years
  • Moderate AS Every 2 years
  • Severe AS Annually

16
Chronic Mitral Regurgitation
  • Pathophysiologic mechanisms
  • Mitral valve leaflets
  • Papillary muscles
  • Chordae tendinae
  • Annulus
  • Classic murmur
  • High pitched, blowing, holosystolic murmur at the
    apex
  • Radiates to the axilla or left sternal border

17
Assessing MR Severity by Exam
  • Mild MR
  • Normal carotid upstroke
  • Normal LV apical impulse
  • Heart Sounds
  • Normal intensity of S1
  • Grade 1-2 murmur
  • Moderate-Severe MR
  • Carotid pulse - Brisk upstroke
  • Hyperdynamic, displaced LV apical impulse
  • Heart Sounds
  • Soft S1
  • S2 widely split
  • S3 gallop

18
Obtaining a TTE Chronic Mitral Regurgitation
  • Indications Class
  • Baseline evaluation to quantify severity of MR
    I
  • and assess LV function
  • Determine the mechanism of MR
    I
  • Annual or semi-annual surveillance of LV function
    I
  • in asymptomatic severe MR
  • To establish cardiac status after a change in sxs
    I
  • Evaluation post MVR to establish baseline status
    I
  • Routine f/u evaluation of mild MR with normal
    III
  • LV size and function

JACC 1998321486-1588.
19
Summary
  • The cardiovascular exam remains the most widely
    used method to screen for heart disease.
  • Echocardiography is an important noninvasive
    method for assessing the significance of cardiac
    murmurs and to define cardiac structure and
    function.
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