Title: Disorders of the Digestive System
1Disorders of the Digestive System
2Disorders of the Digestive System
- Sources of digestive problems
- Mechanical
- Nervous
- Chemical
- Hormonal
3Eating Disorders
- Anorexia
- Chronic loss of
- appetite
- Possible emotional,
- social factors
- Anorexia Nervosa
- Psychological disorder
- Bulimia
- Binge-purge syndrome
4Causes of Anorexia
- Anxiety, depression
- Improper fit of dentures
- Illness, physical discomfort
- Constipation
- Intestinal obstruction
5Anorexia, Nursing Implementation
- Become familiar with patients eating habits
- Permit patient to choose own food
- Dont force patient to eat
- Provide pleasant environment
- Serve small portions
6Dental Plaque and Caries
- Erosive process that dissolves tooth enamel
- Medical management
- Removal of affected area and
- replace with dental material
- Dental check ups
- Fluoridated water
- Nursing interventions
- Teach patient oral care
- Diet changes
7Gingivititis
- Inflammation of the gums
- Symptoms
- Bleeding, swollen, tender gums
- Difficulty chewing
- Causes
- Accumulation of food between teeth
- Vitamin deficiency
- Anemia
- Leukemia
- Prevention
- Brushing teeth gums
- Daily flossing
- Adequate diet
8Periodontitis
- Untreated Periodontitis
- Teeth loosen
- Spreads to mandible
- Prevention
- Impeccable tooth gum
- Regular flossing
- Adequate diet
- Treatment
- Drainage of abscess
- Antibiotics
- Extraction
9Recurrent Aphthous Stomatitis
- Multi system disorder
- Painful ulcers
- Mouth
- Genitals
- Uveal tract of the eye
- Causes
- Viruses, bacteria, fungus
- Chemotherapy
- Vitamin deficiency
- Four Forms
- Minor Type
- Canker sores
- Lesions are 2- 4 mm in diameter
- Usually fewer than 5 in number
10Recurrent Aphthous Stomatitis
- Major Aphthous Stomatitis
- Referred to as Suttons disease
- 10 mm or greater in diameter
- They frequently occur in the
- posterior portion of the mouth
- Take four to six weeks to heal
- These lesions may result in scarring
- Herpetiform
- Confused with herpes
- Numerous, 1-2 mm ulcers, cropped
- together
- When the neutrophil count returns
- to normal the lesions resolve
11Recurrent Aphthous Stomatitis
- Fourth form is known as Behçet's disease
- Complex multi-system disorder including
- Synovitis
- Uveitis
- Vasculitis
- Meningoencephalitis
- Oral and/or genital mucosa are the first
manifestation - It is a rare, non-infectious disease
- Very uncommon in North America and Europe
- Half of the patients have evidence of immune
dysfunction
12Recurrent Aphthous Stomatitis
- Immune Dysfunction seen with Behçet's disease
- 5-20 of Crohn's patients develop disease
- Ulcerative colitis
- Malabsorption syndromes
- Gluten-sensitive enteropathy
- HIV infection
- The lesions appear to be more severe, more
painful and deeper - Cyclic neutropenia
13Behcets
14Herpes Simplex Virus
- Acute viral infection
- Two strains of herpes simplex virus
- HSV-1 Common cold sore
- Forms clusters of fluid filled blisters
- HSV-2
- Sexually transmitted
- Can develop in and around the mouth or genitals
- Shingles
- Form along nerve path
15Candidiasis
- Etiology/Pathophysiology
- Fungus normally present in the mouth, intestine,
vagina, and on the skin - Also referred to as thrush and moniliasis
- Clinical manifestations/assessment
- Small pearly-white patches on the mucous membrane
of the mouth - Thick white discharge from the vagina
16Candidiasis
- Causes
- Cancer
- Diabetes
- Alcoholism
- Treatment
- Nystatin
- Gentician Violet
- OTC medications
- Nursing care
- Good hand washing
- Bland or soft food for patient
- Oral care
- Education on medication administration
17Mumps
- Contagious viral infection of parotid salivary
glands - Possible complications
- Inflammation of testicles
- Sterility in males
- MMR vaccine
18Achalasia
- Etiology/Pathophysiology
- LES cannot relax (cardiospam)
- Causes decreased motility of the lower portion of
the esophagus - Absence of peristalsis and dilation of the lower
portion of the esophagus - Decreased amount or no food can enter the stomach
- The esophagus can hold as much as a liter
- Possible causes
- Nerve degeneration, esophageal dilation, and
hypertrophy - Disruption of the normal neuromuscular activity
of the esophagus
19Achalasia
- Clinical manifestations/Assessment
- Dysphagia-PRIMARY SYMPTOM
- Regurgitation of food
- Substernal chest pain
- Loss of weight
- Weakness
- Poor skin turgor
20Achalasia
- Diagnostic tests
- Radiologic studies- Esophagoscopy
- Medical management
- Medications anticholinergics, nitrates, and
calcium channel blockers - Dilation of cardiac sphincter
- Surgery-Cardiomyectomy
21Gastroesophageal Reflux Disease (GERD)
- Etiology/Pathophysiology
- Backward flow of gastric acid into the esophagus
- Reduced LES pressure
- Clinical manifestations
- Pain may mimic angina
- Heartburn (pyrosis) 20 min 2 hrs after eating
- Regurgitation
- Dysphagia or odynophagia (painful swallowing)
- Eructation (belching)
- Cough
- Wheezing
- Hoarseness
22Gastroesophageal Reflux Disease (GERD)
- Risk factors
- Alcohol, tobacco, smoking
- Delayed gastric motility
- Diagnostic tests to differentiate GERD from
angina - Esophageal motility
- Bernstein tests evaluate LES function
- pH monitoring for 24 hours to record reflux
episodes - Barium swallow endoscopy to evaluate for
- hiatal hernia
-
23Gastroesophageal Reflux Disease (GERD)
- Medical management Nursing interventions
- Antacids or acid-blocking medications
- Diet
- 4-6 small meals/day
- Low fat, adequate protein
- Remain upright for 1-2 hours after eating
- Lifestyle
- Eliminate smoking
- Avoid constrictive clothing
- HOB up at least 6-8 inches for sleep
24Barretts Esophagus
- Etiology/Pathophysiology
- Cells in esophagus change to cells normally found
in the intestine (metaplasia) - Patients feel less discomfort as metaplastic
cells are less sensitive - Cant cure, treat GERD
- Medications
- Antacids
- GI stimulants
- Histamine H2 antagonists
- Proton pump inhibitors
- Surgery
- Fundoplication
25Acute Gastritis
- Etiology/Pathophysiology
- Inflammation of the lining of the stomach
- Associated with
- Alcoholism smoking
- Bacteria viruses
- Chemical toxins
- Stressful physical problems
- Clinical manifestations
- Fever
- Headache
- Epigastric pain
- Nausea and vomiting
- Coating of the tongue
- Anorexia
26Acute Gastritis
- Long term
- Changes in the mucosal lining that interferes
with acid and pepsin secretion - Gastritis usually resolves once the offending
agent is removed - Diagnostic tests
- Stool for occult blood
- WBC
- Electrolytes
27Acute Gastritis
- Medical management
- Antiemetics
- Antacids
- Antibiotics
- IV fluids
- NG tube
- Nutritional Supplementation
- Administration of blood
- NPO until signs and symptoms subside
28Nausea
- Etiology/Pathophysiology
- Feeling the urge to vomit
- May occur independently or precede vomiting
- Specific neural pathways not identified
- Probably controlled by parts of brain that
control involuntary bodily functions - Signs and symptoms
- Increased salivation
- Diminished functional activities of the stomach
- Altered small intestine motilility
29Vomiting
- Etiology/Pathophysiology
- Forceful expulsion of gastric contents
- Increased intrathoracic pressure
- Increased intracranial pressure
- Controlled by
- Vomiting center (medulla)
- Initiates vomiting when stimulated
- Chemo-receptor trigger zone
- Must be stimulated by a drug or toxin
30Causes of Nausea Vomiting
- Stress, fear, and depression
- Pain
- Acute febrile illness
- Medications
- Food poisoning
- Anesthesia
- Diseases of the stomach
- Intestinal obstruction
- Pregnancy
- Head injury
31Nursing Implementation for Nausea Vomiting
- Administer antiemetics
- Monitor fluid electrolyte replacement
- Protect patient from unpleasant sight or smells
- Attempt to keep stomach empty
- Ventilate room
- Observe record the character and quantity of
emesis
32Peptic Ulcers (PUD)
- Gastric ulcers and duodenal ulcers
- Ulcerations of the mucous membrane or deeper
structures of the GI tract - Most commonly occur in the stomach and duodenum
- Result of acid and pepsin imbalances
- Chronic NSAID use
- H. pylori
- 70 of patients with gastric ulcers
- 95 of patients with duodenal ulcers
33Peptic Ulcers (PUD)
- Gastric ulcer
- In the distal half of the stomach
- Bleed more, harder to control
- Duodenal ulcers
- May or may not be caused by hypersecretion of
gastrin - Ulceration occurs when the acid secretion exceeds
the buffering factors - More likely to have chronic bleeding and
perforate
34Peptic Ulcers (PUD)
35Peptic Ulcers (PUD)
- Clinical manifestations
- Pain Dull, burning, boring, or gnawing,
epigastric - Dyspepsia
- Hematemesis
- Melena
- Diagnostic tests
- Esophagogastroduodenoscopy (EGD)
- Breath test for H. pylori
36Peptic Ulcers (PUD)
- Complications of peptic ulcers
- Bleeding
- Hematemesis
- Melena
- Gastric outlet obstruction
37Peptic Ulcers (PUD)
- Diagnosis
- Esophagogastroduodenoscopy (EGD)
- Visualize tissue and Biopsy if necessary
- Wireless capsule endoscope
- Complications
- Stool impaction
- Small bowel stricture
38Peptic Ulcers (PUD)
- Medical management/Nursing interventions
- NG tube until bleeding subsides
- Antacids
- Histamine H2 receptor blockers
- Proton pump inhibitor
- Mucosal healing agents
- Antibiotics
- Anticolingerics
- Reclining for 1 hour post meal
- IV fluid
- Diet
- High in fat and carbohydrates
- Low in protein and milk products
- Small frequent meals
- Limit coffee, tobacco, alcohol, and NSAID use
39Peptic Ulcers (PUD)
- Medical management/Nursing interventions
- Surgery
- Antrectomy-
- Gastrodudodenostomy (Billroth I)
- Gastrojejunostomy (Billroth II)
- Total gastrectomy
- Vagotomy
- Pyloroplasty
40Types of gastric resections with anastomoses. A,
Billroth I. B, Billroth II.
41Peptic Ulcers
- Complications after gastric surgery
- Dumping syndrome
- Eat 6 small meals a day
- high in protein and
- carbohydrates
- Eat slowly and avoid fluid
- with meals
- Pernicious anemia
- Iron deficiency anemia
42Diarrhea
- Rapid movement through intestines of loose,
watery stools resulting from increased
peristalsis - Causes
- Fecal impaction
- Ulcerative colitis
- Intestinal infections
- Drugs
43Diarrhea
- Nursing implications
- Meticulous skin care
- Observe stool for color, odor, consistency,
mucous, blood, or pus - Administer anticholinergics
- Reduce bowel spasticity
- Administer anti-diarrheal agents
- Mild diarrhea
- Moderate diarrhea
- Severe diarrhea (infectious agent)
44Constipation
- Etiology/Pathophysiology
- Fecal mass in rectum
- Water is absorbed and feces hardens
- Painful to pass
- Causes
- Neurological conditions
- Disease
- Medications
- Diet
- Decreased activity
- Ignoring the urge to defecate
- Chronic laxative use
45Constipation
- Clinical manifestations/Assessment
- Abdominal distention
- Indigestion
- Rectal pressure
- Hard, dry stools
- Decrease appetite
- Rectal pressure
- Intestinal rumbling
46Constipation
- Complications
- Fecal impaction
- Cardiac complications from straining
- Dilation of colon (Megacolon)
- Colonic mucosal atrophy
- Fecal incontinence
47Constipation
- Treatment
- High fiber diet
- 2-3 liters of fluid a day
- Strengthening of abdominal muscles
- Behavior changing
- Diet
- Whole grains
- Fresh fruits
- Vegetables
48Constipation
- Medications
- Bulk forming agents
- Absorb fluid and swell in the intestine and
increase peristaltic action - Laxatives
- Bulk forming, stool softeners, stimulant, and
saline - Stool softeners
- Detergent like drugs that permit easier
penetration and mixing of fats and fluids with
the fecal mass - Stimulant
- Increase the motility of GI tract by chemical
irritation of the intestinal mucosa - Golytely
- Causes a large volume of water to be retained in
the colon - Results in diarrhea within 30-60 minutes
49Constipation
- Enemas
- Instilled directly into the lower colon
- Retained in bowel
- Cleansing
- Types of enemas
- Tap water
- Saline
- Soap
- Oil
- Medicated
50Infection
- Etiology/Pathophysiology
- Invasion by pathogenic microorganisms
- Person-to-person contact
- Fecal-oral transmission
- Long-term antibiotic therapy
- Clinical manifestations
- Rectal urgency
- Tenesmus
- Nausea, vomiting diarrhea
- Abdominal cramping
- Fever
51Infection
- E-coli 0157H7
- Not part of normal human intestinal flora
- Found in 1 of food cattle
- Contaminated or under cooked meat or contaminated
water - Signs and symptoms
- Bloody diarrhea, abdominal cramping and
tenderness - Avoid-anti-diarrheals and anti-mobility
- C-Difficle
- Complication of antibiotic because it inhibits
the growth of normal intestinal flora and allows
for the overgrowth of C-Difficile - A toxin is produced that causes tissue damage in
the intestine - Stop the offending antibiotic
- Diagnosis-stool culture
- Treatment-Flagyl or Vancomycin
52Infection
- Risk Factors
- Recent travel to endemic area
- Food borne Illness
- Waterborne Illness
- Day care exposure
- High-risk sexual behavior
- Antibiotic use within 6 months (C-Difficile)
53Infection
- Diagnostic tests
- Stool culture
- Blood chemistry
- Medical management/nursing interventions
- Antibiotics
- Fluid and electrolyte replacement
- Kaopectate
- Pepto-Bismol
54Irritable Bowel Syndrome (IBS)
- Etiology/Pathophysiology
- Episodes of alteration in bowel function
- Spastic and uncoordinated muscle contractions of
the colon - Related to eating coarse or highly seasoned food
- Clinical manifestations
- Abdominal pain and distention relieved by BM
- Frequent bowel movements
- Sense of incomplete evacuation
- Flatulence, constipation, and/or diarrhea
55Irritable Bowel Syndrome (IBS)
- Red Flags Suggestive of other diagnosis
- Nighttime Diarrhea
- Nocturnal stool incontinence
- Nocturnal awakening due to abdominal discomfort
- Abdominal pain that interferes with normal sleep
- Visible or occult blood in stool
- Weight loss
- Recurrent Fever
- Family History of Colon Cancer
- Family History of Inflammatory Bowel Disease
- Laboratory abnormality
- Leukocytosis
- Anemia
- Increased Erythrocyte Sedimentation Rate (ESR)
56Irritable Bowel Syndrome (IBS)
- Increase in functional diarrhea with stress
- Associated with psychiatric issues
- Panic attacks
- Chronic pelvic pain
- Childhood sexual abuse
- Diagnostic tests
- History and physical examination
- Often by exclusion
57Irritable Bowel Syndrome (IBS)
- Medical management Nursing interventions
- Diet and bulking agents
- Education
- Medications
- Anticholinergics
- Milk of Magnesia, fiber, or mineral oil
- Opioids
- Anti-anxiety drugs
58Ulcerative Colitis
- Etiology/Pathophysiology
- Ulceration of the mucosa and submucosa of the
colon - Tiny abscesses form
- Produce purulent drainage, slough the mucosa, and
ulcerations occur - Starts on the left and progresses to the right
side of colon - The capillaries become friable and bleed
- bloody diarrhea
- Pseudopolyps may turn cancerous
- Scar tissue may form
- Loss of elasticity and absorption abilities
59Ulcerative Colitis
- Clinical manifestations/Assessment
- Diarrhea-pus and blood
- 15-20 stools per day
- Fluid/electrolyte imbalance
- Abdominal cramping
- Involuntary leakage of stool
- The urge to defecate lessons as scar tissue
replaces functional tissue - Weight loss and deficient nutrition
- Megacolon
60Ulcerative Colitis
- Diagnostic tests
- Barium studies
- Colonoscopy biopsy
- Stool for occult blood
- X-rays
- Labs such as chemistries, CBC
- Medical management/Nursing interventions
- Diet
- No milk products or spicy foods
- High protein, high calorie
- Total parenteral nutrition (TPN)
- Stress control
- Assist patient to find coping mechanisms
61Ulcerative Colitis
- Medical management/Nursing interventions
- Medications
- Azulfidine
- Dipentum Rowasa
- Corticosteroids
- Imodium
- Anticholinergic drugs are avoided
- Surgical interventions
- Colon resection
- Ileostomy
- Ileoanal anastomosis
- Proctocolectomy
- Kock pouch
62Crohns Disease
- Etiology/Pathophysiology
- Inflammation, fibrosis, scarring, and thickening
of bowel wall - Inflammation of segments of the GI tract from
mouth to anus - Possible association between Crohns and altered
immune mechanisms - Typically occurs in the small intestine (jejunum
and terminal ileum) - Clinical manifestations/Assessment
- Weakness, loss of appetite, dehydration
- Diarrhea, 3-4 daily, contain mucus and pus
(steatorrhea) - Right lower abdominal pain
- Anal fissures and/or fistulas
- Anemia
63Crohns Disease
- Complications
- Malabsorption
- Pernicious anemia
- Acid/base disturbance related to diarrhea
- Fistulas may occur
- Medical management/Nursing interventions
- Diet
- High protein
- High in nutrients
- Avoid lactose-containing foods, vegetables,
caffeine, beer, MSG, highly seasoned foods,
carbonated beverages, fatty foods
64Crohns Disease
- Medical management/Nursing interventions
- Medications
- Corticosteroids
- Antibiotics
- Anti-diarrheals
- Antispasmodics
- Enteric-coated fish oil capsules
- B12 replacement
- Surgery
- Segmental resection of diseased bowel
- Diagnostic tools
- Small bowel barium
- Colonoscopy with biopsy
65Crohns Disease
66Colon surgery and colostomy procedure
67Colostomy and Bowel Resections
- Sigmoid Resection Right
- with colostomy Hemicoloctomy
68Bowel Resections
- Left Hemicolotomy Anterior Recto
Sigmoidectomy
69Appendicitis
- Etiology/Pathophysiology
- Inflammation of the vermiform appendix
- Lumen of the appendix becomes obstructed
- Clinical manifestations/Assessment
- Rebound tenderness over McBurneys point
- Vomiting
- Low-grade fever
- Diagnostic tests
- WBC
- CT scan
- Ultrasound
70Appendicitis
- Medical management/Nursing interventions
- Laparoscopy
- Appendectomy
- Post-op complications
- Peritonitis
- Intra-abdominal abscess
- Mechanical bowel obstruction
71Diverticular Disease
- Etiology/Pathophysiology
- Diverticuli become inflamed
- Fecal matter becomes stuck in the pouches
- Perforation is a risk
- Diverticulosis
- Pouch-like herniations through the muscular layer
of the colon - Diverticulitis
- Inflammation of one or more diverticuli
72Diverticular Disease
- Clinical manifestations/Assessment
- Patient can become septic
- Diverticulosis
- May have few, if any, symptoms
- Constipation, diarrhea, and/or flatulence
- Pain in the left lower quadrant
- Diverticulitis
- Mild to severe pain in the left lower quadrant
- Elevated WBC
- Low-grade fever
- Abdominal distention
- Vomiting
- Blood in stool
73Diverticular Disease
- Medical management/Nursing interventions
- A temporary colostomy may be indicated with
closure in 6 weeks to 3 months - Diverticulosis with muscular atrophy
- Low-residue diet stool softeners
- Bedrest
- Diverticulosis with increased intracolonic
pressure and muscle thickening - High-fiber diet
- Sulfa drugs
- Antibiotics if indicated
- Analgesics
74Diverticular Disease
- Medical management/Nursing interventions
- Surgery
- Hartmanns pouch
- Double-barrel transverse colostomy
75Diverticulosis
76Progression of Diverticular Disease
77Diverticulitis
78Umbilical Stoma Stoma Infected with Candida
Albicans
79Prolapsed Loop Colostomy
80Colostomy Pouch
81Colostomy Irrigation
82Pancreatitis
- Pancreatic enzymes back up into pancreas
- Can be fatal
- Inflammation of the peritoneum following
infection - Localized
- Generalized
83Peritonitis
- Etiology/Pathophysiology
- Inflammation of the abdominal peritoneum
- Bacterial contamination of the peritoneal cavity
from fecal matter or chemical irritation - Medical emergency
- The prognosis for untreated peritonitis is very
poor
84Peritonitis
- Causes
- Internal perforation of the gastrointestinal
tract - Intestinal foreign body
- Colonic diverticulum
- A ruptured appendix
85Peritonitis
- Clinical manifestations/Assessment
- Severe abdominal pain
- May present in the fetal position with knees
drawn up - Pain with movement
- Nausea and vomiting
- Abdomen is rigid
- Tympanic or absent bowel sounds
- Chills
- Weakness
- Weak rapid pulse
- Fever
- Hypotension
86Peritonitis
- Diagnostic tests
- Flat plate of the abdomen
- CT scan
- Medical management
- Semi-Fowlers position
- Surgery
- Fecal contamination
- Removal irritants
- Parenteral antibiotics
- NG tube
- IV fluids
87Meconium peritonitis
88External Hernias
- Etiology/Pathophysiology
- Congenital or acquired weakness of the abdominal
wall or postoperative defect - Hernia is a protrusion of viscus through an
abnormal opening or weakened wall of the cavity
in which it was normally contained - Abdominal or Ventral
- Femoral or inguinal
- Umbilical
89External Hernias
- Clinical manifestations
- Protrusion at umbilicus, groin or incision
- Pain may or may not be present
- Complications
- Incarceration
- Strangulation
- Diagnostic tests
- Radiographs
- Palpation
90External Hernias
- Medical management/Nursing interventions
- If no discomfort, hernia is left unrepaired,
unless it becomes strangulated or obstruction
occurs - Truss
- Surgery
- Synthetic mesh is applied to weakened area of the
abdominal wall
91Umbilical Hernia
92Hiatal Hernia
- Etiology/Pathophysiology
- Protrusion of the stomach and other abdominal
viscera through an opening in the membrane or
tissue of the diaphragm - Contributing factors obesity, trauma, aging
- Clinical manifestations/Assessment
- Most people display few, if any, symptoms
- Gastroesophageal reflux
93Hiatal Hernia
- Medical management/Nursing intervention
- Head of bed should be slightly elevated when
lying down - Small frequent meals
- OTC medications for GERD
- Surgery
- Posterior gastropexy
- Transabdominal
- fundoplication (Nissen)
94Hiatal Hernia
- Intestinal obstruction
- Etiology/pathophysiology
- Intestinal contents cannot pass
- Partial or complete
- Mechanical
- Non-mechanical
- Clinical manifestations/assessment
- Vomiting dehydration
- Abdominal tenderness and distention
- Constipation
95Hiatal Hernia
Sliding hernia
Rolling hernia
96Intestinal Obstruction
- Etiology/Pathophysiology
- Mechanical
- An occlusion of the lumen of the intestinal tract
- Adhesions, incarcerated hernias, impacted feces,
tumor, intussusception, volvulus, strictures of
inflammatory bowel disease - Non-mechanical
- Neuromuscular or vascular disorder
- Paralytic ileus, trauma to the spine, mesenteric
thrombosis that reduces blood flow to the bowel
97Intestinal Obstruction
Intestinal obstructions. A, Adhesions. B,
Volvulus.
98Intestinal Obstruction
- Clinical manifestations/Assessment
- Loud high pitched bowel sounds above the
obstruction and absent bowel sounds below - Inability to pass stool or gas
- Vomiting, abdominal cramps, abdominal distention
- Diagnostic tests
- Radiographic examinations
- BUN, sodium, potassium,
- hemoglobin and hematocrit
99Intestinal Obstruction
- Medical management/Nursing interventions
- Evacuation of intestine
- NG tube to decompress the bowel
- Intestinal tube to evacuate the contents
- Surgery
- Required for mechanical obstructions
- Bowel resection
100Hemorrhoids
- Etiology/Pathophysiology
- Varicosities (dilated anorectal vessels/veins)
- External or internal
- Contributing factors
- Straining with defecation, diarrhea,
constipation, pregnancy, obesity, CHF, portal
hypertension, family history, prolonged sitting
and standing - Anatomic abnormalities
- Intrinsic weakness of anal blood vessels
101Hemorrhoids
- Clinical manifestations/Assessment
- Varicosities in rectal area
- Bright red bleeding with defecation
- Pruritus
- Rectal Pain
- Prolapsed hemorrhoid
- Fullness or mass sensation
102Hemorrhoids
- Diagnostic tests
- Visual inspection
- Digital Rectal Exam
- Anoscopy
- Differential Diagnosis
- Fissure
- Perirectal Abscess
- Rectal Fistula
- Condyloma
- Rectal carcinoma
103Hemorrhoids
- Medical management/Nursing interventions
- Bulk stool softeners
- Hydrocortisone cream
- Analgesic ointment
- Sitz baths
- Ligation
- Sclerotherapy
- Cryotherapy
- Infrared photocoagulation
- Hemorrhoidectomy
104Anal Fissure
- Etiology/Pathophysiology
- Linear ulceration or laceration of the skin of
the anus - Usually caused by trauma
- Lesions usually heal spontaneously
- May be excised surgically
- Usually follows trauma or diarrheal illness
- Increased anodermal blood flow causes fissures
- Affects young and middle-aged adults
105Anal Fissure
- Clinical manifestations/Assessment
- Onset after forced hard bowel movement
- Bright red rectal bleeding
- Pain during bowel movement
- Cut with sharp glass sensation
- Pain persists for an hour after stooling
- Avoid anoscopy if possible
- Painful and usually not needed
- Use local anesthesia if performed
106Anal Fissure
- Clinical manifestations/Assessment (continued)
- Crack or crevice in anoderm at anal verge
- Usually in canal midline (anterior or posterior)
- Lateral suggests other diagnosis
- Best seen with lateral traction on opposite
buttock - Sentinel pile (distal skin tag)
- Tag-like swelling of fissure end
- Results from infection and edema
107Anal Fistula
- Etiology/Pathophysiology
- Abnormal opening on the surface near the anus
- Usually from a local abscess
- Common in Crohns disease
- Treated by a fistulectomy or fistulotomy
- Perirectal abscess sequelae of rupture or surgery
- Anal Fissure
- Anorectal cancer
- Tuberculosis
- Local radiation therapy
- Lymphogranuloma venereum
108Anal Fistula
- Clinical manifestations/Assessment
- Chronic seropurulent or mucus drainage from
fistula - Communicating tract between perianal skin and
anus - One or several external openings tracking toward
anus - Associated conditions
- Perirectal Abscess