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Prepared by Helen Cooke

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Describe the main causes of antepartum haemorrhage. Discuss the management ... Painless vaginal bleeding after 20 weeks gestation. Low lying placnta of praevia ... – PowerPoint PPT presentation

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Title: Prepared by Helen Cooke


1
Antepartum Haemorrhage (APH)
Prepared by Helen Cooke August 2008
2
Objectives
  • Describe the main causes of antepartum
    haemorrhage
  • Discuss the management of main causes

3
The Placenta
4
Causes of APH
  • Placental praevia
  • Placenta abruption
  • Vasa Praevia
  • Incidental and indeterminate causes 50-60 APH
  • Non Placental causes - local genital tract trauma

5
Low Lying Placenta
  • Not an uncommon finding on second trimester
    ultrasound scan.
  • 15-20 of pregnancies have a low lying placenta.
  • Only 5 of these remain low lying at 32 weeks and
  • One third of those are low lying at term (37
    weeks).

6
Placenta Praevia
  • Placenta that has implanted in part or all of the
    lower uterine segment encroaching upon or
    covering the internal cervical os.
  • Responsible for 15-20 of APHs
  • Haemorrhage is likely in the third trimester as
    the lower segment grows and thins or the cervix
    dilates.
  • With the increase in LSCS the clinician should
    consider placenta accreta, increta percreta
    with placenta praevia

7
Grading
  • Grade 1 ( 1st Degree)
  • Part of placenta lies in the lower segment but
    does not reach os
  • Grade 2 ( 2nd Degree)
  • The lower margin of the placenta reaches the
    internal os but does not cover it
  • Grade 3 ( 3rd Degree)
  • The placenta covers the os
  • Grade 4 ( 4th Degree)
  • The placenta lies centrally over the os

8
Maggie Myles Textbook for Midwives
9
Clinical Features
  • Bleeding without abdo pain or uterine tenderness,
    usually bright red
  • Usually between 34-38 wks (20 before 28 weeks)
  • May be associated with contractions
  • Bleeding usually recurs often increasing in
    severity with increasing gestational age
  • Not usually precipitated by any one factor,
    coitus, etc.
  • 30 women with placenta praevia will not have a
    APH

10
Clinical management
  • Active vs expectant management
  • Active management
  • if bleeding continues
  • non-reassuring FHR pattern
  • maternal compromise

11
Vasa Praevia
  • Rare event
  • Umbilical cord vessels are covered only by
    chorion and amnion (membranes)
  • Vessels are exposed and can rupture under
    pressure or ARM
  • Baby at risk of severe bleeding and death
  • May feel like a cord pulsating on VE
  • May be diagnosed on colour Doppler U/S

12
Risk factors vasa praevia
  • Painless vaginal bleeding after 20 weeks
    gestation
  • Low lying placnta of praevia
  • Succenturiate lobe or velementous cord insertion
  • IVF or multiple pregnancy

13
Vasa Praevia
Exposed vessels
14
Placental Abruption
  • Separation of a normally implanted placenta
    usually by haemorrhage into the decidual basalis
    after the 20th week of pregnancy and before birth
    of the baby
  • The amount of bleeding depends on
  • the size of the bleeding vessel/s
  • the amount of placental separation
  • The more extensive the bleeding, the more likely
    it is to strip the membranes from the uterine
    wall and pass through the cervix and vagina

15
Causes of abruption
  • Unknown cause is the most common
  • Hypertensive disorders
  • Previous APH
  • Abdominal trauma MVA, DV, fall
  • Associations have been made with abnormal
    trophoblastic invasion and/or vessel formation
  • Other predisposing factors - Rapid reduction in
    uterine size, ECV, Cocaine use, smoking, poor
    nutrition, advancing parity, multiple pregnancy,
    IOL

16
Incidence
  • 1 to 1.5 of pregnancies
  • Recurs in 10-15 of cases
  • In 5 of these women DIC occurs

17
Bleeding may be
  • Revealed
  • Concealed
  • Partially revealed
  • Painful and the womens clinical symptoms may
    not always match amount of blood loss

18
Clinical Presentation
  • Vaginal bleeding of varying amount (80)
  • Uterine tenderness /- (70)
  • Abnormal FHR pattern /- (60)
  • Uterine contractions /- (high frequency, low
    intensity) (35)
  • Uterine Hypertonus
  • Clinical presentation features are dependant on
    degree of abruption and blood loss.

19
Prevention of abruption
  • Actively treat maternal hypertension
  • Screen for domestic violence
  • Screening brief intervention for smoking and
    substance abuse
  • Seat belt worn under pregnant abdomen

20
Complications of abruption
  • Maternal
  • Haemorrhagic shock
  • Coagulopathy/DIC
  • Uterine rupture
  • Renal failure
  • Maternal death
  • Fetal
  • Fetal Hypoxia
  • Anaemia
  • Growth restriction
  • CNS damage
  • Fetal death

21
Management Considerations - APH
  • Maternal welfare assessment monitoring of vital
    signs, blood loss, urine output. Always think
    about a concealed haemorrhage
  • Insert two large bore cannulars 14 or 16g
  • Fluid replacement
  • Cross match 4 units of packed cells
  • Resuscitation and/or delivery
  • In the presence of significant blood loss -
    oxygen

22
Management Considerations cont
  • Fetal welfare assessment
  • electronic FHR monitoring
  • U/S for placental position/ vasa praevia
  • Steroid cover if preterm
  • Anti D if Rh -ve
  • Make a diagnosis Clinical - Ultrasound (?
    Value)
  • Maternal education and support
  • Maternal biochem, haematology FBC/Kleihaur if
    Rh -ve
  • /- Preparation for Preterm birth transfer if
    required
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